Pulse Shaping Diversity to Enhance Throughput in Ultra-Dense Small Cell Networks

Spatial multiplexing (SM) gains in multiple input multiple output (MIMO) cellular networks are limited when used in combination with ultra-dense small cell networks. This limitation is due to large spatial correlation among channel pairs. More specifically, it is due to i) line-of-sight (LOS) communication between user equipment (UE) and base station (BS) and ii) in-sufficient spacing between antenna elements. We propose to shape transmit signals at adjacent antennas with distinct interpolating filters which introduces pulse shaping diversity eventually leading to improved SINR and throughput at the UEs. In this technique, each antenna transmits its own data stream with a relative offset with respect to adjacent antenna. The delay which must be a fraction of symbol period is interpolated with the pulse shaped signal and generates a virtual MIMO channel that leads to improved diversity and SINR at the receiver. Note that non-integral sampling periods with inter-symbol interference (ISI) should be mitigated at the receiver. For this, we propose to use a fractionally spaced equalizer (FSE) designed based on the minimum mean squared error (MMSE) criterion. Simulation results show that for a 2x2 MIMO and with inter-site-distance (ISD) of 50 m, the median received SINR and throughput at the UE improves by a factor of 11 dB and 2x, respectively, which verifies that pulse shaping can overcome poor SM gains in ultra-dense small cell networks.Comment: Accepted to 17th IEEE International Workshop on Signal Processing Advances in Wireless Communication

Study on Scheduling Techniques for Ultra Dense Small Cell Networks

The most promising approach to enhance network capacity for the next generation of wireless cellular networks (5G) is densification, which benefits from the extensive spatial reuse of the spectrum and the reduced distance between transmitters and receivers. In this paper, we examine the performance of different schedulers in ultra dense small cell deployments. Due to the stronger line of sight (LOS) at low inter-site distances (ISDs), we discuss that the Rician fading channel model is more suitable to study network performance than the Rayleigh one, and model the Rician K factor as a function of distance between the user equipment (UE) and its serving base station (BS). We also construct a cross-correlation shadowing model that takes into account the ISD, and finally investigate potential multi-user diversity gains in ultra dense small cell deployments by comparing the performances of proportional fair (PF) and round robin (RR) schedulers. Our study shows that as network becomes denser, the LOS component starts to dominate the path loss model which significantly increases the interference. Simulation results also show that multi-user diversity is considerably reduced at low ISDs, and thus the PF scheduling gain over the RR one is small, around 10% in terms of cell throughput. As a result, the RR scheduling may be preferred for dense small cell deployments due to its simplicity. Despite both the interference aggravation as well as the multi-user diversity loss, network densification is still worth it from a capacity view point.Comment: 6 pages, 7 figures, Accepted to IEEE VTC-Fall 2015 Bosto

Stimulated quantum phase slips from weak electromagnetic radiations in superconducting nanowires

We study the rate of quantum phase slips in an ultranarrow superconducting nanowire exposed to weak electromagnetic radiations. The superconductor is in the dirty limit close to the superconducting-insulating transition, where fluxoids move in strong dissipation. We use a semiclassical approach and show that external radiation stimulates a significant enhancement in the probability of quantum phase slips. This can help to outline a new type of detector for microwave to submillimetre radiations based on stimulated quantum phase slip phenomenon.Comment: 10 pages, 9 figure

The Fear of COVID-19 Scale: A meta-Analytic structural equation modeling approach

The widespread administration and multiple validations of the Fear of Covid-19 Scale (FCV-19S) in different languages have highlighted the controversy over its underlying structure and the resulting reliability index. In the present study, a meta-analysis based on structural equation modeling (MASEM) was conducted to assess the internal structure of the 7-item, 5-point Likert-type FCV-19S version, estimate an overall reliability index from the underlying model that best reflected the internal structure (one τ-equivalent factor, one congeneric factor, or two-factor models), and perform moderator analyses for the model-implied inter item correlations and estimated factor loadings. A Pearson inter-item correlation matrix was obtained for 48 independent studies, from which a pooled matrix was calculated following a random-effects multivariate meta-analysis. The results from the one-stage MASEM analysis showed that the two-factor model properly fitted the pooled matrix, while the τ-equivalent and congeneric one-factor models did not. Even though, the use of a bifactor model exhibited the predominance of the general factor over the domain-specific ones. High omega coefficients were obtained for the entire scale (.91) and the psychological (.83) and physiological (.83) symptoms subscales. Moderator analyses evidenced an increase in the estimated factor loadings, as well as in the reliability of the FCV-19S, when the standard deviation of the total scores increased and when the FCV-19S was administered to specific (vs. general) populations. The FCV-19S can be therefore considered as a highly related two-factor scale whose reliability makes it suitable for applied and research purposes.2022-2

Global, regional, and national burden of chronic kidney disease, 1990–2017 : a systematic analysis for the Global Burden of Disease Study 2017

Background Health system planning requires careful assessment of chronic kidney disease (CKD) epidemiology, but data for morbidity and mortality of this disease are scarce or non-existent in many countries. We estimated the global, regional, and national burden of CKD, as well as the burden of cardiovascular disease and gout attributable to impaired kidney function, for the Global Burden of Diseases, Injuries, and Risk Factors Study 2017. We use the term CKD to refer to the morbidity and mortality that can be directly attributed to all stages of CKD, and we use the term impaired kidney function to refer to the additional risk of CKD from cardiovascular disease and gout. Methods The main data sources we used were published literature, vital registration systems, end-stage kidney disease registries, and household surveys. Estimates of CKD burden were produced using a Cause of Death Ensemble model and a Bayesian meta-regression analytical tool, and included incidence, prevalence, years lived with disability, mortality, years of life lost, and disability-adjusted life-years (DALYs). A comparative risk assessment approach was used to estimate the proportion of cardiovascular diseases and gout burden attributable to impaired kidney function. Findings Globally, in 2017, 1·2 million (95% uncertainty interval [UI] 1·2 to 1·3) people died from CKD. The global all-age mortality rate from CKD increased 41·5% (95% UI 35·2 to 46·5) between 1990 and 2017, although there was no significant change in the age-standardised mortality rate (2·8%, −1·5 to 6·3). In 2017, 697·5 million (95% UI 649·2 to 752·0) cases of all-stage CKD were recorded, for a global prevalence of 9·1% (8·5 to 9·8). The global all-age prevalence of CKD increased 29·3% (95% UI 26·4 to 32·6) since 1990, whereas the age-standardised prevalence remained stable (1·2%, −1·1 to 3·5). CKD resulted in 35·8 million (95% UI 33·7 to 38·0) DALYs in 2017, with diabetic nephropathy accounting for almost a third of DALYs. Most of the burden of CKD was concentrated in the three lowest quintiles of Socio-demographic Index (SDI). In several regions, particularly Oceania, sub-Saharan Africa, and Latin America, the burden of CKD was much higher than expected for the level of development, whereas the disease burden in western, eastern, and central sub-Saharan Africa, east Asia, south Asia, central and eastern Europe, Australasia, and western Europe was lower than expected. 1·4 million (95% UI 1·2 to 1·6) cardiovascular disease-related deaths and 25·3 million (22·2 to 28·9) cardiovascular disease DALYs were attributable to impaired kidney function. Interpretation Kidney disease has a major effect on global health, both as a direct cause of global morbidity and mortality and as an important risk factor for cardiovascular disease. CKD is largely preventable and treatable and deserves greater attention in global health policy decision making, particularly in locations with low and middle SDI