Hybrid exciton-polaritons in a bad microcavity containing the organic and inorganic quantum wells

We study the hybrid exciton-polaritons in a bad microcavity containing the organic and inorganic quantum wells. The corresponding polariton states are given. The analytical solution and the numerical result of the stationary spectrum for the cavity field are finishedComment: 3 pages, 1 figure. appear in Communications in Theoretical Physic

Carbon emissions intensity reduction target for China's power industry: An efficiency and productivity perspective

© 2018 Elsevier Ltd This paper proposes a scenario analysis to address whether the national and provincial CO2 emissions intensity reduction target during 2016–2020 would be achievable for China's power industry with the identification of change on carbon productivity. This productivity indicator is further decomposed to investigate contributions of different sources to productivity growth when there exists technological heterogeneity. Evaluation results show that even if all electricity-generating units in each region were able to adopt the best practice, the nationwide 18% intensity reduction target is not feasible through improving technical efficiency or upgrading technology on electricity generation and carbon abatement in a short or medium term. The existence of regional technological heterogeneity in power generation and associated CO2 emissions reduction processes implies the necessity of more differentiated regulations and policies for emission reduction across China's regions and inter-regional technology transfer. The emerging national emission trading scheme could easy some challenges in formulating emission policy for heterogeneous regions

Shear viscosity, instability and the upper bound of the Gauss-Bonnet coupling constant

We compute the dimensionality dependence of $\eta/s$ for charged black branes with Gauss-Bonnet correction. We find that both causality and stability constrain the value of Gauss-Bonnet coupling constant to be bounded by 1/4 in the infinite dimensionality limit. We further show that higher dimensionality stabilize the gravitational perturbation. The stabilization of the perturbation in higher dimensional space-time is a straightforward consequence of the Gauss-Bonnet coupling constant bound.Comment: 16 pages,3 figures+3 tables,typos corrected, published versio

Time Periodic Behavior of Multiband Superlattices in Static Electric Fields

We use an analytic perturbation expansion for the two-band system of tight binding electrons to discuss Bloch oscillations and Zener tunneling within this model. We make comparison with recent numerical results and predict analytically the frequency of radiation expected from Zener tunneling, including its disappearance, as a function of the system parameters.Comment: 12 pages, no figure include

On the relative expressiveness of higher-order session processes

By integrating constructs from the λ-calculus and the π-calculus, in higher-order process calculi exchanged values may contain processes. This paper studies the relative expressiveness of HOπ, the higher-order π-calculus in which communications are governed by session types. Our main discovery is that HO, a subcalculus of HOπ which lacks name-passing and recursion, can serve as a new core calculus for session-typed higher-order concurrency. By exploring a new bisimulation for HO, we show that HO can encode HOπ fully abstractly (up to typed contextual equivalence) more precisely and efficiently than the first-order session π-calculus (π). Overall, under session types, HOπ, HO, and π are equally expressive; however, HOπ and HO are more tightly related than HOπ and π

Loop Corrections to Cosmological Perturbations in Multi-field Inflationary Models

We investigate one-loop quantum corrections to the power spectrum of adiabatic perturbation from entropy modes/adiabatic mode cross-interactions in multiple DBI inflationary models. We find that due to the non-canonical kinetic term in DBI models, the loop corrections are enhanced by slow-varying parameter $\epsilon$ and small sound speed $c_s$. Thus, in general the loop-corrections in multi-DBI models can be large. Moreover, we find that the loop-corrections from adiabatic/entropy cross-interaction vertices are IR finite.Comment: 21 pages, 7 figures; v2, typos corrected, ref added; v3 typos corrected, version for publishing in jca

Visual Compositional Learning for Human-Object Interaction Detection

Human-Object interaction (HOI) detection aims to localize and infer relationships between human and objects in an image. It is challenging because an enormous number of possible combinations of objects and verbs types forms a long-tail distribution. We devise a deep Visual Compositional Learning (VCL) framework, which is a simple yet efficient framework to effectively address this problem. VCL first decomposes an HOI representation into object and verb specific features, and then composes new interaction samples in the feature space via stitching the decomposed features. The integration of decomposition and composition enables VCL to share object and verb features among different HOI samples and images, and to generate new interaction samples and new types of HOI, and thus largely alleviates the long-tail distribution problem and benefits low-shot or zero-shot HOI detection. Extensive experiments demonstrate that the proposed VCL can effectively improve the generalization of HOI detection on HICO-DET and V-COCO and outperforms the recent state-of-the-art methods on HICO-DET. Code is available at https://github.com/zhihou7/VCL.Comment: Accepted in ECCV202

Identification of translational activators of glial glutamate transporter EAAT2 through a cell-based high-throughput screening: An approach for preventing excitotoxicity

Excitotoxicity has been implicated as the mechanism of neuronal damage resulting from acute insults such as stroke, epilepsy, and trauma, as well as during the progression of adult-onset neurodegenerative disorders such as Alzheimer’s disease and amyotrophic lateral sclerosis (ALS). Excitotoxicity is defined as excessive exposure to the neurotransmitter glutamate or overstimulation of its membrane receptors, leading to neuronal injury or death. One potential approach to protect against excitotoxic neuronal damage is enhanced glutamate reuptake. The glial glutamate transporter EAAT2 is the quantitatively dominant glutamate transporter and plays a major role in clearance of glutamate. Expression of EAAT2 protein is highly regulated at the translational level. In an effort to identify compounds that can induce translation of EAAT2 transcripts, a cell-based enzyme-linked immunosorbent assay was developed using a primary astrocyte line stably transfected with a vector designed to identify modulators of EAAT2 translation. This assay was optimized for high-throughput screening, and a library of approximately 140,000 compounds was tested. In the initial screen, 293 compounds were identified as hits. These 293 hits were retested at 3 concentrations, and a total of 61 compounds showed a dose-dependent increase in EAAT2 protein levels. Selected compounds were tested in full 12-point dose-response experiments in the screening assay to assess potency as well as confirmed by Western blot, immunohistochemistry, and glutamate uptake assays to evaluate the localization and function of the elevated EAAT2 protein. These hits provide excellent starting points for developing therapeutic agents to prevent excitotoxicity

Tumor necrosis factor superfamily 15 promotes lymphatic metastasis via upregulation of vascular endothelial growth factor-C in a mouse model of lung cancer

Lymphatic metastasis is facilitated by lymphangiogenic growth factor vascular endothelial growth factor-C (VEGFC) that is secreted by some primary tumors. We previously identified tumor necrosis factor superfamily 15 (TNFSF15), a blood vascular endothelium-derived cytokine, in lymphatic endothelial cells, as a key molecular modulator during lymphangiogenesis. However, the effect of TNFSF15 on tumor lymphatic metastasis and the underlying molecular mechanisms remain unclear. We report here that TNFSF15, which is known to inhibit primary tumor growth by suppressing angiogenesis, can promote lymphatic metastasis through facilitating lymphangiogenesis in tumors. Mice bearing tumors induced by A549 cells stably overexpressing TNFSF15 exhibited a significant increase in densities of lymphatic vessels and a marked enhancement of A549 tumor cells in newly formed lymphatic vessels in the primary tumors as well as in lymph nodes. Treatment of A549 cells with TNFSF15 results in upregulation of VEGFC expression, which can be inhibited by siRNA gene silencing of death domain-containing receptor-3 (DR3), a cell surface receptor for TNFSF15. In addition, TNFSF15/DR3 signaling pathways in A549 cells include activation of NF-κB during tumor lymphangiogenesis. Our data indicate that TNFSF15, a cytokine mainly produced by blood endothelial cells, facilitates tumor lymphangiogenesis by upregulating VEGFC expression in A549 cells, contributing to lymphatic metastasis in tumor-bearing mice. This finding also suggests that TNFSF15 may have potential as an indicator for prognosis evaluation.Tingting Qin, Dingzhi Huang, Zhujun Liu, Xiaoling Zhang, Yanan Jia, Cory J. Xian, Kai L