Magnetic properties of transition metal fluorides MF2 (M = Mn, Fe, Co, Ni) via high-energy photon diffraction

We present an overview of recent results from nonresonant magnetic diffraction experiments on the antiferromagnetic compounds MnF2, FeF2, CoF2, and NiF2 using high-energy synchrotron radiation of photon energies above 100 keV. New results are presented on the determination of the spin and of the L/S ratio for CoF2 and NiF2. For CoF2, the saturation value of the long-range-ordered pure spin S-z component S-z=1.11(1) is considerably lower than the value S-z=3/2 for the free Co2+ ion. This is in contrast to our results for NiF2, where the full spin of the free transition-metal ion was found, S-z=0.98(1). The temperature dependence of the magnetization in the critical region as well as in the low-temperature region is also presented. For all compounds, Ising behavior is found in the critical regime, whereas the crossover to the low-temperature spin-wave behavior varies. We attribute this to different anisotropies in this series of compounds

DELTA VIRUS COINFECTION DOES NOT INCREASE, BUT HCV COINFECTION INCREASE THE HBSAG LOSS, IN CHRONIC HBV INFECTION

46th Annual Meeting of the European-Association-for-the-Study-of-the-Liver (EASL) -- 2011 -- Berlin, GERMANYWOS: 000297625600400European Assoc Study Liver (EASL

FIBROSIS SHOULD BE INCLUDED INTO THE NONALCOHOLIC STEATOHEPATITIS SCORING SYSTEM FOR BEING MORE PROGNOSTICATIVE

44th Annual Meeting of the European-Association-for-the-Study-of-the-Liver -- APR 22-26, 2009 -- Copenhagen, DENMARKWOS: 000266384701480European Assoc Study Live

ADPedKD: A Global Online Platform on the Management of Children With ADPKD

Background: Autosomal dominant polycystic kidney disease (ADPKD) is the most common monogenic cause of renal failure. For several decades, ADPKD was regarded as an adult-onset disease. In the past decade, it has become more widely appreciated that the disease course begins in childhood. However, evidence-based guidelines on how to manage and approach children diagnosed with or at risk of ADPKD are lacking. Also, scoring systems to stratify patients into risk categories have been established only for adults. Overall, there are insufficient data on the clinical course during childhood. We therefore initiated the global ADPedKD project to establish a large international pediatric ADPKD cohort for deep characterization. Methods: Global ADPedKD is an international multicenter observational study focusing on childhood-diagnosed ADPKD. This collaborative project is based on interoperable Web-based databases, comprising 7 regional and independent but uniformly organized chapters, namely Africa, Asia, Australia, Europe, North America, South America, and the United Kingdom. In the database, a detailed basic data questionnaire, including genetics, is used in combination with data entry from follow-up visits, to provide both retrospective and prospective longitudinal data on clinical, radiologic, and laboratory findings, as well as therapeutic interventions. Discussion: The global ADPedKD initiative aims to characterize in detail the most extensive international pediatric ADPKD cohort reported to date, providing evidence for the development of unified diagnostic, follow-up, and treatment recommendations regarding modifiable disease factors. Moreover, this registry will serve as a platform for the development of clinical and/or biochemical markers predicting the risk of early and progressive disease