Pathophysiology of the metabolic syndrome: both insulin resistance and lipid overflow contribute to MetS evolution

Abstract

IL-6, interleukin-6; MAP, mitogen-activated protein; TNF-α, tumor necrosis factor-alpha.<p><b>Copyright information:</b></p><p>Taken from "Metabolic syndrome in rheumatic diseases: epidemiology, pathophysiology, and clinical implications"</p><p>http://arthritis-research.com/content/10/3/207</p><p>Arthritis Research & Therapy 2008;10(3):207-207.</p><p>Published online 8 May 2008</p><p>PMCID:PMC2483433.</p><p></p