Macroscopic evidence for quantum criticality and field-induced quantum fluctuations in cuprate superconductors

We present macroscopic experimental evidence for field-induced microscopic quantum fluctuations in different hole- and electron-type cuprate superconductors with varying doping levels and numbers of CuO$_2$ layers per unit cell. The significant suppression of the zero-temperature in-plane magnetic irreversibility field relative to the paramagnetic field in all cuprate superconductors suggests strong quantum fluctuations due to the proximity of the cuprates to quantum criticality.Comment: 3 figures. To appear in Phys. Rev. B, Rapid Communications (2007). For correspondence, contact: Nai-Chang Yeh (e-mail: [email protected]

Experimental investigation of the competing orders and quantum criticality in hole- and electron-doped cuprate superconductors

We investigate the issues of competing orders and quantum criticality in cuprate superconductors via experimental studies of the high-field thermodynamic phase diagrams and the quasiparticle tunneling spectroscopy. Substantial field-induced quantum fluctuations are found in all cuprates investigated, and the corresponding correlation with quasiparticle spectra suggest that both electron- (n-type) and hole-doped (p-type) cuprate superconductors are in close proximity to a quantum critical point that separates a pure superconducting (SC) phase from a phase consisting of coexisting SC and a competing order. We further suggests that the relevant competing order is likely a spin-density wave (SDW) or a charge density wave (CDW), which can couple efficiently to an in-plane Cu-O bond stretching longitudinal optical (LO) phonon mode in the p-type cuprates but not in the n-type cuprates. This cooperative interaction may account for the pseudogap phenomenon above T, only in the p-type cuprate superconductors

Quasiparticle spectroscopy and high-field phase diagrams of cuprate superconductors -- An investigation of competing orders and quantum criticality

We present scanning tunneling spectroscopic and high-field thermodynamic studies of hole- and electron-doped (p- and n-type) cuprate superconductors. Our experimental results are consistent with the notion that the ground state of cuprates is in proximity to a quantum critical point (QCP) that separates a pure superconducting (SC) phase from a phase comprised of coexisting SC and a competing order, and the competing order is likely a spin-density wave (SDW). The effect of applied magnetic field, tunneling current, and disorder on the revelation of competing orders and on the low-energy excitations of the cuprates is discussed.Comment: 10 pages, 5 figures. Accepted for publication in the International Journal of Modern Physics B. (Correspondence author: Nai-Chang Yeh, e-mail: [email protected]

Dimensionality of superconductivity in the infinite-layer high-temperature cuprate Sr0.9M0.1CuO2 (M = La, Gd)

The high magnetic field phase diagram of the electron-doped infinite layer high-temperature superconducting (high-T_c) compound Sr_{0.9}La_{0.1}CuO_2 was probed by means of penetration depth and magnetization measurements in pulsed fields to 60 T. An anisotropy ratio of 8 was detected for the upper critical fields with H parallel (H_{c2}^{ab}) and perpendicular (H_{c2}^c) to the CuO_2 planes, with H_{c2}^{ab} extrapolating to near the Pauli paramagnetic limit of 160 T. The longer superconducting coherence length than the lattice constant along the c-axis indicates that the orbital degrees of freedom of the pairing wavefunction are three dimensional. By contrast, low-field magnetization and specific heat measurements of Sr_{0.9}Gd_{0.1}CuO_2 indicate a coexistence of bulk s-wave superconductivity with large moment Gd paramagnetism close to the CuO_2 planes, suggesting a strong confinement of the spin degrees of freedom of the Cooper pair to the CuO_2 planes. The region between H_{c2}^{ab} and the irreversibility line in the magnetization, H_{irr}^{ab}, is anomalously large for an electron-doped high-T_c cuprate, suggesting the existence of additional quantum fluctuations perhaps due to a competing spin-density wave order.Comment: 4 pages, 4 figures, submitted to Phys. Rev. B, Rapid Communications (2004). Corresponding author: Nai-Chang Yeh (E-mail: [email protected]

First Results from the AMoRE-Pilot neutrinoless double beta decay experiment

The Advanced Molybdenum-based Rare process Experiment (AMoRE) aims to search for neutrinoless double beta decay (0$\nu\beta\beta$) of $^{100}$Mo with $\sim$100 kg of $^{100}$Mo-enriched molybdenum embedded in cryogenic detectors with a dual heat and light readout. At the current, pilot stage of the AMoRE project we employ six calcium molybdate crystals with a total mass of 1.9 kg, produced from $^{48}$Ca-depleted calcium and $^{100}$Mo-enriched molybdenum ($^{48\textrm{depl}}$Ca$^{100}$MoO$_4$). The simultaneous detection of heat(phonon) and scintillation (photon) signals is realized with high resolution metallic magnetic calorimeter sensors that operate at milli-Kelvin temperatures. This stage of the project is carried out in the Yangyang underground laboratory at a depth of 700 m. We report first results from the AMoRE-Pilot $0\nu\beta\beta$ search with a 111 kg$\cdot$d live exposure of $^{48\textrm{depl}}$Ca$^{100}$MoO$_4$ crystals. No evidence for $0\nu\beta\beta$ decay of $^{100}$Mo is found, and a upper limit is set for the half-life of 0$\nu\beta\beta$ of $^{100}$Mo of $T^{0\nu}_{1/2} > 9.5\times10^{22}$ y at 90% C.L.. This limit corresponds to an effective Majorana neutrino mass limit in the range $\langle m_{\beta\beta}\rangle\le(1.2-2.1)$ eV

Quantum Smoluchowski equation: Escape from a metastable state

We develop a quantum Smoluchowski equation in terms of a true probability distribution function to describe quantum Brownian motion in configuration space in large friction limit at arbitrary temperature and derive the rate of barrier crossing and tunneling within an unified scheme. The present treatment is independent of path integral formalism and is based on canonical quantization procedure.Comment: 10 pages, To appear in the Proceedings of Statphys - Kolkata I

PPAR? Downregulation by TGF in Fibroblast and Impaired Expression and Function in Systemic Sclerosis: A Novel Mechanism for Progressive Fibrogenesis

The nuclear orphan receptor peroxisome proliferator-activated receptor-gamma (PPAR-γ) is expressed in multiple cell types in addition to adipocytes. Upon its activation by natural ligands such as fatty acids and eicosanoids, or by synthetic agonists such as rosiglitazone, PPAR-γ regulates adipogenesis, glucose uptake and inflammatory responses. Recent studies establish a novel role for PPAR-γ signaling as an endogenous mechanism for regulating transforming growth factor-ß (TGF-ß)- dependent fibrogenesis. Here, we sought to characterize PPAR-γ function in the prototypic fibrosing disorder systemic sclerosis (SSc), and delineate the factors governing PPAR-γ expression. We report that PPAR-γ levels were markedly diminished in skin and lung biopsies from patients with SSc, and in fibroblasts explanted from the lesional skin. In normal fibroblasts, treatment with TGF-ß resulted in a time- and dose-dependent down-regulation of PPAR-γ expression. Inhibition occurred at the transcriptional level and was mediated via canonical Smad signal transduction. Genome-wide expression profiling of SSc skin biopsies revealed a marked attenuation of PPAR-γ levels and transcriptional activity in a subset of patients with diffuse cutaneous SSc, which was correlated with the presence of a ''TGF-ß responsive gene signature'' in these biopsies. Together, these results demonstrate that the expression and function of PPAR-γ are impaired in SSc, and reveal the existence of a reciprocal inhibitory cross-talk between TGF-ß activation and PPAR-γ signaling in the context of fibrogenesis. In light of the potent anti-fibrotic effects attributed to PPAR-γ, these observations lead us to propose that excessive TGF-ß activity in SSc accounts for impaired PPAR-γ function, which in turn contributes to unchecked fibroblast activation and progressive fibrosis. © 2010 Wei et al