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Aberrant Mucin Assembly in Mice Causes Endoplasmic Reticulum Stress and Spontaneous Inflammation Resembling Ulcerative Colitis
Authors
Altmann
An
+92 more
Andresen
Asker
Bertolotti
Bodger
Campbell
Carlstedt
Chad K Heazlewood
Chin Wen Png
Christopher C Goodnow
Corfield
David J Thornton
Delpre
Deng
Devine
Dixon
Donnellan
Douglas Taupin
Dvorak
Dvorak
Fuss
Fuss
Gareth R Price
Gasche
Gasche
Gionchetti
Godl
Gonzalez-Licea
Hanski
Heller
Heller
Herrmann
Hinoda
Hogan
Hong
Hu
Itoh
Jass
Jenkins
Kadivar
Keats A Nelms
Lidell
Longman
Mahida
Majors
Marciniak
Marciniak
Masato Kato
Matthew C Cook
McDermott
McGuckin
McVay
Meusser
Michael A McGuckin
Michiels
Moehle
Nagle
Nancy A Hong
O&apos
Olson
Pahl
Podolsky
Rachel Adams
Rajaraman Eri
Reimund
Richard J Cornall
Rogler
Ron
Sartor
Shaoul
Sharyn B Tauro
Sheehan
Shkoda
Shroyer
Silverberg
Stefan Schreiber
Swallow
Tanya L Crockford
Thornton
Timothy H. J Florin
Trabucchi
Tysk
Tytgat
Vainer
Van der Sluis
van der Waaij
Van Klinken
Velcich
Wang
Williams
Xavier
Xue
Zhang
Publication date
1 January 2008
Publisher
Public Library of Science
Doi
Cite
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on
PubMed
Abstract
Michael McGuckin and colleagues identify two mutations that cause aberrant mucin oligomerization in mice. The resulting phenotype, including endoplasmic reticulum stress, resembles clinical and pathologic features of human ulcerative colitis
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