Microorganisms isolated in patients with ventilator-associated pneumonia.

<p>Microorganisms isolated in patients with ventilator-associated pneumonia.</p

Patient outcomes.

<p>Patient outcomes.</p

MOESM1 of Respiratory changes of the inferior vena cava diameter predict fluid responsiveness in spontaneously breathing patients with cardiac arrhythmias

Additional file 1: Figure S1. A, Receiver operating characteristics (ROC) curve of the collapsibility index (cIVC-st) and the inspiratory diameter (iIVC-st) of the inferior vena cava during a standardized inspiratory maneuver before volume expansion (VE) to discriminate responders from nonresponders to VE in the overall population. B, ROC curve of the collapsibility index (cIVC-sp) and the inspiratory diameter (iIVC-sp) of the inferior vena cava during unstandardized spontaneous breathing before VE to discriminate responders from nonresponders to VE in the overall population. Figure S2. A, Linear correlation between the collapsibility index of the inferior vena cava under standardized breathing (cIVC-st) before volume expansion (VE) and VE-induced change in the velocity time integral of aortic blood flow (VTIao). B, Linear correlation between the inspiratory diameter of the inferior vena cava under standardized breathing (iIVC-st) before VE and VE-induced change in VTIao. Figure S3. Scatterplot of individual values before volume expansion (VE) for the collapsibility index (cIVC-sp), minimum-inspiratory diameter (iIVC-sp), and the end-expiratory diameter of the inferior vena cava (eIVC-sp) under unstandardized spontaneous breathing in responders and nonresponders to VE

Patient characteristics at ICU admission.

<p>Patient characteristics at ICU admission.</p

MOESM2 of Respiratory changes of the inferior vena cava diameter predict fluid responsiveness in spontaneously breathing patients with cardiac arrhythmias

Additional file 2: Table S1. Respiratory variables in responders and nonresponders before and after volume expansion. Table S2. Volume expansion-induced changes in hemodynamic variables in responders and nonresponders. Table S3. Baseline characteristics of the patients (VE-related change in VTIao ≥ 15% to define responders). Table S4. Hemodynamic variables before and after volume expansion in responders and nonresponders (VE-related change in VTIao ≥ 15% to define responders). Table S5. Accuracy of the inferior vena cava variables for predicting response to volume expansion (VE-related change in VTIao ≥ 15% to define responders)

Few-Electron Edge-State Quantum Dots in a Silicon Nanowire Field-Effect Transistor

We investigate the gate-induced onset of few-electron regime through the undoped channel of a silicon nanowire field-effect transistor. By combining low-temperature transport measurements and self-consistent calculations, we reveal the formation of one-dimensional conduction modes localized at the two upper edges of the channel. Charge traps in the gate dielectric cause electron localization along these edge modes, creating elongated quantum dots with characteristic lengths of ∼10 nm. We observe single-electron tunneling across two such dots in parallel, specifically one in each channel edge. We identify the filling of these quantum dots with the first few electrons, measuring addition energies of a few tens of millielectron volts and level spacings of the order of 1 meV, which we ascribe to the valley orbit splitting. The total removal of valley degeneracy leaves only a 2-fold spin degeneracy, making edge quantum dots potentially promising candidates for silicon spin qubits