72 research outputs found
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Takotsubo cardiomyopathy causing “transient heart failure” after cardiac surgery: A rare phenomenon
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Aspartame Disease A Possible Cause for Concomitant Graves' Disease and Pulmonary Hypertension
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Takotsubo cardiomyopathy caused by emotional stressors in the coronavirus disease 2019 (COVID-19) pandemic era
The COVID-19 pandemic has had far-reaching consequences beyond the disease itself, including economic, social, political, religious, and psychological implications. This novel coronavirus has been shown to have cardiovascular manifestations in the form of arrhythmias, conduction disturbances, myocarditis, stress cardiomyopathy, myocardial injury and myocardial ischemia or infarction from increased microvascular and/or macrovascular coagulopathy. However, in addition to these direct effects, we are now starting to recognize indirect cardiovascular effects of COVID-19 in the form of increased incidence of Takutsobo cardiomyopathy in patients without any evidence of coronavirus infection presumably due to the increased psychological stress of social isolation and societal turbulence. In this case series, we present two post-menopausal women, presenting with chest pain and acute coronary syndrome, who are finally diagnosed with stress cardiomyopathy, triggered by increased emotional stress-related to the pandemic. There is data from a retrospective cohort analysis showing a fourfold increase in the incidence of acute coronary syndrome resulting from stress cardiomyopathy during the pandemic period compared to similar times periods before the pandemic. Hence, health care providers need to be cognizant of the emotional ramifications of the ongoing pandemic in the form of increased risk of Takotsubo cardiomyopathy. Moreover, urgent measures need to be taken to help the at-risk population cope with the ongoing stressors to help decrease the incidence of this cardiomyopathy
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Mastering structural heart disease
"The aim of this book is to incorporate the different structural cardiology areas, latest techniques, devices, clinical research trials, future directions and innovation ideas into a practical and friendly "questions and answers" format, augmented by cases and reviews. The most renowned worldwide specialists on each of the topics will provide a concise overview, with valuable insights, knowledge and practical tips based on their experience. This will be supplemented by online clinical cases and vignettes for a complete overview of the topics in an absorbing, simplified method. The aim of this book is to be a valuable educational resource for interventional cardiologists, general cardiologists, cardiac surgeons, fellows in training and internists"-
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Orthotopic Cardiac Transplant and Straight-Back Syndrome: A Case Report
Straight-back syndrome is a common malformation of the thoracic spine that is associated with a marked decrease in the anterior-posterior intrathoracic cavitary space. The authors describe their experience with a patient with straight-back syndrome who required orthotopic cardiac transplantation. Mi nor modification of the procurement and surgical procedure was required to facilitate graft implantation without cardiac compression
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Comparisons of paired orthotopic cardiac transplant donor and recipient electrocardiograms
Electrocardiograms of 20 consecutive donor and paired recipient orthotopic heart transplant patients were compared. Recipients were markedly older than donors; all patients were males, and both groups had similar body weight. The heart rate was faster, QRS was longer, QT was shorter, QRS axis was more shift to the left, and precordial voltage was decreased in the recipient ECGs in the first 72 hours after transplant. These changes persisted during late follow-up (12 ± 6 months after transplant). Seventy percent and 60% of recipients within the first 72 hours after transplant and during late follow-up, respectively, had a new RBBB delay compared to their paired donor ECG. There was a trend toward less clockwise rotation in the ECGs of patients who had a new incomplete RBBB in the late follow-up compared to patients who did not. Thus new RBBB delays, QT shortening, QRS axis left shift, and decrease in precordial voltage are commonly seen on ECGs after orthotopic cardiac transplantation. A new heart position and recipient's weight do not seem to account for the development of a new RBBB delay and decrease in precordial voltage. Other factors, such as mechanical or thermal injury and change in donor-to-recipient age, may be involved
Recommended from our members
Mastering structural heart disease
"The aim of this book is to incorporate the different structural cardiology areas, latest techniques, devices, clinical research trials, future directions and innovation ideas into a practical and friendly "questions and answers" format, augmented by cases and reviews. The most renowned worldwide specialists on each of the topics will provide a concise overview, with valuable insights, knowledge and practical tips based on their experience. This will be supplemented by online clinical cases and vignettes for a complete overview of the topics in an absorbing, simplified method. The aim of this book is to be a valuable educational resource for interventional cardiologists, general cardiologists, cardiac surgeons, fellows in training and internists"-
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Right ventricular diastolic function in systemic hypertension
Right (RV) and left ventricular (LV) diastolic function was evaluated in 50 patients with mild, uncomplicated essential hypertension using pulsed-wave Doppler echocardiography. Patients with pulmonary, valvular or coronary artery disease were excluded and antihypertensive drugs were discontinued for the 2 weeks preceding the study. Ten normotensive patients without heart disease acted as control subjects. In the hypertensive patients, RV peak velocity of atrial filling was higher (42 ± 10 vs 31 ± 7 cm/s, p < 0.01) and deceleration half-time was prolonged (96 ± 20 vs 83 ± 10 ms, difference not significant); ratio of early/atrial filling velocity (1.1 ± 0.3 vs 1.7 ± 0.4, p < 0.001) and peak filling rate corrected to stroke volume (3.6 ± 0.7 vs S.3 ± 0.9 SV/s, p < 0.001) were lower. LV filling parameters showed similar changes. RV filling parameters did not correlate with age, LV mass or septal thickness but correlated weakly with LV radius/thickness ratio. There was good correlation between RV and the following corresponding LV filling parameters: peak filling rate, r = 0.68, p < 0.001; ratio of early/atrial filling, r = 0.88, p < 0.0001; and deceleration half-time, r = 0.62, p < 0.001. Data indicate that RV diastolic function is abnormal in essential hypertension and these abnormalities are closely related to those of LV diastolic function
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Interrelationship of variable coupling,multiformity and repetitive forms: Implications for classification of ventricular arrhythmias
One hundred 24-hour ambulatory electrocardiograms were prospectively examined to determine the relationship of variable coupling and multiformity of single premature ventricular complexes to repetitive forms. Premature ventricular complexes were present in 86 patients and were categorized by a three-tier decision-making tree using (1) multiformity confirmed in two channels, (2) variable coupling of >80 msec of premature ventricular complexes of similar QRS morphologies, and (3) repetitive forms of ≥2 premature ventricular complexes. Variable coupling was present in 51 patients, among whom 35 (69%) had repetitive forms; multiformity was present in 46 patients, among whom 37 (80%) had repetitive forms; repetitive forms were present in 41 patients, among whom only one patient (2%) did
not demonstrate multiformity or variable coupling. Variable coupling, multiformity, or either were significantly associated with the occurrence of repetitive forms (chi square=34, 15, 29, respectively, each
p<0.005). There was a bimodal distribution between patients with uniform, fixed coupled premature ventricular complexes who had rare repetitive forms (1 of 26) and patients with multiformity and variable coupling of premature ventricular complexes who had significantly more repetitive forms (30 of 37;
p<0.001). The frequency distribution of repetitive form length suggested a natural break point between five and six consecutive complexes. The findings suggest that variable coupling be considered as an intermediate form of ventricular arrhythmia; that isolated multiformity is rare and may not require a separate category; and support the division of ventricular ectopy considering salvos as runs of 3 to 5 in a row and nonsustained ventricular tachycardia as runs of≥6
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