227 research outputs found
H-Dibaryon from Lattice QCD with Improved Anisotropic Actions
The six quark state(uuddss) called H dibaryon(,) has been
calculated to study its existence and stability. The simulations are performed
in quenched QCD on and anisotropic lattices
with Symanzik improved gauge action and Clover fermion action. The gauge
coupling is and aspect ratio . Preliminary results
indicate that mass of H dibaryon is 2134(100)Mev on lattice and
2167(59)Mev on respectively. It seems that the radius of H
dibaryon is very large and the finite size effect is very obvious
Search for H dibaryon on the lattice
We investigate the H-dibaryon, an with , in the
chiral and continuum regimes on anisotropic lattices in quenched QCD.
Simulations are performed on very coarse lattices with refined techniques to
obtain results with high accuracy over a spatial lattice spacing in the range
of fm. We present results for the energy difference
between the ground state energy of the hexa-quark stranglet and the free
two-baryon state from our ensembles. A negative binding energy observed in the
chirally extrapolated results leads to the conclusion that the measured
hexa-quark state is bound. This is further confirmed by the attractive
interaction in the continuum limit with the observed H-dibaryon bound by MeV.Comment: 7 pages, 5 figures, Accepted for publication in Phys. Rev.
Reinforcement Learning Based Robust Volt/Var Control in Active Distribution Networks With Imprecisely Known Delay
Active distribution networks (ADNs) incorporating massive photovoltaic (PV)
devices encounter challenges of rapid voltage fluctuations and potential
violations. Due to the fluctuation and intermittency of PV generation, the
state gap, arising from time-inconsistent states and exacerbated by imprecisely
known system delays, significantly impacts the accuracy of voltage control.
This paper addresses this challenge by introducing a framework for delay
adaptive Volt/Var control (VVC) in the presence of imprecisely known system
delays to regulate the reactive power of PV inverters. The proposed approach
formulates the voltage control, based on predicted system operation states, as
a robust VVC problem. It employs sample selection from the state prediction
interval to promptly identify the worst-performing system operation state.
Furthermore, we leverage the decentralized partially observable Markov decision
process (Dec-POMDP) to reformulate the robust VVC problem. We design Multiple
Policy Networks and employ Multiple Policy Networks and Reward Shaping-based
Multi-agent Twin Delayed Deep Deterministic Policy Gradient (MPNRS-MATD3)
algorithm to efficiently address and solve the Dec-POMDP model-based problem.
Simulation results show the delay adaption characteristic of our proposed
framework, and the MPNRS-MATD3 outperforms other multi-agent reinforcement
learning algorithms in robust voltage control
Critical Behavior of Ferromagnetic Ising Model on Triangular Lattice
We apply a new updating algorithm scheme to investigate the critical behavior
of the two-dimensional ferromagnetic Ising model on a triangular lattice with
nearest neighbour interactions. The transition is examined by generating
accurate data for large lattices with . The spin
updating algorithm we employ has the advantages of both metropolis and
single-update methods. Our study indicates that the transition to be continuous
at . A convincing finite-size scaling analysis of the model
yield , , ,
, (scaling) and
(hyperscaling) respectively. Estimates of present scheme
yield accurate estimates for all critical exponents than those obtained with
Monte Carlo methods and show an excellent agreement with their well-established
predicted values
3-Chloropyridin-2-amine
In the title compound, C5H5ClN2, a by-product in the synthesis of ethyl 2-(3-chloropyridin-2-yl)-5-oxopyrazolidine-3-carboxylate, the amine groups form intermolecular hydrogen-bonding associations with pyridine N-atom acceptors, giving centrosymmetric cyclic dimers. Short intermolecular Cl⋯Cl interactions [3.278 (3) Å] also occur
Poly[[μ2-aqua-μ3-(4-carboxy-2-propyl-1H-imidazole-5-carboxylato-κ4 N 3,O 4:O 4:O 5)-sodium] hemihydrate]
In the title compound, {[Na(C8H9N2O4)(H2O)]·0.5H2O}n, the Na+ ion is coordinated by two bridging water molecules, one N atom and three O atoms from three 4-carboxy-2-propyl-1H-imidazole-5-carboxylate (H2pimdc) ligands. Adjacent Na+ ions are linked alternately by two water O atoms and two carboxy O atoms into a chain along [001]. These chains are connected through the coordination of the carboxylate O atoms to the Na+ ions, forming a three-dimensional structure. An intramolecular O—H⋯O hydrogen bond and intermolecular N—H⋯O and O—H⋯O hydrogen bonds are present in the crystal structure
Lowest-lying Tetra-Quark Hadrons in Anisotropic Lattice QCD
We present a detailed study of lowest-lying hadrons in
quenched improved anisotropic lattice QCD. Using the and
diquark-antidiquark local and smeared operators, we attempt to isolate the
signal for and states in two flavour
QCD. In the chiral limit of light-quark mass region, the lowest scalar
state is found to have a mass, MeV, which is slightly
lower than the experimentally observed . The results from our
variational analysis do not indicate a signature of a tetraquark resonance in
I=1 and I=2 channels. After the chiral extrapolation the lowest
state is found to have a mass, MeV. We analysed the
static potential extracted form a tetraquark Wilson loop and illustrated
the behaviour of the state as a bound state, unbinding at some critical
diquark separation. From our analysis we conclude that scalar system
appears as a two-pion scattering state and that there is no spatially-localised
state in the light-quark mass region.Comment: 9 pages, 10 figure
A novel 7-chemokine-genes predictive signature for prognosis and therapeutic response in renal clear cell carcinoma
Background: Renal clear cell carcinoma (ccRCC) is one of the most prevailing type of malignancies, which is affected by chemokines. Chemokines can form a local network to regulate the movement of immune cells and are essential for tumor proliferation and metastasis as well as for the interaction between tumor cells and mesenchymal cells. Establishing a chemokine genes signature to assess prognosis and therapy responsiveness in ccRCC is the goal of this effort.Methods: mRNA sequencing data and clinicopathological data on 526 individuals with ccRCC were gathered from the The Cancer Genome Atlas database for this investigation (263 training group samples and 263 validation group samples). Utilizing the LASSO algorithm in conjunction with univariate Cox analysis, the gene signature was constructed. The Gene Expression Omnibus (GEO) database provided the single cell RNA sequencing (scRNA-seq) data, and the R package “Seurat” was applied to analyze the scRNA-seq data. In addition, the enrichment scores of 28 immune cells in the tumor microenvironment (TME) were calculated using the “ssGSEA” algorithm. In order to develop possible medications for patients with high-risk ccRCC, the “pRRophetic” package is employed.Results: High-risk patients had lower overall survival in this model for predicting prognosis, which was supported by the validation cohort. In both cohorts, it served as an independent prognostic factor. Annotation of the predicted signature’s biological function revealed that it was correlated with immune-related pathways, and the riskscore was positively correlated with immune cell infiltration and several immune checkpoints (ICs), including CD47, PDCD1, TIGIT, and LAG-3, while it was negatively correlated with TNFRSF14. The CXCL2, CXCL12, and CX3CL1 genes of this signature were shown to be significantly expressed in monocytes and cancer cells, according to scRNA-seq analysis. Furthermore, the high expression of CD47 in cancer cells suggested us that this could be a promising immune checkpoint. For patients who had high riskscore, we predicted 12 potential medications.Conclusion: Overall, our findings show that a putative 7-chemokine-gene signature might predict a patient’s prognosis for ccRCC and reflect the disease’s complicated immunological environment. Additionally, it offers suggestions on how to treat ccRCC using precision treatment and focused risk assessment
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