Lotte shopping valuation using residual operating inocme model

Thesis(masters) --서울대학교 대학원 :경영학과(SNU Global MBA),2008. 8.Maste

A Study on the Design of Low Noise Amplifier and Low Frequency Noise Model of GaAs FET

Docto

기후 변화 시나리오에 따른 데이비드 빙하 빙상 거동 민감도 분석

학위논문 (석사)-- 서울대학교 대학원 : 자연과학대학 지구환경과학부, 2018. 2. 이강근.As global mean temperature rises, there has been a growing interest in how much sea level would rise occurred due to polar glacial discharge to ocean. Currently, West Antarctica has been spotlighted due to ice discharge from the glaciers, compared to sea level contribution in East Antarctic glaciers. Bed elevation at East Antarctica is largely lying above sea level, which is likely to stable compared to West Antarctica. However, East Antarctic regions contain 10 times large sea level rise potential than West Antarctica. David Glacier, located in East Antarctica, is a region of fjord-like valley glacier, and ice drains into ocean through Drygalski Ice Tongue, which of length is about 80 km. To understand what mechanism modulates sea level rise, it is necessary to identify key factors affecting the acceleration of mass discharge in David glacier. Based on current knowledge, ice shelf buttressing effect, basal melting, and SMB (surface mass balance) are the components that have been known to affect glacier speed. Here, 2D Shallow Shelf Approximation model of the Ice Sheet System Model was used to predict response of glacier velocity distribution and contribution of sea level equivalent change depending on various forcing scenarios. Firstly, friction coefficient beneath glacier and ice rigidity on floating ice were estimated through inversion method, which constructed the initial condition of the regional model. Then, changing SMB, floating ice melting rate, and ice front position retreat could alter the sea level rise contribution and ice velocity. In the results, basal drag stress obtained through inversion method was largely calculated in the ice fall area where the subglacial ridge existed. Sea level equivalent for control model was -2.0 mm equivalent to ice mass gain of 15 Gt/yr during 50 years, and relatively stable than other fast flow regions, such as Pine Island Glacier. Ice front retreat over threshold, which was about 90 km from ice front, accelerated the ice velocity near grounding line larger than twofold floating ice melting rate. This ice velocity acceleration influenced increase in sea level equivalent of -1.95 mm in case of furthermost ice front retreat. However, ice tongue and 8 km region of ice shelf position did not affect the ice velocity acceleration.1. INTRODUCTIONS 1 1.1. Background 1 1.2. David Glacier 4 1.3. Objectives 8 2. METHODS 9 2.1. Stress Balance Models 11 2.1.1. Full Stokes Model (FS) 11 2.1.2. Higher Order Model (HO) 13 2.1.3. Shallow Shelf Approximation Model (SSA) 14 2.1.4. Boundary Conditions 15 2.1.5. Friction Coefficient Parameterization at Grounding Line 17 2.2. Mass Transport Model 19 2.3. Initialization 20 2.3.1. Input Data 20 2.3.2. Basal Friction and Ice Rigidity 36 2.3.3. Relaxation 42 2.4. Climate Forcing Scenarios 44 3. RESULTS AND DISCUSSION 48 3.1. Initialization 48 3.1.1. Inversion Results 48 3.1.2. Relaxation Results 54 3.2. Results of Climate Forcing Scenarios 58 4. CONCLUSIONS 69 5. REFRENCES 71Maste

ACCURACY AND INTEROBSERVER- INTRAOBSERVER AGREEMENT IN THE RADIOLOGIC DIAGNOSIS OF AMELOBLASTOMA AND ODONTOGENIC KERATOCYST

Six observers with different amount of experience in the field of Oral and Maxillofacial Radiology interpreted the radiographs of 13 cases of ameloblastoma and 8 cases of odontogenic keratocyct which were confirmed histopathologically and showed pseudo-multilocular appearance and scalloped border radiographically. The authors examined the accuracy of radiologic diagnoses, interobserver agrement and intraobserver agreement. The obtained results were as follows; 1. The accuracy of radiologic diagnosis ranged from 0.48 to 0.81. The average value was 0.61. 2. The accuracy of radiologic diagnosis for ameloblastoma(0.55) was lower than that for odontogenic keratocyst(0.70) (P<0.05). 3. The overall agreement among the 6 observers was 14.3％ at the first interpretation and 19.0％ at the second interpretation. 4. The intraobserver agreement of each observer expressed in kappa index ranged from -0.14 to 0.64. The overall intraobserver agreement was 0.29. 5. The intraobserver agreement of each observer for ameloblastoma and odontogenic keratocyst ranged from -0.07 to 0.65 and from -0.25 to 1.00, respectively. The overall intraobserver agreement for ameloblatoma and odontogenic keratocyst were 0.27 and 0.26, respectively. 6. The diagnostic accuracy highly correlated to the intraobserver agreement(r=0.6370)

Effect of Radiation Dosage Changes on the Cell Viability and the Apoptosis Induction on Normal and Tumorigenic Cells

Purpose : The study was aimed to detect the differences in the cell viability and the apoptosis induction after irradiation on normal and tumorigenic cells. Materials and Methods : The study. that was generated for two human normal cells(RHEK, HGF-l) and two human tumor cells(KB. HT-1080). was tested using MTT assay at 1 day and 3 day after irradiation and TUNEL assay under confocal laser scanning microscope at 1 day after irradiation. Single irradiation of 0.5. 1, 2. 4. and 8Gy were applied to the cells. The two fractions of 1. 2. 4. and 8Gy were separated with a 4-hour time interval. The irradiation was done with 5.38Gy/min dose rate using Cs-137 irradiator at room temperature. Results and Conclusions : 1. In 3-day group. the cell viability of HGF-1 cell was significantly decreased at 2. 4 and 8Gy irradiation, the cell viability of KB cell was significantly decreased at 8Gy irradiation and the cell viability of HT-I080 cell was significantly decreased at 4 and 8Gy irradiation. 2. There was significant difference between RHEK and KB cell line in the cell viability of 3-day group at 8Gy irradiation. There was significant difference between RHEK and HGF-1 cell line in the cell viability of 3-day group at 4 and 8Gy irradiation. 3. There was a significantly decreased cell viability in 3-day group than those in 1-day group at 2. 4 and 8Gy on HGF-1 cell. at 4 and 8Gy on HT-I080 cell. at 8Gy on KB cell. 4. We could detect DNA fragmented cells only on KB cell. Number of apoptotic cells of KB cell was significantly increased at 4 and 8Gy irradiation. However, there was no correlation between cell viability and apoptosis. 5. On all 4 cell lines, there were no differences between single and split irradiation method in cell viability and apoptosis

A CASE REPORT OF PRIMARY INTRA-OSSEOUS CARCINOMA OF THE MAXILLA

The primary intra-osseous carcinoma (PIOC) is a very rare lesion. PIOC is an odontogenic carcinoma defined as a squamous cell carcinoma arisinig within a jaw having no initial connection with the oral mucosa, and presumably developing from residues of the odontogenic epithelium. The authors diagnosed a 51-year-old female as primary intra-osseous carcinoma after undergoing clinical, radiological and histological examinations. The characteristics were as followed : 1. The patient complained of gingival bleeding on the premolar area in the left maxilla 2. The conventional radiograms showed a relatively well-defined unilocular radiolucent lesion from the mesial aspect of the upper left canine to the mesial aspect of the upper left 1st molar. The 2nd premolar was separated from the 1st molar and the floor of the maxillary sinus was elevated by the lesion. There was a external root resorption of the upper left canine, the 1st premolar, and the 2nd premolar. 3. On the computed tomograms, the osteolytic bony lesion expanded the cortical plate of the left maxilla and displaced the margin of the left maxillary sinus upwards. But the bony lesion was separated from the maxillary sinus by a bony septum. 4. Bone scintigram with /sup 99m/Tc demonstrated the increased uptake in the left maxilla. Sonograms in the neck area and chest P-A radiogram didn't show any abnormalities. 5. Histologically, the tumor islands infiltrating into the surrounding bone increased in alveolar pattern, composed of the malignant cells, and there was a necrosis in the center of the tumor islands