Abstract no. 0136The mechanism for the development of Complex Regional Pain Syndrome (CRPS) is not yet well understood. The clinical features and recent studies of CRPS have suggested that a combination of exaggerated regional inflammatory responses in injury may contribute. A Chronic Post-ischemia Pain (CPIP) model of CRPS type I was built and it was suggested the pathogenesis of CRPS could be attributed to the involvement of ischemia-reperfusion injury and inflammatory response. In this study, we evaluated the effects of ketamine on CPIP model in Sprague-...postprin
