Aartsma-Rus AM

Doorenweerd N

Hendriksen JGM

Kaliyaperumal R

Kan HE

Lelieveldt BPF

Mahfouz A

Niks EH

Reinders MJT

T' Hoen PAC

van Putten M

Verschuuren JJGM

A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has not been fixed in the paper

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1SCIENTIFIC RePoRTs |  (2018) 8:4058  | DOI:10.1038/s41598-018-22154-7www.nature.com/scientificreportsAuthor Correction: Timing and localization of human dystrophin isoform expression provide insights into the cognitive phenotype of Duchenne muscular dystrophyNathalie Doorenweerd1,2,6,9, Ahmed Mahfouz  3,4, Maaike van Putten5, Rajaram Kaliyaperumal5, Peter A. C. t’ Hoen5, Jos G. M. Hendriksen7,8, Annemieke M. Aartsma-Rus5, Jan J. G. M. Verschuuren  2, Erik H. Niks2, Marcel J. T. Reinders  4, Hermien E. Kan1,6 & Boudewijn P. F. Lelieveldt  3,4Correction to: Scientific Reports https://doi.org/10.1038/s41598-017-12981-5, published online 03 October 2017In this Article, some of the wording in the Results section is ambiguous and should be corrected as follows:“In contrast to previous reports18,29, the Purkinje isoform Dp427p was virtually absent in the brain throughout development, with expression levels even lower than muscle dystrophin Dp427m.”should read:“In contrast to previous reports on mouse brain18, 29, the Purkinje isoform Dp427p was virtually absent in human brain throughout development, with expression levels even lower than muscle dystrophin Dp427m.”In addition“Yet in line with previous studies30,31, Dp427p was expressed in the mouse cerebellum and not in the mouse cer-ebral cortex.”should read:“Yet in line with previous studies29,30,31, Dp427p was expressed in the mouse cerebellum and not in the mouse cerebral cortex.”1C.J. Gorter Center for High Field MRI, Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands. 2Department of Neurology, Leiden University Medical Center, Leiden, The Netherlands. 3Division of Image Processing, Department of Radiology, Leiden University Medical Center, Leiden, The Netherlands. 4Delft Bioinformatics Lab, Delft University of Technology, Delft, The Netherlands. 5Department of Human Genetics, Leiden University Medical Center, Leiden, The Netherlands. 6Leiden Institute for Brain and Cognition, Leiden University, Leiden, The Netherlands. 7Department of Neurological Learning Disabilities, Kempenhaeghe Epilepsy Center, Heeze, The Netherlands. 8Department of Neurology, Maastricht University Medical Center, Maastricht, The Netherlands. 9John Walton Muscular Dystrophy Research Centre, Newcastle University, Newcastle Upon Tyne, United Kingdom. Nathalie Doorenweerd and Ahmed Mahfouz contributed equally to this work. Correspondence and requests for materials should be addressed to N.D. (email: N.Doorenweerd@lumc.nl)Published: xx xx xxxxOPENwww.nature.com/scientificreports/2SCIENTIFIC RePoRTs |  (2018) 8:4058  | DOI:10.1038/s41598-018-22154-7Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Cre-ative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not per-mitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. © The Author(s) 2018

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Erratum: Author Correction: Timing and localization of human dystrophin isoform expression provide insights into the cognitive phenotype of Duchenne muscular dystrophy (Scientific reports (2017) 7 1 (12575))

Erratum: Author Correction: Timing and localization of human dystrophin isoform expression provide insights into the cognitive phenotype of Duchenne muscular dystrophy (Scientific reports (2017) 7 1 (12575))

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