Effect of the annealing process on the properties of ZnO thin films prepared by the sol-gel method

This work aims to study the influence of shock/ gradual annealing temperature on the properties of ZnO thin film with different film thickness. ZnO thin films have been prepared on glass substrates using a sol-gel method deposing by dip-coating technique. Zinc acetate dihydrate, monoethanolamine, and 2-methoxyethanol were used as starting materials, stabilizer, and solvent respectively. The X-ray diffraction patterns show that all films have the wurtzite structure and the (002) diffraction peak dominated in all of them. The ZnO films with a higher thickness and annealed under thermal shock, show higher peaks intensity and lower grain size compared to the films annealed by gradually increasing temperature. Scanning electron microscopy images confirmed the results of DRX. The best electrical and optical properties have been obtained for the samples with higher thicknesses and annealed by heat shock. ZnO thin films show a resistance value of 1,296 Ω.cm and a band gap value of 3.245 eV with high transmittance (>90%)

Fanconi anemia in Tunisia: high prevalence of group�A and identification of new FANCA mutations

Appeal From the Final Judgment Of The Second Judicial District Court of Davis County, The Honorable Douglas L. Cornaby Presidin

Mutation spectrum of glycogen storage disease type Ia in Tunisia: implication for molecular diagnosis.

International audienceGlycogen storage disease type Ia (GSD Ia; OMIM 232200) is an autosomal recessive disorder of glycogen metabolism caused by a deficiency of the microsomal glucose-6-phosphatase (G6Pase). It is characterized by short stature, hepatomegaly, hypoglycaemia, hyperuricaemia, and lactic acidaemia. Various mutations have been reported in the G6Pase gene (G6PC). In order to determine the mutation spectrum in Tunisia, we performed mutation analysis in 22 Tunisian type I glycogen storage disease (GSD I) patients belonging to 18 unrelated families. All patients were clinically classified as GSD Ia. The R83C mutation was found to be the major cause of GSD Ia, accounting for 24 of 36 mutant alleles (66.6%), The R170Q mutation was the second most frequent mutation; it accounts for 10 of 36 mutant alleles (27.7%). The R83C and R170Q mutations could be rapidly detected by PCR/RFLP. Since the majority of Tunisian patients carried R83C and/or R170Q mutations, we propose direct screening of these mutations as a rapid, valuable and noninvasive tool for diagnosis of GSD Ia in Tunisian as well as in Northern African populations