Durable, Yet Different: Monarchies in the Arab Spring

Over three years into the Arab Spring, the Middle East is characterized by a striking difference in durability between monarchies and republics. Beyond this difference, some significant gaps within the group of the eight Middle East monarchies have so far been overlooked. Drawing on the existing monarchy research, we first make the case that there were three distinct types of durable monarchies prior to the Arab Spring. Confronted with social and political crises, each type reacted differently to the challenges presented to them after 2011. While five "rentier" and "dynastic" Gulf monarchies (Bahrain, Kuwait, Qatar, Saudi Arabia and the UAE (United Arab Emirates)) mainly rely on material distribution and family rule, the non-oil "linchpins" of Jordan and Morocco, attracting additional external funds, undertook constitutional changes in an attempt at procedural legitimation. The Sultanate of Oman, however, falls in between. This "linchtier" monarchy used modest material cooptation, a selected personal reshuffing at the top of the regime as well as targeted institutional adaptations. We illustrate our findings with similarly structured brief case studies of the three prototypes of Qatar, Jordan and Oman

Protecting Retired Workers from Inflation: Collective Bargaining for Retiree Benefits

The purpose of this article is to explore whether the collective bargaining process in its present form, or with certain modifications, can provide workers with meaningful protection against inflation. Part I evaluates the adequacy of the collective bargaining process by examining the internal dynamics of unions, the interests of employers and the application of the doctrine of fair representation to collective bargaining. After concluding that the current system inadequately protects retirees, Part II proposes alternative methods to strengthen the role of retirees in the collective bargaining process

Labor Market Reform and Price Stability: an Application to the Euro

Abstract This paper studies the effect of labor market reform, in the form of reductions in firing costs and unemployment benefits, on inflation volatility. With this purpose, we build a New Keynesian model with search and matching frictions in the labor market, and estimate it using Euro Area data. Qualitatively, changes in labor market policies alter the volatility of inflation in response to shocks, by affecting the volatility of the three components of real marginal costs (hiring costs, firing costs and wage costs). Quantitatively, we find however that neither policy is likely to have an important effect on inflation volatility, due to the small impact of changes in the volatility of the labor market on inflation dynamics

China’s Asset Management Companies as State Spatial–Temporal Strategy

Chinese authorities created four new asset management companies (AMCs) in 1999, which have since undergone profound transformations that have been influential in China’s contemporary integration into the world market. Conventional interpretations see these powerful AMCs in largely technical and asocial terms. By contrast, we employ a critical geographical analytical framework to understand the transformation of the AMCs as an expression of state spatial–temporal strategy, which involved attempts to create conditions of political economic stability now by displacing the conditions of financial instability and crisis into the future. This strategy does not come without unintended and destabilizing consequences, nor is it without class-based social and political implications

Temporal changes in size distributions of the Southern Ocean diatom Fragilariopsis kerguelensis through high-throughput microscopy of sediment trap samples

Some aspects of the life cycle of the Southern Ocean diatom Fragilariopsis kerguelensis have been investigated previously, but many of its details have not been surveyed in nature. We investigated material from a two-year sediment trap time series by high-throughput imaging and image analysis, looking for morphometric signals of life cycle stages. Valve length distributions appeared close to unimodal but positively (right-) skewed. Size cohorts resulting from synchronized sexual reproduction events were not clearly distinguishable. Nevertheless, based on changes in valve length distributions, we found three general seasonal phases. These corresponded to periods of proliferation (with higher proportions of smaller cells during late spring/early summer), cessation of growth (relative loss of smaller cells during late summer/early autumn), and overwintering (little change in size distributions, with an increased proportion of large cells). We discuss possible causes of these signals, and their relevance to growth, sexual activity and adaption to environmental conditions, such as grazing pressures and the need for an overwintering strategy.This work was supported by the Deutsche Forschungsgemeinschaft (DFG) in the framework of the priority programme 1158 ‘Antarctic Research with comparative investigations in Arctic ice areas’ under grant nr. BE4316/4-1, KA1655/3-1; and by an outgoing scholarship, as well as travel expenses, granted by the Helmholtz Graduate School for Polar and Marine Research (POLMAR). Part of this work was supported by the Australian Government’s Australian Antarctic Science Grant Program under project number 4078, and Macquarie University (A. RigualHernández and L. Armand)

Caspase-3 suppresses diethylnitrosamine-induced hepatocyte death, compensatory proliferation and hepatocarcinogenesis through inhibiting p38 activation

It is critical to understand the molecular mechanisms of hepatocarcinogenesis in order to prevent or treat hepatocellular carcinoma (HCC). The development of HCC is commonly associated with hepatocyte death and compensatory proliferation. However, the role of Caspase-3, a key apoptotic executor, in hepatocarcinogenesis is unknown. In this study, we used Caspase-3-deficient mice to examine the role of Caspase-3 in hepatocarcinogenesis in a chemical (diethylnitrosamine, DEN)-induced HCC model. We found that Caspase-3 deficiency significantly increased DEN-induced HCC. Unexpectedly, Caspase-3 deficiency increased apoptosis induced by DEN and the subsequent compensatory proliferation. Intriguingly, we discovered that Caspase-3 deficiency increased the activation of p38 with and without DEN treatment. Moreover, we demonstrated that TNFα and IL1α stimulated increased activation of p38 in Caspase-3 KO hepatocytes compared with wild-type hepatocytes. Finally, we found that inhibition of p38 by SB202190 abrogated enhanced hepatocyte death, compensatory proliferation and HCC induced by DEN in Caspase-3-deficient mice. Overall, our data suggest that Caspase-3 inhibits chemical-induced hepatocarcinogenesis by suppressing p38 activation and hepatocyte death