55 research outputs found
Preparation of Mouse Monoclonal Antibody for RB1CC1 and Its Clinical Application
- Author
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2012
- Field of study
RB1-inducible coiled-coil 1 (RB1CC1; also known as FIP200) plays important roles in several biological pathways such as cell proliferation and autophagy. Evaluation of RB1CC1 expression can provide useful clinical information on various cancers and neurodegenerative diseases. In order to realize the clinical applications, it is necessary to establish a stable supply of antibody and reproducible procedures for the laboratory examinations. In the present study, we have generated mouse monoclonal antibodies for RB1CC1, and four kinds of antibodies (N1-8, N1-216, N3-2, and N3-42) were found to be optimal for clinical applications such as ELISA and immunoblots and work as well as the pre-existing polyclonal antibodies. N1-8 monoclonal antibody provided the best recognition of RB1CC1 in the clinico-pathological examination of formalin-fixed paraffin-embedded tissues. These monoclonal antibodies will help to generate new opportunities in scientific examinations in biology and clinical medicine
Screening for Microsatellite Instability Identifies Frequent 3âČ-Untranslated Region Mutation of the RB1-Inducible Coiled-Coil 1 Gene in Colon Tumors
- Author
- A Duval
- A Pietschmann
- A Siepel
- A Suzuki
- AS Halees
- B Gan
- B Gan
- B John
- Barbara A. Leggett
- Bogdan C. Paun
- BP Lewis
- BS Strauss
- CR Boland
- D Karolchik
- EA Sia
- G Mao
- H Ueda
- H Wei
- H Zhang
- I Lopez de Silanes
- J Reeder
- J Shin
- Joanne Young
- K Heinimann
- K Levenberg
- K Shiomi
- K Tenney
- L Wu
- M Ariza
- M Kertesz
- M Wierdl
- N Martin
- N Suraweera
- Patrick Tan
- S Abbi
- S Mitomo
- SJ Leedham
- SM Woerner
- SM Woerner
- Stephen J. Meltzer
- T Chano
- T Chano
- T Ruggiero
- TS Wong
- WM Grady
- Y Mori
- Y Mori
- Yulan Cheng
- Yuriko Mori
- ZK Melkoumian
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
BACKGROUND: Coding region microsatellite instability (MSI) results in loss of gene products and promotion of microsatellite-unstable (MSI-H) carcinogenesis. Recent studies have indicated that MSI within 3'-untranslated regions (3'UTRs) may post-transcriptionally dysregulate gene products. Within this context, we conducted a broad mutational survey of 42 short 3'UTR microsatellites (MSs) in 45 MSI-H colorectal tumors and their corresponding normal colonic mucosae. METHODOLOGY/PRINCIPAL FINDINGS: In order to estimate the overall susceptibility of MSs to MSI in MSI-H tumors, the observed MSI frequency of each MS was correlated with its length, interspecies sequence conservation level, and distance from some genetic elements (i.e., stop codon, polyA signal, and microRNA binding sites). All MSs were stable in normal colonic mucosae. The MSI frequency at each MS in MSI-H tumors was independent of sequence conservation level and distance from other genetic elements. In contrast, MS length correlated significantly with MSI frequency in MSI-H tumors (r=0.86, p=7.2x10(-13)). 3'UTR MSs demonstrated MSI frequencies in MSI-H tumors higher than the 99% upper limit predicted by MS length for RB1-inducible coiled-coil 1(RB1CC1, mutation frequency 68.4%), NUAK family SNF1-like kinase 1(NUAK1, 31.0%), and Rtf1, Paf1/RNA polymerase II complex component, homolog (RTF1, 25.0%). An in silico prediction of RNA structure alterations was conducted for these MSI events to gauge their likelihood of affecting post-transcriptional regulation. RB1CC1 mutant was predicted to lose a microRNA-accessible loop structure at a putative binding site for the tumor-suppressive microRNA, miR-138. In contrast, the predicted 3'UTR structural change was minimal for NUAK1- and RTF1 mutants. Notably, real-time quantitative RT-PCR analysis revealed significant RB1CC1 mRNA overexpression vs. normal colonic mucosae in MSI-H cancers manifesting RB1CC1 3'UTR MSI (9.0-fold; p = 3.6x10(-4)). CONCLUSIONS: This mutational survey of well-characterized short 3'UTR MSs confirms that MSI incidence in MSI-H colorectal tumors correlates with MS length, but not with sequence conservation level or distance from other genetic elements. This study also identifies RB1CC1 as a novel target of frequent mutation and aberrant upregulation in MSI-H colorectal tumors. The predicted loss of a microRNA-accessible structure in mutant RB1CC1 RNA fits the hypothesis that 3'UTR MSI involves in aberrant RB1CC1 posttranscriptional upregulation. Further direct assessments are indicated to investigate this possibility.Bogdan C. Paun, Yulan Cheng, Barbara A. Leggett, Joanne Young, Stephen J. Meltzer, Yuriko Mor
Genetic evaluation of dementia with Lewy bodies implicates distinct disease subgroups
- Author
- Aarsland D
- Al-Sarraj S
- Albert MS
- Alcolea D
- Attems J
- Barrett MJ
- Beach TG
- Bekris LM
- Bennett DA
- Black SE
- Boeve BF
- Brett F
- Brice A
- Calvo A
- Canosa A
- Chia R
- Chio A
- Clarimon J
- Dalgard CL
- Dawson TM
- Dickson DW
- Faber K
- Ferman T
- Ferrucci L
- Flanagan ME
- Foroud TM
- Fortea J
- Gan-Or Z
- Gentleman S
- Ghetti B
- Gibbs JR
- Goate A
- Goldstein DS
- Gonzalez-Aramburu I
- Graff-Radford NR
- Hardy JA
- Hodges AK
- Infante J
- Kaivola K
- Kaufmann H
- Keith J
- KrĂŒger R
- Kukull WA
- Lage C
- Lesage S
- Leverenz JB
- LleĂł A
- Logroscino G
- Love S
- Mao Q
- Masellis M
- Masliah E
- May P
- Mckeith IG
- Monuki E
- Mora G
- Morris CM
- Morris HR
- Newell KL
- Palma JA
- Palmer L
- Pastor P
- Pickering-Brown S
- Pletnikova O
- Renton AE
- Resnick SM
- Rogaeva E
- Rosenthal LS
- Ross OA
- Ryten M
- Sanchez-Juan P
- Scherzer CR
- Scholz SW
- Serrano GE
- Shah Z
- Shakkottai VG
- Sidransky E
- Tanaka T
- Thomas AJ
- Topol E
- Torkamani A
- Troakes C
- Troncoso JC
- Woltjer R
- Wszolek ZK
- Xiromerisiou G
- Publication venue
- OXFORD UNIV PRESS
- Publication date
- 01/05/2022
- Field of study
The APOE locus is strongly associated with risk for developing Alzheimer's disease and dementia with Lewy bodies. In particular, the role of the APOE Ï”4 allele as a putative driver of α-synuclein pathology is a topic of intense debate. Here, we performed a comprehensive evaluation in 2466 dementia with Lewy bodies cases versus 2928 neurologically healthy, aged controls. Using an APOE-stratified genome-wide association study approach, we found that GBA is associated with risk for dementia with Lewy bodies in patients without APOE Ï”4 (P = 6.58 Ă 10-9, OR = 3.41, 95% CI = 2.25-5.17), but not with dementia with Lewy bodies with APOE Ï”4 (P = 0.034, OR = 1.87, 95%, 95% CI = 1.05-3.37). We then divided 495 neuropathologically examined dementia with Lewy bodies cases into three groups based on the extent of concomitant Alzheimer's disease co-pathology: Pure dementia with Lewy bodies (n = 88), dementia with Lewy bodies with intermediate Alzheimer's disease co-pathology (n = 66) and dementia with Lewy bodies with high Alzheimer's disease co-pathology (n = 341). In each group, we tested the association of the APOE Ï”4 against the 2928 neurologically healthy controls. Our examination found that APOE Ï”4 was associated with dementia with Lewy bodies + Alzheimer's disease (P = 1.29 Ă 10-32, OR = 4.25, 95% CI = 3.35-5.39) and dementia with Lewy bodies + intermediate Alzheimer's disease (P = 0.0011, OR = 2.31, 95% CI = 1.40-3.83), but not with pure dementia with Lewy bodies (P = 0.31, OR = 0.75, 95% CI = 0.43-1.30). In conclusion, although deep clinical data were not available for these samples, our findings do not support the notion that APOE Ï”4 is an independent driver of α-synuclein pathology in pure dementia with Lewy bodies, but rather implicate GBA as the main risk gene for the pure dementia with Lewy bodies subgroup
Genome sequencing analysis identifies new loci associated with Lewy body dementia and provides insights into its genetic architecture
- Author
- Aarsland D
- Abramzon Y
- Ahmed S
- Al-Sarraj S
- Albani D
- Albert MS
- Alcolea D
- American Genome Center
- Ansorge O
- Attems J
- Bandres-Ciga S
- Barrett MJ
- Beach TG
- Bekris LM
- Bennett DA
- Besser LM
- Bigio EH
- Black SE
- Blauwendraat C
- Boeve BF
- Bohannan RC
- Brett F
- Brice A
- Brunetti M
- Cairns NJ
- Calvo A
- Canosa A
- Caraway CA
- Chia R
- ChiĂČ A
- Clarimon J
- Clark L
- Cusi D
- Dalgard CL
- Dawson TM
- Dickson DW
- Diez-Fairen M
- Ding J
- Duyckaerts C
- Eicher J
- Faber K
- Ferman T
- Ferrari R
- Ferrucci L
- Flanagan ME
- Floris G
- Foroud TM
- Fortea J
- Galasko D
- Gan-Or Z
- Gasser T
- Geiger JT
- Gentleman S
- Ghetti B
- Gibbs JR
- Goate A
- Goldstein DS
- GonzĂĄlez-Aramburu I
- Graff-Radford NR
- Grassano M
- Gustavsson E
- Halliday GM
- Hardy JA
- Hernandez DG
- Hodges AK
- Honig LS
- Infante J
- Jaunmuktane Z
- Kaiser SM
- Kaufmann H
- Keith J
- Kim RC
- Klein G
- KrĂŒger R
- Kukull WA
- Kuzma A
- Lage C
- Landi F
- Lashley T
- Lemstra A
- Lesage S
- Leverenz JB
- LleĂł A
- Logroscino G
- Londos E
- Lopez G
- Love S
- Makarious MB
- Mao Q
- Marder K
- Masellis M
- Masliah E
- May P
- McKeith IG
- Monuki ES
- Moore A
- Mora G
- Morenas-Rodriguez E
- Morgan K
- Morris CM
- Morris HR
- Morris JC
- Myllykangas L
- Nalls MA
- Newell KL
- Norcliffe-Kaufmann L
- Oinas M
- Palma J-A
- Palmer L
- Parkkinen L
- Pastor P
- Perkins M
- Pickering-Brown S
- Pletnikova O
- Portley MK
- Renton AE
- Resnick SM
- Reynolds RH
- RodrĂguez-RodrĂguez E
- Rogaeva E
- Rosenthal LS
- Ross OA
- Ryten M
- Sabir MS
- Saez-Atienzar S
- Salvi E
- Sanchez-Juan P
- Santana I
- Scherzer CR
- Scholz SW
- Schulte C
- Serrano GE
- Shah Z
- Shakkottai VG
- Sidransky E
- Singleton AB
- St George-Hyslop P
- Stone DJ
- Tanaka T
- Tayebi N
- Thomas AJ
- Tienari PJ
- Tilley BS
- Topol EJ
- Torkamani A
- Traynor BJ
- Troakes C
- Trojanowski JQ
- Troncoso JC
- Van Deerlin VM
- Viollet C
- Walton RL
- Warner TT
- Woltjer R
- Wszolek ZK
- Xiromerisiou G
- Publication venue
- Publication date
- 01/03/2021
- Field of study
The genetic basis of Lewy body dementia (LBD) is not well understood. Here, we performed whole-genome sequencing in large cohorts of LBD cases and neurologically healthy controls to study the genetic architecture of this understudied form of dementia, and to generate a resource for the scientific community. Genome-wide association analysis identified five independent risk loci, whereas genome-wide gene-aggregation tests implicated mutations in the gene GBA. Genetic risk scores demonstrate that LBD shares risk profiles and pathways with Alzheimer's disease and Parkinson's disease, providing a deeper molecular understanding of the complex genetic architecture of this age-related neurodegenerative condition
Pathogenic Huntingtin Repeat Expansions in Patients with Frontotemporal Dementia and Amyotrophic Lateral Sclerosis.
- Author
- Aarsland D
- Abramzon Y
- Adams DJ
- Adeleye A
- Aguilar M
- Ahmed S
- Al-Chalabi A
- Al-Sarraj S
- Al-Sarraj S
- Alba C
- Alba C
- Albani D
- Albert MS
- Altschuler S
- Alvarez I
- Amar K
- Ambrose JC
- Archibald N
- Arcila-Londono X
- Arepalli S
- Aronica E
- Arumugam P
- Asress S
- Atassi N
- Attems J
- Auluck P
- Baas F
- Baas F
- Babu S
- Bacikova D
- Bacikova D
- Baloh RH
- Baloh RH
- Bandmann O
- Baple EL
- Barrett MJ
- Beach TG
- Bekris LM
- Bennett DA
- Benussi L
- Berry JD
- Bertolin C
- Besser LM
- Bigio EH
- Binetti G
- Black SE
- Blair IP
- Bleda M
- Boada M
- Boardman-Pretty F
- Boeve BF
- Bohannan RC
- Boissiere JM
- Borroni B
- Boustred CR
- Bowser R
- Bowser R
- Boylan KB
- Brady CB
- Brett F
- Brice A
- Brice A
- Brittain H
- Broach J
- Broach JR
- Broce I
- Brown RH
- Brunetti M
- Bruni A
- Butovsky O
- Calini D
- Calvo A
- Camu W
- Canosa A
- Capps E
- Caraway CA
- Caroppo P
- Castellotti B
- Caulfield MJ
- Cereda C
- Ceroni M
- Chan GC
- Chandran S
- Chia R
- Chiang H-H
- ChiĂČ A
- ChiĂČ A
- Church A
- Clarimon J
- Clarimon J
- Coebergh J
- Colombrita C
- Comi GP
- Cooper-Knock J
- Corcia P
- Corrado L
- Corti S
- Costantini A
- Cox GA
- Craig CEH
- Crary J
- Critchley P
- Dalgard CL
- Danek A
- Danielson EW
- Dardiotis E
- Daugherty LC
- De Belleroche J
- De Burca A
- Del Bo R
- Devereau A
- Dewan R
- Dickson D
- Diehl-Schimd J
- Diekstra FP
- Diez-Fairen M
- Diez-Fairen M
- Ding J
- Dols-Icardo O
- Drepper C
- Drory V
- Drory VE
- Dubnau J
- Duga S
- Dunckley TL
- Duyckaerts C
- DâAlfonso S
- Elgar G
- Esteban-PĂ©rez J
- Faber K
- Faghri F
- Fallini C
- Farren J
- Feldman E
- Ferman T
- Ferrari R
- Ferrucci L
- Finkbeiner S
- Flanagan ME
- Floeter MK
- Floris G
- Foroud TM
- Fortea J
- Foulger RE
- Fowler T
- Fraenkel E
- Fratta P
- Fratta P
- FuriĂł-TarĂ P
- Gagliardi S
- Galimberti D
- Gan-Or Z
- GarcĂa-Redondo A
- Gellera C
- Gentleman S
- Gerhard G
- Ghetti B
- Ghidoni R
- Ghosh BCP
- Gibbs JR
- Gibbs JR
- Gibson SB
- Gkazi AS
- Glass JD
- Goate A
- Goldstein D
- GonzĂĄlez-Aramburu I
- Gotkine M
- Goutman SA
- Graff C
- Graff-Radford NR
- Hackett JM
- Halai D
- Hamblin A
- Hammell MG
- Hardiman O
- Hardy JA
- Harms M
- Harms MB
- Harris B
- Heiman-Patterson T
- Heiman-Patterson TD
- Henderson S
- Hernandez DG
- HernĂĄndez I
- Hickman RA
- Hjermind LE
- Hodges AK
- Holman JE
- Hoover B
- Hornstein E
- Hu H-C
- Hu MTM
- Hubbard TJP
- Hupalo D
- Hupalo DN
- Ibåñez K
- IllĂĄn-Gala I
- Infante J
- Iranzo A
- Jabbari E
- Jackson R
- Jansson L
- Johansson PM
- Jones LJ
- Kaiser SM
- Kamel F
- Kasperaviciute D
- Kaufmann H
- Kayikci M
- Keagle P
- Keith J
- Kenna KP
- Khoshnood B
- Kim RC
- King A
- Kirby J
- Klein G
- Kobylecki C
- Kost J
- Kowal N
- Kowall NW
- KrĂŒger R
- Kukull W
- Kuzma A
- Kwan J
- Laaksovirta H
- Lage C
- Lahnstein L
- Landers JE
- Landi F
- Lange DJ
- Lauria G
- Lawson K
- Le Ber I
- Leblond-Manry C
- Lee EB
- Leigh PN
- Leigh SEA
- Leong IUS
- Lesage S
- Leverenz JB
- Lewis EA
- LleĂł A
- LleĂł A
- Logroscino G
- Lopez G
- Lopez JF
- Love S
- Lumbroso S
- MacGowan DJ
- MacGowan DJL
- Malaspina A
- Maleady-Crowe F
- Mann C
- Mao Q
- Maragakis NJ
- Marti MJ
- Martinez EM
- Martinez-McGrath E
- Masellis M
- Masliah E
- Mason J
- Massey LA
- May P
- Mazzini L
- McDonagh EM
- McKeith I
- McKenna-Yasek D
- McLaughlin RL
- McMillan C
- Menon V
- Mesulam M-M
- Miller JW
- Miller TM
- Molina-Porcel L
- Monuki ES
- Mora G
- Moreno-Grau S
- Morris CM
- Morris CM
- Morris HR
- Morris HR
- Morrison KE
- Moutsianas L
- Mouzat K
- Mueller M
- Murugaesu N
- Muñoz-Blanco JL
- Myllykangas L
- Nacmias B
- Nalls MA
- Nath A
- Nath U
- Need AC
- Newell KL
- Nicholson GA
- Nielsen JE
- Nilsson CF
- Norcliffe-Kaufmann L
- Novelli V
- Odhams CA
- Orrell RW
- Ostrow LW
- Ostrow LW
- Padovani A
- Paganoni S
- Pal S
- Pall H
- Palma J-A
- Palmer L
- Pamphlett R
- Pastor P
- Pastor P
- Patch C
- Patsopoulos NA
- Pavese N
- Paviour D
- Pensato V
- Perez-Gil D
- Perkins M
- Perneczky R
- Phatnani H
- Phillips-Cremins J
- Pickering-Brown S
- Pioro E
- Pletnikova O
- Polak M
- Pollard HB
- Polychronopoulos D
- Portley MK
- Poss M
- Puca AA
- Pullinger J
- Pulst SM
- Querin G
- Rademakers R
- Rahim T
- Raj T
- Ratti A
- Ravits J
- Ravits JM
- Rendon A
- Renton AE
- Renton AE
- Resnick SM
- Reynolds RH
- Riesgo-Ferreiro P
- Robberecht W
- Robey I
- RodrĂguez-RodrĂguez E
- Rogaeva E
- Rogaeva E
- Rogaeva E
- Rogers T
- Rohrer JD
- Ross OA
- Rossi G
- Rossi M
- Rothstein JD
- Rouleau GA
- Rowe JB
- Ruiz A
- Ryten M
- Sabir MS
- Sanchez-Juan P
- Sapp PC
- Sareen D
- Savage K
- Sawant K
- Scarpini E
- Scherzer CR
- Schlachetzki JC
- Scholz SW
- Scott RH
- Scotter EL
- Sendtner M
- Serpente M
- Serrano GE
- Shakkottai V
- Shalem O
- Shankaracharya FNU
- Sharma J
- Shaw CE
- Shaw PJ
- Shaw PJ
- Shneider NA
- Siddiq A
- Sidle KC
- Sidransky E
- Sieghart A
- Silani V
- Simmons Z
- Simmons Z
- Singleton AB
- Smedley D
- Smith BN
- Smith C
- Smith KR
- Soltis AR
- SorarĂč G
- Sorbi S
- Sosinsky A
- Spooner W
- Stefanis L
- Stein TD
- Stevens HE
- Stevic Z
- Stone DJ
- Stuckey A
- Sukumar G
- Sultana R
- Tagliavini F
- Talbot K
- Tanaka T
- Taroni F
- Tayebi N
- Ten Asbroek ALMA
- Thomas AJ
- Thomas ERA
- Thompson LM
- Thompson SR
- Ticozzi N
- Tienari PJ
- Tilley BS
- Tiloca C
- Topp SD
- Torkamani A
- Traynor BJ
- Tregidgo C
- Troakes C
- Troakes C
- Trojanowski JQ
- Troncoso JC
- Troncoso JC
- Tucci A
- Tuck M
- Turner MR
- Valori M
- Van Blitterswijk M
- Van Damme P
- Van Deerlin V
- Van den Berg LH
- Van Den Bosch L
- Van Rheenen W
- Vance C
- Vaughan J
- Veldink JH
- Viollet C
- Vonsattel JP
- Waldo ML
- Walsh E
- Walton RL
- Watters SA
- Welland MJ
- Wilkerson MD
- Williams E
- Williams KL
- Witkowska K
- Woltjer R
- Wood SM
- Wszolek ZK
- Wu L
- Wu Z
- Xiromerisiou G
- Zaitlen N
- Zarowiecki M
- Zecca C
- Zhang B
- Zhang X
- Zinman L
- Ăijerstedt L
- Publication venue
- Neuron
- Publication date
- 01/01/2020
- Field of study
We examined the role of repeat expansions in the pathogenesis of frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS) by analyzing whole-genome sequence data from 2,442 FTD/ALS patients, 2,599 Lewy body dementia (LBD) patients, and 3,158 neurologically healthy subjects. Pathogenic expansions (range, 40-64 CAG repeats) in the huntingtin (HTT) gene were found in three (0.12%) patients diagnosed with pure FTD/ALS syndromes but were not present in the LBD or healthy cohorts. We replicated our findings in an independent collection of 3,674 FTD/ALS patients. Postmortem evaluations of two patients revealed the classical TDP-43 pathology of FTD/ALS, as well as huntingtin-positive, ubiquitin-positive aggregates in the frontal cortex. The neostriatal atrophy that pathologically defines Huntington's disease was absent in both cases. Our findings reveal an etiological relationship between HTT repeat expansions and FTD/ALS syndromes and indicate that genetic screening of FTD/ALS patients for HTT repeat expansions should be considered
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Search for leptonic decays of WâČ bosons in pp collisions at âs = 7 TeV
- Author
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullin S
- Abdulsalam A
- Acosta D
- Acosta JG
- Adair A
- Adam N
- Adam W
- Adams MR
- Adams T
- Adiguzel A
- Adler V
- Adzic P
- Agostino L
- Agram J-L
- Aguilar-Benitez M
- Aguilo E
- Ahmad M
- Ahuja S
- Akchurin N
- Akgun B
- Akgun U
- Akin IV
- Alagoz E
- Albajar C
- Albayrak EA
- Albergo S
- Albrow M
- Alcaraz Maestre J
- Aldaya Martin M
- AldĂĄ JĂșnior WL
- Alexander J
- Aliev T
- Alimena J
- Almeida N
- Alverson G
- Alves GA
- Amapane N
- Amsler C
- Anagnostou G
- Anastassov A
- Anderson J
- Anderson M
- Andrea J
- Andreev V
- Andreev Y
- Andrews W
- Anghel IM
- Anjos TS
- Antonelli L
- Antunovic Z
- Apanasevich L
- Appelt E
- Apresyan A
- Arce P
- Arcidiacono R
- Arenton MW
- Arfaei H
- Argiro S
- Arneodo M
- Arora S
- Asawatangtrakuldee C
- Asghar MI
- Askew A
- Assran Y
- Ata M
- Attikis A
- Auffray E
- Autermann C
- Auzinger G
- Avdeeva E
- Avery P
- Avetisyan A
- Avila C
- Azarkin M
- Azhgirey I
- Aziz T
- Azzi P
- Azzolini V
- Azzurri P
- Baarmand MM
- Babb J
- Bacchetta N
- Bachtis M
- Baden A
- Baffioni S
- Bagliesi G
- Bai Y
- Baillon P
- Bainbridge R
- Bakhshiansohi H
- Bakirci MN
- Balazs M
- Ball AH
- Ball G
- Ban Y
- Banerjee S
- Bansal S
- Banzuzi K
- Barbagli G
- Barberis E
- Barbone L
- Barfuss AF
- Bargassa P
- Barge D
- Baringer P
- Barker A
- Barnes VE
- Barnett BA
- Barney D
- Barone L
- Barrett M
- Bartalini P
- Barth C
- Basegmez S
- Basso L
- Battilana C
- Bauer G
- Bauerdick LAT
- Baumgartel D
- Baur U
- Bayshev I
- Bazterra VE
- Bean A
- Beauceron S
- Beaudette F
- Beaupere N
- Beernaert K
- Behr J
- Behrenhoff W
- Behrens U
- Belforte S
- Beliy N
- Belknap D
- Bell AJ
- Bell KW
- Bellan P
- Bellan R
- Belotelov I
- Belyaev A
- Benaglia A
- Bencze G
- Bendavid J
- Benedetti D
- Benelli G
- Benhabib L
- Beni N
- Benussi L
- Benvenuti AC
- Beranek S
- Beretvas A
- Bergauer T
- Berger J
- Bergholz M
- Beri SB
- Bernardes CA
- Bernet C
- Berry D
- Berry E
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- Willmott C
- Wimpenny S
- Winer BL
- Winn D
- Winstrom L
- Wissing C
- Wittich P
- Wittmer B
- Wolf M
- Wolf R
- Womersley WJ
- Won S
- Wood D
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- Worm SD
- Wright D
- Wrochna G
- Wu W
- Wulz C-E
- Wyslouch B
- Wöhri HK
- WĂŒrthwein F
- Xiao H
- Xie S
- Xu M
- Yagil A
- Yalvac M
- Yang F
- Yang M
- Yang Y
- Yang ZC
- Yazgan E
- Yelton J
- Yetkin T
- Yi K
- Yildirim E
- Yilmaz Y
- Yohay R
- Yoo HD
- Yoo J
- Yoon AS
- York A
- Yu I
- Yu SS
- Yumiceva F
- Yun JC
- Zabel J
- Zabi A
- Zablocki J
- Zaganidis N
- Zakaria M
- Zalewski P
- Zanetti M
- Zang J
- Zarubin A
- Zatserklyaniy A
- Zeinali M
- Zeise M
- Zeuner WD
- Zeyrek M
- Zhang J
- Zhang Z
- Zheng Y
- Zhokin A
- Zhu B
- Zhu RY
- Zhukov V
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- Ziebarth EB
- Ziegler J
- Zielinski M
- Zito G
- Zotto P
- Zou W
- Zumerle G
- Zuranski A
- Publication venue
- Publication date
- 02/03/2018
- Field of study
Upstream regulatory architecture of rice genes: summarizing the baseline towards genus-wide comparative analysis of regulatory networks and allele mining
- Author
- A Guo
- A Ikeda
- A Johansson
- A Lovegrove
- A Santino
- A Wager
- A Yilmaz
- AB Rose
- AC Vlot
- AH Christensen
- AJK Koo
- AM Moabbi
- AQ Srivastav
- AY Guo
- B Lenhard
- B Mohanty
- Benildo G de los Reyes
- Bijayalaxmi Mohanty
- C Cheng
- C Garcion
- C Grierson
- C He
- C Molina
- C Wasternack
- CA Lu
- CM Fleet
- CM Hart
- CMJ Pieterse
- D Desveaux
- D Hao
- D Liu
- D McElroy
- D McElroy
- D Todaka
- D Wang
- DA Dempsey
- DG Zarka
- DM Riaño-Pachón
- Dong-Yup Lee
- DW Choi
- E Rojo
- E Scarpella
- E Scarpella
- E Wingender
- EE Farmer
- EE Helliwell
- EJ Chapman
- EJ Stockinger
- EJ Stockinger
- F Takaiwa
- F Takaiwa
- F Waller
- F Xu
- FA Razem
- FB Abeles
- G Gao
- G Hagen
- G Parra
- H Abe
- H Guo
- H Hu
- H Itoh
- H Liu
- H Ohyanagi
- H Peng
- H Thomas
- H Washida
- H Zhang
- HG Nam
- HS Ryu
- I Ciolkowski
- IC Jang
- J Bhattacharyya
- J Chen
- J Giraudat
- J Griffiths
- J Jacquemin
- J Jacquemin
- J Li
- J Lu
- J Wang
- J Yazaki
- J Zhang
- JEF Butler
- JG Dubouzet
- JK Thakur
- JL Gomez-Porras
- JL Nemhauser
- JM Chandlee
- JM Manners
- JM Vogel
- JT Vogel
- K Baumann
- K Gomi
- K Hamada
- K Higo
- K Kaufmann
- K Miyamoto
- K Nakashima
- K Nakashima
- K Sato
- K Shinozaki
- K Sutoh
- K Suzuki
- K Yamaguchi-Shinozaki
- K Yamaguchi-Shinozaki
- KR Bradnam
- KR Jaglo
- KR Jaglo-Ottosen
- KY Yun
- L Liu
- LG Guedes Correa
- LM Hartweck
- LM Hartweck
- LQ Qu
- M Cai
- M Clancy
- M Freeling
- M Jain
- M Jain
- M Jain
- M Jakoby
- M Kaneko
- M Kasuga
- M Kieffer
- M Lescot
- M Ogawa
- M Ohme-Takagi
- M Ohme-Takagi
- M Ohta
- M Quint
- M Seki
- M Shimono
- M Shimono
- M Ueguchi-Tanaka
- M Ueguchi-Tanaka
- M Zourelidou
- MA Rabbani
- ME Alvarez
- MF Thomashow
- MJ Guiltinan
- MK Mejia-Guerra
- MP Singh
- MR Park
- Myoung-Ryoul Park
- N Baranowskij
- N Bodenhausen
- N Darmasiri
- N Yokotani
- NE Buchler
- P Bagnaresi
- P Clivan
- P Hai
- P Perez-Rodriguez
- P Silverman
- PO Lim
- PW Chen
- PW Chen
- Q Liu
- Q Zhu
- Q Zhu
- QJ Shen
- QJ Shen
- R Finkelstein
- R Mehrotra
- RA Creelman
- RA Wing
- RC Hardison
- RL Brown
- RL Hong
- RT Morris
- RV Davuluri
- S Bodt De
- S Fowler
- S Gan
- S Ishiguro
- S Meier
- S Robatzek
- S Rounsley
- SB Tiwari
- SH Hwang
- SH Park
- SJ Gilmour
- SK Lenka
- Song Joong Yun
- ST Smale
- ST Smale
- SY Fujimoto
- SY Kim
- T Hobo
- T Juven-Gershon
- T Shoji
- T Shoji
- T Ulmasov
- T Ulmasov
- T Urao
- TW Burke
- TW Okita
- V Buchanan-Wolaston
- V Buchanan-Wollaston
- V Chinnusamy
- V Matys
- W Zhang
- X Peng
- X Wang
- X Xie
- X Xu
- X Zhang
- X Zheng
- XQ Liu
- Y Croissant-Sych
- Y Gu
- Y Inukai
- Y Kitomi
- Y Narusaka
- Y Nishizawa
- Y Song
- Y Yang
- Y Zhao
- YM Jeong
- YS Noh
- YY Yamamoto
- Z Wang
- Z Zhang
- Z Zheng
- ZK Shinwari
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Combination of searches for heavy spin-1 resonances using 139 fbâ1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Aikot A
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Aktas S
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
- Alderweireldt S
- Alegria ZL
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi F
- Algren M
- Alhroob M
- Ali B
- Ali HMJ
- Ali S
- Alibocus SW
- Aliev M
- Alimonti G
- Alkakhi W
- Allaire C
- Allbrooke BMM
- Allen JF
- Allendes Flores CA
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alvarez Fernandez A
- Alves Cardoso M
- Alviggi MG
- Aly M
- Amaral Coutinho Y
- Ambler A
- Amelung C
- Amerl M
- Ames CG
- Amidei D
- Amirie KJ
- Amor Dos Santos SP
- Amos KR
- An S
- Ananiev V
- Anastopoulos C
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Anulli F
- Aoki M
- Aoki T
- Aparisi Pozo JA
- Aparo MA
- Aperio Bella L
- Appelt C
- Apyan A
- Arbiol Val SJ
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Arnaez O
- Arnold H
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Assamagan K
- Astalos R
- Astrand KSV
- Atashi S
- Atkin RJ
- Atkinson M
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Auriol AD
- Austrup VA
- Avolio G
- Axiotis K
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
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- Ă kesson TPA
- Ăncel ĂO
- ĆœeniĆĄ T
- ĆœivkoviÄ L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fbâ1 of proton-proton collisions at
= 13 TeV collected during 2015â2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (ÏÎœ and ÏÏ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
- âŠ