Proviral changes in the autophagy status of infected cells differentially affect the outcome of secondary bacterial infection at the cellular level.

(A) A simplified representation of the ordered steps that constitute the autophagy flux (see main text) (AR: autophagy receptors). (B) Summary of available observations on the outcome of secondary bacterial infection of measles virus-infected epithelial cells (Salmonella Typhimurium or Shigella flexneri) or HIV-1-infected macrophages (Mycobacterium tuberculosis).</p