∙ The authors have no financial conflicts of interest. © Copyright: Yonsei University College of Medicine 2012 This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Licens

Dai, FeiFeng

Mao, ZhiFu

Wu, ZhiYong

Xia, Jun

Zhu, ShaoPing

English

PubMed

Feifeng Dai

Zhifu Mao

Jun Xia

Shaoping Zhu

Zhiyong Wu

CiteSeerX

Fluoxetine Protects against Big Endothelin-1 Induced Anti-Apoptosis by Rescuing Kv1.5 Channels in Human Pulmonary Arterial Smooth Muscle Cells

[Ca2+]i inhibition of K+ channels in canine pulmonary artery. Novel mechanism for hypoxiainduced membrane depolarization. Circ Res

848 ulation of pulmonary artery smooth muscle cell proliferation and apoptosis: pharmacotherapeutic implications.

An abnormal mitochondrial-hypoxia inducible factor-1alpha-Kv channel pathway disrupts oxygen sensing and triggers pulmonary arterial hypertension in fawn hooded rats: similarities to human pulmonary arterial hypertension.

Augmented K(+) currents and mitochondrial membrane depolarization in pulmonary artery myocyte apoptosis.

Big endothelin-1 and endothelin-1 plasma levels are correlated with the severity of primary pulmonary hypertension.

Bone morphogenetic protein-2 upregulates expression and function of voltage-gated K+ channels in human pulmonary artery smooth muscle cells.

Continuous fluoxetine administration prevents recurrence of pulmonary arterial hypertension and prolongs survival in rats.

Dichloroacetate prevents and reverses pulmonary hypertension by inducing pulmonary artery smooth muscle cell apoptosis.

Fluoxetine protects against monocrotaline-induced pulmonary arterial hypertension: potential roles of induction of apoptosis and upregulation of Kv1.5 channels in rats.

Function of Kv1.5 channels and genetic variations of KCNA5 in patients with idiopathic pulmonary arterial hypertension.

Impaired voltage gated potassium channel responses in a fetal lamb model of persistent pulmonary hypertension of the newborn.

Inhibition of voltage-gated K(+) currents by endothelin-1 in human pulmonary arterial myocytes.

Potassium channels and vascular proliferation.

Pulmonary arterial hypertension.

Serotonin transporter inhibitors protect against hypoxic pulmonary hypertension.

Sildenafil inhibits human pulmonary artery smooth muscle cell proliferation by decreasing capacitative Ca2+ entry.

Sustained membrane depolarization and pulmonary artery smooth muscle cell proliferation.

The endothelin system in pulmonary hypertension.

The nuclear factor of activated T cells in pulmonary arterial hypertension can be therapeutically targeted.

ftp://ftp.ncbi.nlm.nih.gov/pub/pmc/b9/78/ymj-53-842.PMC3381489.pdf

Fluoxetine Protects against Big Endothelin-1 Induced Anti-Apoptosis by Rescuing Kv1.5 Channels in Human Pulmonary Arterial Smooth Muscle Cells

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