1,417 research outputs found

    The Spin Alignment of Vector Mesons in High Energy pp Collisions

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    The spin alignment of vector meson produced in high energy reactions is determined by the spin-dependent fragmentation function D1LL(z,μf)D_{1LL} (z,\mu_f) that is shown to be independent of the polarization of the fragmenting quark. In this paper, we extract the spin-dependent fragmentation function D1LL(z,μf)D_{1LL} (z,\mu_f) from data on the spin alignment of K∗0K^{*0} in e+e−e^+e^- annihilation at LEP in two different scenarios and apply them to make predictions in pppp collisions. We make detailed analysis of contributions from different sub-processes and show that the spin alignment should be quite significant also in high energy pppp collisions.Comment: 12 pages, 16 fgiures,version printed in PR

    Nonextensive effects on QCD chiral phase diagram and baryon-number fluctuations within Polyakov-Nambu-Jona-Lasinio model

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    In this paper, a version of the Polyakov-Nambu-Jona-Lasinio (PNJL) model based on nonextensive statistical mechanics is presented. This new statistics summarizes all possible factors that violate the assumptions of the Boltzmann-Gibbs (BG) statistics to a dimensionless nonextensivity parameter qq, and when qq tends to 1, it returns to the BG case. Within the nonextensive PNJL model, we found that as qq increases, the location of the critical end point (CEP) exhibits non-monotonic behavior. That is, for q<1.15q<1.15, CEP moves in the direction of lower temperature and larger quark chemical potential. But for q>1.15q>1.15, CEP turns to move in the direction of lower temperature and lower quark chemical potential. In addition, we studied the moments of the net-baryon number distribution, that is, the variance (σ2\sigma^{2}), skewness (S), and kurtosis (κ\kappa). Our results are generally consistent with the latest experimental data, especially for SNN>19.6 GeV\sqrt{S_{NN}}>19.6\ \mathrm{GeV}, when qq is set to 1.071.07

    The study of nonstrange quark stars within a modified NJL model

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    In this work, we employ a modified Nambu-Jona-Lasinio (NJL) model with proper-time regularization to study the structure of nonstrange quark stars. Considering the physical meaning of the four-fermion interaction coupling constant GG in the conventional NJL model, we modify it by G=G1+G2⟨ψˉψ⟩G=G_1+G_2\langle\bar{\psi}\psi\rangle to highlight the feedback of quark propagator to gluon propagator. To study the dependence of the equation of state (EOS) on this modification as well as the vacuum pressure, we choose nine representative EOSs for comparison. As a result, we find that a smaller G1G_1 corresponds to a stiffer EOS, but at low energy densities, a higher vacuum pressure (i.e., a smaller bag constant) yields a softer one. Furthermore, the heaviest quark star obtained with the modified NJL model satisfies not only recent mass measurement of PSR J0740+6620, but also radius constraints from x-ray timing observations. The corresponding tidal deformability is also in agreement with GW170817.Comment: 8 pages, 9 figure

    Potential biomarkers relating pathological proteins, neuroinflammatory factors and free radicals in PD patients with cognitive impairment: a cross-sectional study

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    BACKGROUND: Cognitive impairment strikingly reduces the quality of life of Parkinson’s disease (PD) patients. Studies find that pathological proteins, neuroinflammatory factors and free radicals may involve in the pathogenesis of cognitive impairment of PD, however, results are inconclusive. METHODS: We recruited 62 PD patients and 31 healthy controls. PD patients were identified with cognitive impairment, including PD with mild cognitive impairment (PD-MCI) and PD with dementia (PDD) according to the diagnostic criteria for PD-MCI and PDD issued by Movement Disorder Society Task Force. The levels of pathological proteins, including β-amyloid 1–42 (Aβ1-42),Total-tau (T-tau) and phosphorelated tau (P-tau), neuroinflammatory factors,including tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), interleukin-6 (IL-6), interferon-γ (INF-γ) and prostaglandin E(2) (PGE(2)), free radicals, including hydroxyl radical (·OH), hydrogen peroxide (H(2)O(2)) and nitric oxide (NO) in cerebrospinal fluid(CSF) were detected. The levels of above factors in CSF were compared among healthy controls and patients with and without cognitive impairment. Correlation analyses were performed between Montreal Cognitive Assessment (MoCA) score and the levels of above factors in CSF. RESULTS: T-tau level in CSF from PD-CI patients are significantly elevated comparing with those without cognitive impairment and controls (P = 0.016 and 0.004, respectively). The levels of P-tau (S396) and · OH in PD-CI patients are significantly higher than controls (P = 0.001 and 0.014, respectively). IL-6 levels in PD-CI patients are strikingly enhanced comparing with those without cognitive impairment (P = 0.005). MoCA score is negatively correlated with the levels of T-tau (r = -0.340), P-tau (S396) (r = -0.448), IL-6 (r = -0.489) and · OH (r = -0.504) in PD-CI patients. CONCLUSIONS: Elevated levels of T-tau, P-tau (S396), IL-6 and · OH in CSF are significantly correlated with cognitive impairment in PD patients. This investigation may suggest the potential biomarkers relating pathological proteins, neuroinflammatory factors and free radicals in PD patients with cognitive impairment
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