Causal variant identification performance in simulations.

<p>True positive rate as a function of false positive rate in simulations with all forty replicate datasets combined within each configuration (i.e., each dataset has the same cutoff for calling positives and the number of true and false positives are summed over the datasets).</p

BMA 95% central posterior intervals for the number of causal variants in simulations.

<p>Each of the forty replicate datasets within all configurations are shown for BMA A, A/AH and A/D/R. The true value is indicated with a vertical line.</p

Bayesian model averaging formulations and genetic effects.

<p>Model space refers to the possible combinations of and . is the number of variants. Pseudo refers to including a pseudo-SNP with heterozygosity coding for each SNP.</p

Comparison of region-wise posterior association probabilities for BMA A and A/AH.

<p>Similar plot for PLINK is included for reference (region-wise maximum values; one point with values is shown with a cross).</p

BMA 95% central posterior intervals for heritability in simulations.

<p>Each of the forty replicate datasets within all configurations are shown for BMA A, A/AH and A/D/R. The true value is indicated with a vertical line.</p

Posterior distributions of heritability for HDL-C and LDL-C.

<p>Median values are 0.08 for all except for LDL-C BMA A/AH, which has a median of 0.09.</p

Age-adjusted 12-month-prevalence of depressive episodes in the men by quartiles of fish consumption.

<p>Age-adjusted 12-month-prevalence of depressive episodes in the men by quartiles of fish consumption.</p

Age-adjusted 12-month-prevalence of depressive episodes in the women by quartiles of fish consumption.

<p>Age-adjusted 12-month-prevalence of depressive episodes in the women by quartiles of fish consumption.</p