26 research outputs found

    Do tabloids poison the well of social media? Explaining democratically dysfunctional news sharing

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    This paper was accepted for publication in the journal New Media and Society and the definitive published version is available at https://doi.org/10.1177/1461444818769689The use of social media for sharing political information and the status of news as an essential raw material for good citizenship are both generating increasing public concern. We add to the debates about misinformation, disinformation, and “fake news” using a new theoretical framework and a unique research design integrating survey data and analysis of observed news sharing behaviors on social media. Using a media-as-resources perspective, we theorize that there are elective affinities between tabloid news and misinformation and disinformation behaviors on social media. Integrating four data sets we constructed during the 2017 UK election campaign—individual-level data on news sharing (N = 1,525,748 tweets), website data (N = 17,989 web domains), news article data (N = 641 articles), and data from a custom survey of Twitter users (N = 1313 respondents)—we find that sharing tabloid news on social media is a significant predictor of democratically dysfunctional misinformation and disinformation behaviors. We explain the consequences of this finding for the civic culture of social media and the direction of future scholarship on fake news

    Human malarial disease: a consequence of inflammatory cytokine release

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    Malaria causes an acute systemic human disease that bears many similarities, both clinically and mechanistically, to those caused by bacteria, rickettsia, and viruses. Over the past few decades, a literature has emerged that argues for most of the pathology seen in all of these infectious diseases being explained by activation of the inflammatory system, with the balance between the pro and anti-inflammatory cytokines being tipped towards the onset of systemic inflammation. Although not often expressed in energy terms, there is, when reduced to biochemical essentials, wide agreement that infection with falciparum malaria is often fatal because mitochondria are unable to generate enough ATP to maintain normal cellular function. Most, however, would contend that this largely occurs because sequestered parasitized red cells prevent sufficient oxygen getting to where it is needed. This review considers the evidence that an equally or more important way ATP deficency arises in malaria, as well as these other infectious diseases, is an inability of mitochondria, through the effects of inflammatory cytokines on their function, to utilise available oxygen. This activity of these cytokines, plus their capacity to control the pathways through which oxygen supply to mitochondria are restricted (particularly through directing sequestration and driving anaemia), combine to make falciparum malaria primarily an inflammatory cytokine-driven disease

    Print and online newspapers as material artefacts

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    Traditional newspaper journalism is in a state of crisis and there have been several attempts to overcome this. Many discourses have reiterated the triumphal march of a digital revolution in newspaper journalism and anticipated the end of the print newspaper. This moment calls for an in-depth analysis of reader habits of news consumption and use in order to understand the audiences for journalistic output and their relationship with the journalistic objects. In this study, we adopt a multi-method approach, integrating (1) qualitative content analysis of student essays dealing with the physicality of printed and online newspapers, (2) ethnographic observation of the use practices of readers and (3) expert interview. The findings show that informants perceive print and online newspapers as objects with which they have a different experience and highlight the need to develop bridging strategies combining print and digital media in order to overcome the crisis facing printed news media.peerReviewe
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