Bloom-Gilman duality of the nucleon structure function and the elastic peak contribution

The occurrence of the Bloom-Gilman duality in the nucleon structure function is investigated by analyzing the Q**2-behavior of low-order moments, both including and excluding the contribution arising from the nucleon elastic peak. The Natchmann definition of the moments has been adopted in order to cancel out target-mass effects. It is shown that the onset of the Bloom-Gilman duality occurs around Q**2 ~ 2 (GeV/c)**2 if only the inelastic part of the nucleon structure function is considered, whereas the inclusion of the nucleon elastic peak contribution leads to remarkable violations of the Bloom-Gilman duality.Comment: in Proc. of the XVI European Conference on Few-body Problems in Physics, Autrans (France), July 199

On Justification, Idealization, and Discursive Purchase

Conceptions of acceptability-based moral or political justification take it that authoritative acceptability, widely conceived, constitutes, or contributes to, validity, or justification. There is no agreement as to what bar for authoritativeness such justification may employ. The paper engages the issue in relation to (i) the level of idealization that a bar for authoritativeness, ψ, imparts to a standard of acceptability-based justification, S, and (ii) the degree of discursive purchase of the discursive standing that S accords to people when it builds ψ. I argue that (i) and (ii) are interdependent: high idealization values entail low discursive purchase, while high degrees of purchase require low idealization values. I then distinguish between alethic conceptions of justification that prioritize ends that commit to high idealization values, and recognitive conceptions that favor high discursive purchase. On this basis, I argue for a moderately recognitivist constraint on idealization. To render the recognitive discursive minimum available to relevant people at the site of justification, S should set ψ low enough so that it is a genuine option for actual people to reject relevant views in ways that S recognizes as authoritative. (The Appendix applies this to a Forst-type view of reciprocity of reasons to draw out some limitations of this view.) [Draft available from author on request.

CERN West Area neutrino facility beam line alignment

This papers describes the alignment of the West Area Neutrino Beam Line at CERN to the two neutrino experiments CHORUS and NOMAD. The T9 neutrino (n) target position and the position of the magnetic horn were optimised using the secondary muon intensity profiles from the muon pits in the shielding. In the experiments the improved geometry provides a better centred beam (< 5 cm) and a measured increase in the n flux of 8%

In non-transformed cells Bak activates upon loss of anti-apoptotic Bcl-X-L and Mcl-1 but in the absence of active BH3-only proteins

Mitochondrial apoptosis is controlled by proteins of the B-cell lymphoma 2 (Bcl-2) family. Pro-apoptotic members of this family, known as BH3-only proteins, initiate activation of the effectors Bcl-2-associated X protein (Bax) and Bcl-2 homologous antagonist/killer (Bak),which is counteracted by anti-apoptotic family members. How the interactions of Bcl-2 proteins regulate cell death is still not entirely clear. Here, we show that in the absence of extrinsic apoptotic stimuli Bak activates without detectable contribution from BH3-only proteins, and cell survival depends on anti-apoptotic Bcl-2 molecules. All anti-apoptotic Bcl-2 proteins were targeted via RNA interference alone or in combinations of two in primary human fibroblasts. Simultaneous targeting of B-cell lymphoma-extra large and myeloid cell leukemia sequence 1 led to apoptosis in several cell types. Apoptosis depended on Bak whereas Bax was dispensable. Activator BH3-only proteins were not required for apoptosis induction as apoptosis was unaltered in the absence of all BH3-only proteins known to activate Bax or Bak directly, Bcl-2-interacting mediator of cell death, BH3-interacting domain death agonist and p53-upregulated modulator of apoptosis. These findings argue for auto-activation of Bak in the absence of anti-apoptotic Bcl-2 proteins and provide evidence of profound differences in the activation of Bax and Bak

Vector Meson Photoproduction with an Effective Lagrangian in the Quark Model

A quark model approach to the photoproduction of vector mesons off nucleons is proposed. Its starting point is an effective Lagrangian of the interaction between the vector meson and the quarks inside the baryon, which generates the non-diffractive s- and u- channel resonance contributions. Additional t-channel $\pi^0$ and $\sigma$ exchanges are included for the $\omega$ and $\rho^0$ production respectively to account for the large diffractive behavior in the small $t$ region as suggested by Friman and Soyeur. The numerical results are presented for the $\omega$ and $\rho$ productions in four isospin channels with the same set of parameters, and they are in good agreement with the available data not only in $\omega$ and $\rho^0$ productions but also in the charged $\rho$ productions where the additional t-channel $\sigma$ exchange does not contribute so that it provides an important test to this approach. The investigation is also extended to the $\phi$ photoproduction, and the initial results show that the non-diffractive behavior of the $\phi$ productions in the large $t$ region can be described by the s- and u- channel contributions with significantly smaller coupling constants, which is consistent with the findings in the similar studies in the QHD framework. The numerical investigation has also shown that polarization observables are essential for identifying so-called "missing resonances".Comment: 36 pages, 10 PS figures, extended version of nucl-th/9711061 and nucl-th/9803021, submitted to PR

MAJOR 1.5 - A Monte Carlo Generator for Heavy Majorana Neutrinos in epep Collisions

The Monte Carlo generator {\sc Major} 1.5 simulates the production and decay of heavy Majorana neutrinos via lepton mixing or exchange of `light' right-handed $W$-bosons in deep inelastic scattering, i.e. $e^{\pm} p \rightarrow {N} X \rightarrow{e}^{\pm} W^{\mp} X$ or $\nu_e Z X$. Physics and programming aspects are described in this manual.Comment: 13 pages LaTeX, 2 PostScript figure

In non-transformed cells Bak activates upon loss of anti-apoptotic Bcl-X-L and Mcl-1 but in the absence of active BH3-only proteins

Mitochondrial apoptosis is controlled by proteins of the B-cell lymphoma 2 (Bcl-2) family. Pro-apoptotic members of this family, known as BH3-only proteins, initiate activation of the effectors Bcl-2-associated X protein (Bax) and Bcl-2 homologous antagonist/killer (Bak),which is counteracted by anti-apoptotic family members. How the interactions of Bcl-2 proteins regulate cell death is still not entirely clear. Here, we show that in the absence of extrinsic apoptotic stimuli Bak activates without detectable contribution from BH3-only proteins, and cell survival depends on anti-apoptotic Bcl-2 molecules. All anti-apoptotic Bcl-2 proteins were targeted via RNA interference alone or in combinations of two in primary human fibroblasts. Simultaneous targeting of B-cell lymphoma-extra large and myeloid cell leukemia sequence 1 led to apoptosis in several cell types. Apoptosis depended on Bak whereas Bax was dispensable. Activator BH3-only proteins were not required for apoptosis induction as apoptosis was unaltered in the absence of all BH3-only proteins known to activate Bax or Bak directly, Bcl-2-interacting mediator of cell death, BH3-interacting domain death agonist and p53-upregulated modulator of apoptosis. These findings argue for auto-activation of Bak in the absence of anti-apoptotic Bcl-2 proteins and provide evidence of profound differences in the activation of Bax and Bak

Verifiable Model of Neutrino Masses from Large Extra Dimensions

We propose a new scenario of neutrino masses with a Higgs triplet $(\xi^{++},\xi^+,\xi^0)$ in a theory of large extra dimensions. Lepton number violation in a distant brane acts as the source of a very small trilinear coupling of $\xi$ to the standard Higgs doublet in our brane. Small realistic Majorana neutrino masses are \underline{naturally} obtained with the fundamental scale $M_* \sim {\cal O}(1)$ TeV, foretelling the possible discovery of $\xi$ (m_\xi\lsim M_*) at future colliders. Decays of $\xi^{++}$ into same-sign dileptons are fixed by the neutrino mass matrix. Observation of $\mu-e$ conversion in nuclei is predicted.Comment: A comment on Tevatron reach and two references added. Discussion and conclusions unchange

Parton model versus color dipole formulation of the Drell-Yan process

In the kinematical region where the center of mass energy is much larger than all other scales, the Drell-Yan process can be formulated in the target rest frame in terms of the same color dipole cross section as low Bjorken-x deep inelastic scattering. Since the mechanisms for heavy dilepton production appear very different in the dipole approach and in the conventional parton model, one may wonder whether these two formulations really represent the same physics. We perform a comparison of numerical calculations in the color dipole approach with calculations in the next-to-leading order parton model. For proton-proton scattering, the results are very similar at low x_2 from fixed target to RHIC energies, confirming the close connection between these two very different approaches. We also compare the transverse momentum distributions of Drell-Yan dileptons predicted in both formulations. The range of applicability of the dipole formulation and the impact of future Drell-Yan data from RHIC for determining the color dipole cross section are discussed. A detailed derivation of the dipole formulation of the Drell-Yan process is also included.Comment: 20 pages, 5 figure

Hard diffraction in hadron--hadron interactions and in photoproduction

Hard single diffractive processes are studied within the framework of the triple--Pomeron approximation. Using a Pomeron structure function motivated by Regge--theory we obtain parton distribution functions which do not obey momentum sum rule. Based on Regge-- factorization cross sections for hard diffraction are calculated. Furthermore, the model is applied to hard diffractive particle production in photoproduction and in $p\bar{p}$ interactions.Comment: 13 pages, Latex, 13 uuencoded figure