Average Size and size distribution of the used NP. Figure S2. Measurement of cell viability. Figure S3. Exposure to NP reactivates lytic virus in persistently infected cells in vitro in a dose dependent manner. Figure S4. Exposure to NP reactivates lytic virus in persistently infected cells independently of the particle aspect ratio. Figure S5. Short-time exposure of latently infected mice to NP differentially regulates gene expression in whole lung tissue cells independently of the particle aspect ratio. Figure S6. Confirmation of gene expression data by real-time quantitative PCR for selected genes. Figure S7. Exposure of latently infected mice to CNP leads to an increase in glycerophospholipids. Figure S8. Exposure of latently infected mice to DWCNT leads to an increase in glycerophospholipids. Figure S9. Exposure of persistently infected cells to TiO2 NP or DEP has differential effects on virus reactivation in vitro. Table S1. Gene expression values of selected genes (PDF 1767Â kb
