Br  ning

CHARGE

d'Albis

Grefte

H. Wang

Hirsch

I. H. Lee

J. Du

Madhan

Mourkioti

Musar  

S. Modi

TSCHOPE

Vasudevan

Vignaud

W. E. Mitch

Wang

Wijesekara

X. Wang

Z. Hu

English

PubMed

Crossref

PTEN Inhibition Improves Muscle Regeneration in Mice Fed a High-Fat Diet

A muscle -speciﬁc insulin receptor knockout exhibits features of the metabolic syndrome of NIDDM without altering glucose tolerance. Mol Cell

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Cellular and molecular regulation of muscle regeneration.

Chemokine CXCL16 regulates neutrophil and macrophage inﬁltration into injured muscle, promoting muscle regeneration.

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Diabetes provides an unfavorable environment for muscle mass and function after muscle injury in mice.

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Exercise ameliorates chronic kidney disease-induced defects in muscle protein metabolism and progenitor cell function.

Farnesoid X receptor modulates renal lipid metabolism, ﬁbrosis, and diabetic nephropathy.

High glucose stimulates proliferation and collagen type I synthesis in renal cortical ﬁbroblasts: mediation by autocrine activation of TGF-beta.

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PTEN expression contributes to the regulation of muscle protein degradation in diabetes.

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Suppression of PTEN expression by NF-kB prevents apoptosis.

Targeted ablation of IKK2 improves skeletal muscle strength, maintains mass, and promotes regeneration.

Targeted expression of IGF-1 transgene to skeletal muscle accelerates muscle and motor neuron regeneration.

http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2874691

PTEN Inhibition Improves Muscle Regeneration in Mice Fed a High-Fat Diet

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