Exposure to pesticides is very common world-wide, and is broadly
known the acute toxic effects to humans of pesticides following a high
dose exposure; however, knowledge about chronic low-dose adverse
effects to specific pesticides is more limited. Reproductive functions
can be affected, with birth defects, impaired fecund ability, infertility
and altered growth. This paper will focus on the deleterious effects
that may appear in the offspring, during early and later stages of life,
after prenatal exposure to insecticides, not only on women with direct
exposure but also on subjects with indirect exposure such as consumers
or residents of rural communities. Prenatal exposure to pesticides could
alter normal fetal development and could threaten future welfare. The
main changes observed in prenatal exposure to organophosphates
are alterations in the central nervous system, in the metabolic and
hormonal system as endocrine disruptor and over the birth outcomes.
Carbamates may cause developmental delay when the applications
of carbamates during pregnancy were nearby the home. Pyrethroids
are among the most frequently used pesticides and account for more
than one-third of the insecticides currently marketed in the world. For
this reason the prenatal exposition used to be for long periods causing
clinical, biochemical and neurological changes
