CORE
🇺🇦
make metadata, not war
Services
Services overview
Explore all CORE services
Access to raw data
API
Dataset
FastSync
Content discovery
Recommender
Discovery
OAI identifiers
OAI Resolver
Managing content
Dashboard
Bespoke contracts
Consultancy services
Support us
Support us
Membership
Sponsorship
Community governance
Advisory Board
Board of supporters
Research network
About
About us
Our mission
Team
Blog
FAQs
Contact us
Helikobakter pylori enfeksiyonu B12 eksikliğinin gerçekten bir nedeni midir?
Authors
Yusuf Aydın
Gökhan Celbek
+4 more
Funda Ceran
Seyfettin Köklü
Yasemin Ateş Tütüncü
Mehmet Yıldız
Publication date
1 January 2011
Publisher
Abstract
Aim: Aim of this study is to determine Helicobacter pylori infection rate in Vitamin B12 deficiency and determine whether H.pylori infection is really actual cause of Vitamin B12 deficiency. Material and Methods: We evaluated 40 vitamin B12 deficient patients and 25 healthy subjects. Those patients did not have known cause of vitamin B12 deficiency. We performed endoscopic evaluation and pathological examination for H.pylori. We determined 50% atrophic gastritis in vitamin B12 deficient group, but we did not observe any atrophic gastritis in the control group endoscopically. Results: There were H.pylori positivity in 11/40 (27.5%) in the patients group but 16/25 (64) in the control group in pathological examination (p<0.01). Pathological examination of patient group also illustrated that 11 patients had mild atrophy in 11 (27.5%), 5 had moderate atrophy (12.5%) and 3 patient (7.5%) had severe atrophy. In contrast, 3 patients had only mild atrophy (12%) in the control group. We also found that H.pylori (-) 17/29 (58.6%) patients and H.pylori (+) 3/11 (27.3%) patients had atrophic gastritis in gastroscopic evaluation. Conclusion: Patients who have vitamin B12 deficiency had lower ratio of H.pylori in gastric mucosa. On the other hand, the patient group had higher ratio of atrophic gastritis in pathological and gastroscopic examination. H.pylori positivity had been decreased progressively with aging in patients group. H.pylori has disappeared in following years as a result of atrophy in gastric mucosa. We conluded that atrophic mucosa is not suitable for colonization of H.pylori infection. © 2011 Düzce Medical Journal
Similar works
Full text
Available Versions
Duzce University Open Access
See this paper in CORE
Go to the repository landing page
Download from data provider
oai:acikerisim.duzce.edu.tr:20...
Last time updated on 14/05/2020
Duzce University Open Access
See this paper in CORE
Go to the repository landing page
Download from data provider
oai:acikerisim.duzce.edu.tr:20...
Last time updated on 14/05/2020