Additional file 3: Table S3. of Impact of earplugs and eye mask on sleep in critically ill patients: a prospective randomized study

Main sleep characteristics in patient subgroups according to whether patients wore earplugs all night long. (DOC 33 kb

MOESM1 of Diaphragm function and weaning from mechanical ventilation: an ultrasound and phrenic nerve stimulation clinical study

Additional file 1. Full description of the Methods. Figure E1. Flow chart of the patients

Diaphragmatic contractile properties.

<p>Diaphragmatic contractile properties.</p

Prolonged mechanical ventilation worsens sepsis-induced diaphragmatic dysfunction in the rat - Fig 1

<p><b>Maximal tetanic force (Panel A), maximal twitch force (Panel B) and force-frequency relationship (Panel C) of the diaphragm in the four experimental groups.</b> Control = spontaneous ventilation without endotoxemia; MV = mechanical ventilation without sepsis; SV-LPS = endotoxemia with spontaneous ventilation; MV-LPS = endotoxemia with mechanical ventilation. Results are presented in the form of box plots. Boxes are drawn between the first and third quartiles of the distribution, black bars indicate the median, and whiskers indicate the minimum and maximum values, n = 8 per group. *: P<0.05 versus Control; †: P<0.05 versus MV; $: P< 0.05 versus SV-LPS.</p

Total force (TF)-velocity (V) curves in control (n = 12) and diabetic (n = 12) diaphragms.

<p>Panel A: absolute values were used to determine the peak power output (E_max). Panel B: the normalized TF and V values were used to determine the normalized peak power output and the curvature (G) of the F-V curve. Diabetic diaphragms were characterized by a significant increase in maximum TF (TF_max) and a significant decrease in maximum shortening velocity (V_max) which resulted in non significant (NS) changes in E_max and G.</p

Mean arterial blood pressure and arterial blood gases at the end of the 12-h protocol.

<p>Mean arterial blood pressure and arterial blood gases at the end of the 12-h protocol.</p

Prolonged mechanical ventilation worsens sepsis-induced diaphragmatic dysfunction in the rat - Fig 4

<p><b>Relationship between maximum isometric tetanic force of the diaphragm and tumor necrosis factor (TNF)-α (Panel A), interleukin (IL)-1β (Panel B) and IL-6 (Panel C) protein concentration in the diaphragm (left panels) and in the plasma (right panels).</b> Correlation determined by Spearman test.</p

Prolonged mechanical ventilation worsens sepsis-induced diaphragmatic dysfunction in the rat - Fig 3

<p><b>Tumor necrosis factor (TNF)-α (Panel A), interleukin (IL)-1β (Panel B) and IL-6 (Panel C) protein concentration in the diaphragm (left panels) and in the plasma (right panels) in the four experimental groups.</b> Control = spontaneous ventilation without endotoxemia; MV = mechanical ventilation without sepsis; SV-LPS = endotoxemia with spontaneous ventilation; MV-LPS = endotoxemia with mechanical ventilation. Results are presented in the form of box plots as well as individual values. Boxes are drawn between the first and third quartiles of the distribution, black bars indicate the median, and whiskers indicate the minimum and maximum values, n = 8 per group. *: P<0.05 versus Control; †: P<0.05 versus MV; $: P< 0.05 versus SV-LPS.</p

Prolonged mechanical ventilation worsens sepsis-induced diaphragmatic dysfunction in the rat - Fig 2

<p><b>Proportion of diaphragm myofibers in which intramyocellular fat droplets was observed (panel A) and diaphragm cross-sectional area (<i>CSA</i>) of type I (panel B), type IIa, (panel C) and type IIx fibres (panel D) in the four experimental groups.</b> ORO = Oil Red O; Control = spontaneous ventilation without endotoxemia; MV = mechanical ventilation without sepsis; SV-LPS = endotoxemia with spontaneous ventilation; MV-LPS = endotoxemia with mechanical ventilation. Results are presented in the form of box plots as well as individual values. Boxes are drawn between the first and third quartiles of the distribution, black bars indicate the median, and whiskers indicate the minimum and maximum values, n > 4 per group. *: P<0.05 versus Control; †: P<0.05 versus MV.</p

MOESM1 of Removal of totally implanted venous access ports for suspected infection in the intensive care unit: a multicenter observational study

Additional file 1: Table 1. Complementary microbiological findings in patients with TIVAP (totally implanted venous-access ports) related infections. Table E2. Complementary microbiological findings in patients without TIVAP (totally implanted venous-access ports) related infections. Table E3. Variables associated with ICU mortality (univariate analysis). Figure E1. Flow chart of the patients. ICU: intensive care unit; TIVAP: totally implanted venous access ports