Characteristic length of a Holographic Superconductor with dd-wave gap

After the discovery of the $s$-wave and $p$-wave holographic superconductors, holographic models of $d$-wave superconductor have also been constructed recently. We study analytically the perturbation of the dual gravity theory to calculate the superconducting coherence length $\xi$ of the $d$-wave holographic superconductor near the superconducting phase transition point. The superconducting coherence length $\xi$ divergents as $(1-T/T_c)^{-1/2}$ near the critical temperature $T_c$. We also obtain the magnetic penetration depth $\lambda\propto(T_c-T)^{-1/2}$ by adding a small external homogeneous magnetic field. The results agree with the $s$-wave and $p$-wave models, which are also the same as the Ginzburg-Landau theory.Comment: last version, 10 pages, accepted by PR

Proton Mass Decomposition from the QCD Energy Momentum Tensor

We report results on the proton mass decomposition and also on related quark and glue momentum fractions. The results are based on overlap valence fermions on four ensembles of $N_f = 2+1$ DWF configurations with three lattice spacings and three volumes, and several pion masses including the physical pion mass. With fully non-perturbative renormalization (and universal normalization on both quark and gluon), we find that the quark energy and glue field energy contribute 33(4)(4)\% and 37(5)(4)\% respectively in the $\overline{MS}$ scheme at $\mu = 2$ GeV. A quarter of the trace anomaly gives a 23(1)(1)\% contribution to the proton mass based on the sum rule, given 9(2)(1)\% contribution from the $u, d,$ and $s$ quark scalar condensates. The $u,d,s$ and glue momentum fractions in the $\overline{MS}$ scheme are in good agreement with global analyses at $\mu = 2$ GeV.Comment: 10 pages, 6 figure

Ilexonin A Promotes Neuronal Proliferation and Regeneration via Activation of the Canonical Wnt Signaling Pathway after Cerebral Ischemia Reperfusion in Rats

Aims. Ilexonin A (IA), a component of the Chinese medicine Ilex pubescens, has been shown to be neuroprotective during ischemic injury. However, the specific mechanism underlying this neuroprotective effect remains unclear. Methods. In this study, we employed a combination of immunofluorescence staining, western blotting, RT-PCR, and behavioral tests, to investigate the molecular mechanisms involved in IA regulation of neuronal proliferation and regeneration after cerebral ischemia and reperfusion in rodents. Results. Increases in β-catenin protein and LEF1 mRNA and decreases in GSK3β protein and Axin mRNA observed in IA-treated compared to control rodents implicated the canonical Wnt pathway as a key signaling mechanism activated by IA treatment. Furthermore, rodents in the IA treatment group showed less neurologic impairment and a corresponding increase in the number of Brdu/nestin and Brdu/NeuN double positive neurons in the parenchymal ischemia tissue following middle cerebral artery occlusion compared to matched controls. Conclusion. Altogether, our data indicate that IA can significantly diminish neurological deficits associated with cerebral ischemia reperfusion in rats as a result of increased neuronal survival via modulation of the canonical Wnt pathway

Experimental Equilibrium Moisture Content of Wood Under Vacuum

Wood equilibrium moisture content (EMC) was measured under vacuum by an electronic method. A wafer was used to measure EMC using an in-house designed vacuum instrument. EMC at 4 to 100 kPa and temperature from 30 to 90°C were measured. The relationships among temperature, pressure, and EMC were determined, and a diagram of wood EMC was produced. The results showed there are obvious differences between experimental EMC values obtained and theoretical EMC values of other researchers. It is suggested that corrections should be introduced into theoretical models or a new model for the vacuum condition developed

RI/MOM and RI/SMOM renormalization of quark bilinear operators using the overlap fermion

We present the vector, scalar and tensor renormalization constants (RCs) using the overlap fermion on dynamical fermion gauge configurations with either the Domain wall fermion action or HISQ actions, using RI/MOM and RI/SMOM as the intermediate scheme at the lattice spacing $a$ from 0.04 fm to 0.12 fm. The results show that RI/MOM and RI/SMOM can provide consistent renormalization constants under the $\bar{\textrm{MS}}$ scheme, after proper $a^2p^2$ extrapolations. But at $p\sim 2$ GeV, both the RI/MOM and RI/SMOM suffer from nonperturbative effects which cannot be removed by the perturbative matching. The comparison between the results with different sea actions also suggests that the renormalization constant is discernibly sensitive to the lattice spacing but not to the bare gauge coupling in the gauge action.Comment: 21 pages, 15 figure

Molecular Characterization of a Debilitation-Associated Partitivirus Infecting the Pathogenic Fungus Aspergillus flavus

The opportunistic human pathogenic fungus Aspergillus flavus is known to be infected with mycoviruses. In this study, we report a novel mycovirus A. flavus partitivirus 1 (AfPV1) that was originally isolated from the abnormal colonial morphology isolate LD-3-8 of A. flavus. AfPV1 has spherical virus-like particles about 40 nm in diameter, and three double-stranded RNA (dsRNA) segments (dsRNA1, 2, and 3 with lengths of 1.7, 1.4, and 1.1 kbp, respectively) were packaged in the virions. dsRNA1, dsRNA2, and dsRNA3 each contained a single open reading frame and potentially encoded 62, 42, and 32 kDa proteins, respectively. The dsRNA1 encoded protein shows similarity to the RNA-dependent RNA polymerase (RdRp) of partitiviruses, and the dsRNA2 product has no significant similarity to any other capsid protein (CP) in the GenBank databases, beside some homology with the hypothetical “capsid” protein of a few partitiviruses. The dsRNA3 encodes a protein with no similarity to any protein in the GenBank database. SDS-PAGE and polypeptide mass fingerprint-mass spectrum (PMF-MS) analyses indicated that the CP of the AfPV1 was encoded by dsRNA2. Phylogenetic analysis showed that the AfPV1 and relative viruses were found in an unclassified group inside the Partitiviridae family. AfPV1 seems to result in debilitation symptoms, but had no significant effects to murine pathogenicity. These findings provide new insights into the partitiviruses taxonomy and the interactions between viruses and A. flavus