241 research outputs found
The R-Process Alliance: Chemical Abundances for a Trio of R-Process-Enhanced Stars -- One Strong, One Moderate, One Mild
We present detailed chemical abundances of three new bright (V ~ 11),
extremely metal-poor ([Fe/H] ~ -3.0), r-process-enhanced halo red giants based
on high-resolution, high-S/N Magellan/MIKE spectra. We measured abundances for
20-25 neutron-capture elements in each of our stars. J1432-4125 is among the
most r-process rich r-II stars, with [Eu/Fe]= +1.44+-0.11. J2005-3057 is an r-I
star with [Eu/Fe] = +0.94+-0.07. J0858-0809 has [Eu/Fe] = +0.23+-0.05 and
exhibits a carbon abundance corrected for evolutionary status of [C/Fe]_corr =
+0.76, thus adding to the small number of known carbon-enhanced r-process
stars. All three stars show remarkable agreement with the scaled solar
r-process pattern for elements above Ba, consistent with enrichment of the
birth gas cloud by a neutron star merger. The abundances for Sr, Y, and Zr,
however, deviate from the scaled solar pattern. This indicates that more than
one distinct r-process site might be responsible for the observed
neutron-capture element abundance pattern. Thorium was detected in J1432-4125
and J2005-3057. Age estimates for J1432-4125 and J2005-3057 were adopted from
one of two sets of initial production ratios each by assuming the stars are
old. This yielded individual ages of 12+-6 Gyr and 10+-6 Gyr, respectively.Comment: 30 pages, includes a long table, 5 figure
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Macrophages promote epithelial proliferation following infectious and non-infectious lung injury through a Trefoil factor 2-dependent mechanism.
Coordinated efforts between macrophages and epithelia are considered essential for wound healing, but the macrophage-derived molecules responsible for repair are poorly defined. This work demonstrates that lung macrophages rely upon Trefoil factor 2 to promote epithelial proliferation following damage caused by sterile wounding, Nippostrongylus brasiliensis or Bleomycin sulfate. Unexpectedly, the presence of T, B, or ILC populations was not essential for macrophage-driven repair. Instead, conditional deletion of TFF2 in myeloid-restricted CD11cCre TFF2 flox mice exacerbated lung pathology and reduced the proliferative expansion of CD45- EpCAM+ pro-SPC+ alveolar type 2 cells. TFF2 deficient macrophages had reduced expression of the Wnt genes Wnt4 and Wnt16 and reconstitution of hookworm-infected CD11cCre TFF2flox mice with rWnt4 and rWnt16 restored the proliferative defect in lung epithelia post-injury. These data reveal a previously unrecognized mechanism wherein lung myeloid phagocytes utilize a TFF2/Wnt axis as a mechanism that drives epithelial proliferation following lung injury
Model-based motion estimation for synthetic animations
One approach to performing motion estimation on syn-thetic animations is to treat them as video sequences and use standard image-based motion estimation meth-ods. Alternatively, we can take advantage of informa-tion used in rendering the animation to guide the motion estimation algorithm. This information includes the 3D movements of the objects in the scene and the projec-tion transformations from 3D world space into screen space. In this paper we examine how to use this high level object motion information to perform fast, accu-rate block-based motion estimation for synthetic anima-tions. The optical
ow eld is a 2D vector eld describ-ing the translational motion of each pixel from frame to frame. Our motion estimation algorithm rst com-putes the optical
ow eld, based on the object motion information. We then combine the per-pixel motion in-formation for a block of pixels to create a single 2D projective matrix that best encodes the motion of all the pixels in the block. The entries of the 2D matrix are determined using a least squares formulation. Our algo-rithms are more accurate and much faster in algorithmic complexity than many image-based motion estimation algorithms.
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