Cohen–Macaulay polymatroidal ideals

AbstractAll Cohen–Macaulay polymatroidal ideals are classified. The Cohen–Macaulay polymatroidal ideals are precisely the principal ideals, the Veronese ideals, and the square-free Veronese ideals

On certain equidimensional polymatroidal ideals

The class of equidimensional polymatroidal ideals are studied. In particular, we show that an unmixed polymatroidal ideal is connected in codimension one if and only if it is Cohen-Macaulay. Especially a matroidal ideal is connected in codimension one precisely when it is a squarefree Veronese ideal. As a consequence we indicate that for polymatroidal ideals, the Serre's condition $(S_n)$ for some $n\geq 2$ is equivalent to Cohen-Macaulay property. We also give a classification of generalized Cohen-Macaulay polymatroidal ideals.Comment: To appear in Manuscripta Mathematic

Generalizing the Borel property

We introduce the notion of Q-Borel ideals: ideals which are closed under the Borel moves arising from a poset Q. We study decompositions and homological properties of these ideals, and offer evidence that they interpolate between Borel ideals and arbitrary monomial ideals.Comment: 19 pages, 1 figur

European Journal of Combinatorics Index, Volume 27

BACKGROUND: Diabetes is an inflammatory condition associated with iron abnormalities and increased oxidative damage. We aimed to investigate how diabetes affects the interrelationships between these pathogenic mechanisms. METHODS: Glycaemic control, serum iron, proteins involved in iron homeostasis, global antioxidant capacity and levels of antioxidants and peroxidation products were measured in 39 type 1 and 67 type 2 diabetic patients and 100 control subjects. RESULTS: Although serum iron was lower in diabetes, serum ferritin was elevated in type 2 diabetes (p = 0.02). This increase was not related to inflammation (C-reactive protein) but inversely correlated with soluble transferrin receptors (r = - 0.38, p = 0.002). Haptoglobin was higher in both type 1 and type 2 diabetes (p &lt; 0.001) and haemopexin was higher in type 2 diabetes (p &lt; 0.001). The relation between C-reactive protein and haemopexin was lost in type 2 diabetes (r = 0.15, p = 0.27 vs r = 0.63, p &lt; 0.001 in type 1 diabetes and r = 0.36, p = 0.001 in controls). Haemopexin levels were independently determined by triacylglycerol (R(2) = 0.43) and the diabetic state (R(2) = 0.13). Regarding oxidative stress status, lower antioxidant concentrations were found for retinol and uric acid in type 1 diabetes, alpha-tocopherol and ascorbate in type 2 diabetes and protein thiols in both types. These decreases were partially explained by metabolic-, inflammatory- and iron alterations. An additional independent effect of the diabetic state on the oxidative stress status could be identified (R(2) = 0.5-0.14). CONCLUSIONS: Circulating proteins, body iron stores, inflammation, oxidative stress and their interrelationships are abnormal in patients with diabetes and differ between type 1 and type 2 diabetes</p