Studies of long-term weathering of aluminium in the atmosphere

Much information is available on the short- and mid-term atmospheric corrosion of aluminium, in the form of corrosion data (mass loss and pit penetration) as a function of atmosphere type, corrosion mechanisms, environmental variables, surface orientation, etc. However, information on the atmospheric corrosion of aluminium over longer tune periods (10-20 years) is scarce, despite its great usefulness. Furthermore, few studies have focused on the morphology of corrosion product layers. This paper reports corrosion data and morphology of corrosion products formed on aluminium panels after long-term exposure (13-16 years) in various types of atmospheres in Spain: rural, urban, industrial and marine. © 2007 Elsevier Ltd. All rights reserved.Peer Reviewe

Effect of Aprotinin on Liver Injury after Transplantation of Extended Criteria Donor Grafts in Humans: A Retrospective Propensity Score Matched Cohort Analysis

The number of patients awaiting liver transplantation still widely exceeds the number of donated organs available. Patients receiving extended criteria donor (ECD) organs are especially prone to an aggravated ischemia reperfusion syndrome during liver transplantation leading to massive hemodynamic stress and possible impairment in organ function. Previous studies have demonstrated aprotinin to ameliorate reperfusion injury and early graft survival. In this single center retrospective analysis of 84 propensity score matched patients out of 274 liver transplantation patients between 2010 and 2014 (OLT), we describe the association of aprotinin with postreperfusion syndrome (PRS), early allograft dysfunction (EAD: INR 1,6, AST/ALT > 2000 within 7–10 days) and recipient survival. The incidence of PRS (52.4% vs. 47.6%) and 30-day mortality did not differ (4.8 vs. 0%; p = 0.152) but patients treated with aprotinin suffered more often from EAD (64.3% vs. 40.5%, p = 0.029) compared to controls. Acceptable or poor (OR = 3.3, p = 0.035; OR = 9.5, p = 0.003) organ quality were independent predictors of EAD. Our data do not support the notion that aprotinin prevents nor attenuates PRS, EAD or mortality