Data of PWTL and PWMT values in the study. (XLS 19 kb

Guodong He

Hai Lin

Jianping Yang

Jiehao Sun

Wendong Lin

BMC Anesthesiology

Data of ratio of grey values by Î˛-actin in the western blot. (XLS 19 kb

Jiehao Sun (3519239)

Hai Lin (163369)

Guodong He (3770947)

Wendong Lin (3770950)

Jianping Yang (322369)

The Francis Crick Institute

Additional file 2: of Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

Data of PWTL and PWMT values in the previous model tests from the referred literature. (TIF 812 kb

Additional file 4: of Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

The PWTL and PWMT tests in the previous model tests from the referred literature (A and B) and the ones after the NMDA antagonists therapy in this study (C and D). (XLS 18 kb

Additional file 3: of Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

Springer - Publisher Connector

Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

Additional file 1: of Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

https://figshare.com/articles/Additional_file_4_of_Magnesium_sulphate_attenuate_remifentanil-induced_postoperative_hyperalgesia_via_regulating_tyrosine_phosphorylation_of_the_NR2B_subunit_of_the_NMDA_receptor_in_the_spinal_cord/4680010

Magnesium sulphate attenuate remifentanil-induced postoperative hyperalgesia via regulating tyrosine phosphorylation of the NR2B subunit of the NMDA receptor in the spinal cord

Abstract

Similar works

Full text

Available Versions

The Francis Crick Institute

The Francis Crick Institute

The Francis Crick Institute

Springer - Publisher Connector

Springer - Publisher Connector

The Francis Crick Institute