Peritraumatic dissociation, e.g. an out-of-body experience (OBE) during a traumatic event, is an established risk factor for post-traumatic stress disorder (PTSD). However, its neural basis is not yet well understood. Experimental neuroscience and lesion studies suggest that disrupted brain function in the temporo-parietal junction (TPJ), a critical hub for multisensory integration, is associated with such anomalous bodily experiences. To explore neural markers of disrupted embodiment, we investigated how a transcranial magnetic stimulation (TMS)-induced temporary inhibition of the TPJ affects 1) dissociative experiencing including susceptibility to OBEs, and 2) the brain network of body awareness in healthy participants. First, we tested the paradigm to manipulate bodily self-consciousness called the OBE illusion and threat-evoked changes in heart rate as objective marker of OBEs. Secondly, functional connectivity ! patterns and the OBE paradigm were assessed before and after TMS (n = 26) vs. sham TMS (n = 26). It was expected that a virtual lesion at the TPJ increases self-report dissociation, elicit stronger OBE sensations, and disrupt the brain network of body awareness (e.g. insula, posterior parietal cortex, premotor cortex). Results will be discussed within the framework of dissociative processing