Spencer S. Eccles Health Sciences Library, University of Utah
Abstract
Traumatic optic neuropathy is a well-recognised consequence of head trauma and frequently results in significant irreversible loss of vision. Previous reports have suggested that up to 10% of patients with craniofacial fractures have traumatic optic neuropathy. Corticosteroids and orbital decompression have previously been advocated as treatment options, although studies have previously shown no significant improvement in visual outcome with steroids and some studies have suggested that steroids may result in increased mortality. There is limited understanding of the exact pathophysiology of traumatic optic neuropathy and of the correlation between injury mechanism, anatomical location and final visual outcome
