In this study we especially associate eye movements in tracking a visual target with dysfunctions caused by symptoms of intoxication in both frontal and parietal lobes. Visual targets used in this study were of apparent movements and of periodical triangular wave form on display. Two moving patterns of a visual target were provided for the experiment One is designed so as to demand a perceptual task, and another for a cognitive task. Each subject carried out the tracking process under three conditions: of no-alcohol, of low and high concentrations. In the case of an intoxicated subject, the latency and the duration of saccade are more prolonged, the mean velocity is reduced, and the amplitude is enlarged, especially in the case of tracking a visual target with a moving pattern which requires a cognitive task. There is no difference between the two tasks on the neural network for setting up necessary parameters to start off a saccade, but the quality of eye tracking is definitely affected by high quantities of alcohol. This corroborates Terao\u27s hypothesis that the neural network can compensate for the damage only at one area in the brain, but does not cooperate well if damage exists in multiple areas of the brain