Report of diffusion-weighted MRI in two cases with different cerebral hydatid disease

WOS: 000221495600016PubMed ID: 15164785Purpose: To present MRI findings in two cases of cerebral hydatid disease with an emphasis oil diffusion-weighted imaging (DWI) findings of Echinococcus granulosus (EG) versus Echinococcus alveolaris (EA). Results: EG lesions were isointense with cerebrospinal fluid in all sequences including DWI. On DWI, EA lesions remained hypointense on b=1000 s/mm(2) diffusion-weighted images. Apparent diffusion coefficient (ADC) values of EG and EA lesions were completely different from each other, 2.88+/-0.24x10(-3) s/mm(2) and 1.33+/-0.15x10(-3) s/mm(2), respectively. Conclusion: The ADC values Could not be used to discriminate from other differential diagnoses

DWI findings of periventricular ischemic changes in patients with leukoaraiosis

WOS: 000184800600006PubMed ID: 12821031In this report the diffusion-weighted imaging (DWI) findings and ADC values of leukoaraiosis (LA) and associated periventricular ischemic lesions were assessed. Seventy-eight patients with LA were examined with magnetic resonance imaging (MRI) and DWI. Twenty one patients (28%) were found to have focal acute white matter infarction on DWI which could not be detected and discriminated on the basis of MRI findings alone. LA and acute white matter infarction both showed hyperintensity on standard T2W MRI, whereas acute infarction revealed focal hyperintensity on DWI. Thirteen patients (16%) had chronic lacunar infarctions in the white matter. ADC values of LA, acute and chronic white matter infarctions were calculated and found to be significantly different from each other (P < 0.05). It is concluded that DWI is necessary in the detection of acute periventricular white matter infarction from LA. (C) 2003 Published by Elsevier Science Ltd

Intramural Hydatid Cysts of Pulmonary-Arteries - Ct and Mr Findings

WOS: A1995TJ35200019Involvement of the pulmonary artery by echinococcosis usually occurs due to pulmonary embolization after rupture of the cyst in the right ventricle or, rarely, by dissemination from a hepatic focus. This paper presents an adult patient with intramural hydatid cysts of the pulmonary arteries, which apparently grew slowly toward the lumina resulting in complete luminal occlusion, enabling sufficient time for development of sufficient perfusion from the bronchial arteries. The condition apparently resulted from embryos of the parasite, which entered the ''vasa vasorum'' of the pulmonary arteries, a previously unreported mechanism

Juvenile distal spinal muscular atrophy: A case with Arnold-Chiari malformation

WOS: 000075392700012PubMed ID: 974792

Spectrum of anterior cerebral artery territory infarction: xClinical and MRI findings

PubMed ID: 12453077To evaluate and review the clinical spectrum of anterior cerebral artery (ACA) territory infarction, we studied 48 consecutive patients who admitted to our stroke unit over a 6-year period. We performed magnetic resonance imaging (MRI) and magnetic resonance angiography (MRA) in all patients, and diffusion magnetic resonance imaging (DWI) in 21. In our stroke registry, patients with ACA infarction represented 1.3% of 3705 patients with ischemic stroke. The main risk factors of ACA infarcts was hypertension in 58% of patients, diabetes mellitus in 29%, hypercholesterolemia in 25%, cigarette smoking in 19%, atrial fibrillation in 19%, and myocardial infarct in 6%. Presumed causes of ACA infarct were large-artery disease and cardioembolism in 13 patients each, small-artery disease (SAD) in the territory of Heubner's artery in two and atherosclerosis of large-arteries (< 50% stenosis)in 16. On clinico-radiologic analysis there were three main clinical patterns depending on lesion side; left-side infarction (30 patients) consisting of mutism, transcortical motor aphasia, and hemiparesis with lower limb predominance; right side infarction (16 patients) accompanied by acute confusional state, motor hemineglect and hemiparesis; bilateral infarction (two patients) presented with akinetic mutism, severe sphincter dysfunction, and dependent functional outcome. Our findings suggest that clinical and etiologic spectrum of ACA infarction may present similar features as that of middle cerebral artery infarction, but frontal dysfunctions and callosal syndromes can help to make a clinical differential diagnosis. Moreover, at the early phase of stroke, DWI is useful imaging method to locate and delineate the boundary of lesion in the territory of ACA