<p>The <i>motA</i> mutations are predicted to result in non-functional proteins. A missense mutation at base 262 (T1, Type B) resulted in an A to P mutation at amino acid 88; a nonsense mutation at base 612 (T3, Type D) resulted in a premature truncation; a duplication of 49 bp created a direct repeat within <i>motA</i> and led to the replacement of the last 73 residues in the C-terminus with a sequence of 39 new residues that are highlighted (T108, Type C); and a deletion at base 64 resulting in a truncated protein (T158, Type A).</p
