Eric R. Hugo

Heard DJ

J. Wesley Alexander

Jean Loftus

Jessica G. Woo

Masuno H

Nira Ben-Jonathan

Terry D. Brandebourg

Environmental Health Perspectives

English

PubMed

Crossref

Bisphenol A at Environmentally Relevant Doses Inhibits Adiponectin Release from Human Adipose Tissue Explants and Adipocytes

Adiponectin and adiponectin receptors in insulin resistance, diabetes, and the metabolic syndrome.

Adiponectin is present in human milk and is associated with maternal factors.

Adiponectin—a key adipokine in the metabolic syndrome. Diabetes Obes Metab 8:264–280.

Adiponectin—journey from an adipocyte secretory protein to biomarker of the metabolic syndrome.

Binding and activation of the seventransmembrane estrogen receptor GPR30 by environmental estrogens: a potential novel mechanism of endocrine disruption.

Bisphenol A accelerates terminal differentiation of 3T3-L1 cells into adipocytes through the phosphatidylinositol 3-kinase pathway. Toxicol Sci 84:319–327.

Bisphenol A affects glucose transport in mouse 3T3-F442A adipocytes.

Bisphenol A in combination with insulin can accelerate the conversion of 3T3-L1 ﬁbroblasts to adipocytes.

Bisphenol A is released from polycarbonate drinking bottles and mimics the neurotoxic actions of estrogen in developing cerebellar neurons. Toxicol Lett 176:149–156.

Bisphenol-A and chlorinated derivatives in adipose tissue of women.

Comparison of the release of adipokines by adipose tissue, adipose tissue matrix, and adipocytes from visceral and subcutaneous abdominal adipose tissues of obese humans.

Demonstration of estrogen receptor subtypes alpha and beta in human adipose tissue: inﬂuences of adipose cell differentiation and fat depot localization. Mol Cell Endocrinol 182:27–37.

Direct evidence revealing structural elements essential for the high binding ability of bisphenol A to human estrogen-related receptor-γ. Environ Health Perspect 116:32–38.

Effect of postmenopause and hormone replacement therapy on serum adiponectin levels.

Endocrine disruptors and the obesity epidemic. Toxicol Sci 76:247–249.

Estrogen receptor null mice: what have we learned and where will they lead us? Endocr Rev 20:358–417.

Estrogen-related receptors as emerging targets in cancer and metabolic disorders. Curr Top Med Chem 6:203–215.

Evidence for functional estrogen receptors alpha and beta in human adipose cells: regional speciﬁcities and regulation by estrogens.

for Biotechnology Information.

Gender differences of oligomers and total adiponectin during puberty: a cross-sectional study of 859 Danish school children.

GPR30: a seven-transmembranespanning estrogen receptor that triggers EGF release. Trends Endocrinol Metab 16:362–367.

Hormesis: U-shaped dose responses and their centrality in toxicology. Trends Pharmacol Sci 22:285–291.

Human ERRγ, a third member of the estrogen receptor-related receptor (ERR) subfamily of orphan nuclear receptors: tissue-speciﬁc isoforms are expressed during development and in the adult. Mol Endocrinol 14:382–392.

Induction of prolactin expression and release in human preadipocytes by cAMP activating ligands.

Interaction of estrogenic chemicals and phytoestrogens with estrogen receptor beta.

Large effects from small exposures. III. Endocrine mechanisms mediating effects of bisphenol A at levels of human exposure. Endocrinology 147:S56–S69.

Low doses of bisphenol A and diethylstilbestrol impair Ca2+ signals in pancreatic alpha-cells through a nonclassical membrane estrogen receptor within intact islets of Langerhans. Environ Health Perspect 113:969–977.

LS14: a novel human adipocyte cell line that produces prolactin.

Measurement of bisphenol A concentrations in human colostrum.

Perinatal exposure to environmental estrogens and the development of obesity. Mol Nutr Food Res 51:912–917.

Problems for risk assessment of endocrine-active estrogenic compounds. Environ Health Perspect 110(suppl 6):925–929.

Relative expression software tool (REST) for group-wise comparison and statistical analysis of relative expression results in real-time PCR. Nucleic Acids Res 30:e36.

Signaling from the membrane via membrane estrogen receptor-alpha: estrogens, xenoestrogens, and phytoestrogens.

The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance. Environ Health Perspect 114:106–112.

The link between abdominal obesity, metabolic syndrome and cardiovascular disease. Nutr Metab Cardiovasc Dis 17:319–326.

What can we learn from rodents about prolactin in humans? Endocr Rev 29:1–41.

http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2599757

Bisphenol A at Environmentally Relevant Doses Inhibits Adiponectin Release from Human Adipose Tissue Explants and Adipocytes

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