Image_1_The absence of a trade-off between morphological and syntactic complexity.tif

The hypothesis that all languages are equally complex often invokes a trade-off principle, according to which if a language is more complex in one particular domain, it will be simpler in another different domain. In this paper, we use data from WALS to test the existence of a trade-off between two specific domains: morphology and syntax. Contrary to widespread views, we did not find a negative correlation between these two language domains, but in fact a positive correlation. At the same time, this positive correlation seems to be driven by some language families, and it disappears when one considers purely morphological and purely syntactic features only. We discuss these findings in relation to ongoing research about language complexity, and in particular, the effects of factors external to language on linguistic structure.</p

Table_2_The absence of a trade-off between morphological and syntactic complexity.XLSX

The hypothesis that all languages are equally complex often invokes a trade-off principle, according to which if a language is more complex in one particular domain, it will be simpler in another different domain. In this paper, we use data from WALS to test the existence of a trade-off between two specific domains: morphology and syntax. Contrary to widespread views, we did not find a negative correlation between these two language domains, but in fact a positive correlation. At the same time, this positive correlation seems to be driven by some language families, and it disappears when one considers purely morphological and purely syntactic features only. We discuss these findings in relation to ongoing research about language complexity, and in particular, the effects of factors external to language on linguistic structure.</p

Table_1_The absence of a trade-off between morphological and syntactic complexity.XLSX

The hypothesis that all languages are equally complex often invokes a trade-off principle, according to which if a language is more complex in one particular domain, it will be simpler in another different domain. In this paper, we use data from WALS to test the existence of a trade-off between two specific domains: morphology and syntax. Contrary to widespread views, we did not find a negative correlation between these two language domains, but in fact a positive correlation. At the same time, this positive correlation seems to be driven by some language families, and it disappears when one considers purely morphological and purely syntactic features only. We discuss these findings in relation to ongoing research about language complexity, and in particular, the effects of factors external to language on linguistic structure.</p

Image_2_The absence of a trade-off between morphological and syntactic complexity.tif

The hypothesis that all languages are equally complex often invokes a trade-off principle, according to which if a language is more complex in one particular domain, it will be simpler in another different domain. In this paper, we use data from WALS to test the existence of a trade-off between two specific domains: morphology and syntax. Contrary to widespread views, we did not find a negative correlation between these two language domains, but in fact a positive correlation. At the same time, this positive correlation seems to be driven by some language families, and it disappears when one considers purely morphological and purely syntactic features only. We discuss these findings in relation to ongoing research about language complexity, and in particular, the effects of factors external to language on linguistic structure.</p

Table_3_Narrowing the Genetic Causes of Language Dysfunction in the 1q21.1 Microduplication Syndrome.XLSX

<p>The chromosome 1q21.1 duplication syndrome (OMIM# 612475) is characterized by head anomalies, mild facial dysmorphisms, and cognitive problems, including autistic features, mental retardation, developmental delay, and learning disabilities. Speech and language development are sometimes impaired, but no detailed characterization of language problems in this condition has been provided to date. We report in detail on the cognitive and language phenotype of a child who presents with a duplication in 1q21.1 (arr[hg19] 1q21.1q21.2(145,764,455-147,824,207) × 3), and who exhibits cognitive delay and behavioral disturbances. Language is significantly perturbed, being the expressive domain the most impaired area (with significant dysphemic features in absence of pure motor speech deficits), although language comprehension and use (pragmatics) are also affected. Among the genes found duplicated in the child, CDH1L is upregulated in the blood of the proband. ROBO1, a candidate for dyslexia, is also highly upregulated, whereas, TLE3, a target of FOXP2, is significantly downregulated. These changes might explain language, and particularly speech dysfunction in the proband.</p

Table_2_Narrowing the Genetic Causes of Language Dysfunction in the 1q21.1 Microduplication Syndrome.XLSX

<p>The chromosome 1q21.1 duplication syndrome (OMIM# 612475) is characterized by head anomalies, mild facial dysmorphisms, and cognitive problems, including autistic features, mental retardation, developmental delay, and learning disabilities. Speech and language development are sometimes impaired, but no detailed characterization of language problems in this condition has been provided to date. We report in detail on the cognitive and language phenotype of a child who presents with a duplication in 1q21.1 (arr[hg19] 1q21.1q21.2(145,764,455-147,824,207) × 3), and who exhibits cognitive delay and behavioral disturbances. Language is significantly perturbed, being the expressive domain the most impaired area (with significant dysphemic features in absence of pure motor speech deficits), although language comprehension and use (pragmatics) are also affected. Among the genes found duplicated in the child, CDH1L is upregulated in the blood of the proband. ROBO1, a candidate for dyslexia, is also highly upregulated, whereas, TLE3, a target of FOXP2, is significantly downregulated. These changes might explain language, and particularly speech dysfunction in the proband.</p

Table_1_Narrowing the Genetic Causes of Language Dysfunction in the 1q21.1 Microduplication Syndrome.XLSX

<p>The chromosome 1q21.1 duplication syndrome (OMIM# 612475) is characterized by head anomalies, mild facial dysmorphisms, and cognitive problems, including autistic features, mental retardation, developmental delay, and learning disabilities. Speech and language development are sometimes impaired, but no detailed characterization of language problems in this condition has been provided to date. We report in detail on the cognitive and language phenotype of a child who presents with a duplication in 1q21.1 (arr[hg19] 1q21.1q21.2(145,764,455-147,824,207) × 3), and who exhibits cognitive delay and behavioral disturbances. Language is significantly perturbed, being the expressive domain the most impaired area (with significant dysphemic features in absence of pure motor speech deficits), although language comprehension and use (pragmatics) are also affected. Among the genes found duplicated in the child, CDH1L is upregulated in the blood of the proband. ROBO1, a candidate for dyslexia, is also highly upregulated, whereas, TLE3, a target of FOXP2, is significantly downregulated. These changes might explain language, and particularly speech dysfunction in the proband.</p