103 research outputs found

    Recommending with an Agenda: Active Learning of Private Attributes using Matrix Factorization

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    Recommender systems leverage user demographic information, such as age, gender, etc., to personalize recommendations and better place their targeted ads. Oftentimes, users do not volunteer this information due to privacy concerns, or due to a lack of initiative in filling out their online profiles. We illustrate a new threat in which a recommender learns private attributes of users who do not voluntarily disclose them. We design both passive and active attacks that solicit ratings for strategically selected items, and could thus be used by a recommender system to pursue this hidden agenda. Our methods are based on a novel usage of Bayesian matrix factorization in an active learning setting. Evaluations on multiple datasets illustrate that such attacks are indeed feasible and use significantly fewer rated items than static inference methods. Importantly, they succeed without sacrificing the quality of recommendations to users.Comment: This is the extended version of a paper that appeared in ACM RecSys 201

    Privacy Tradeoffs in Predictive Analytics

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    Online services routinely mine user data to predict user preferences, make recommendations, and place targeted ads. Recent research has demonstrated that several private user attributes (such as political affiliation, sexual orientation, and gender) can be inferred from such data. Can a privacy-conscious user benefit from personalization while simultaneously protecting her private attributes? We study this question in the context of a rating prediction service based on matrix factorization. We construct a protocol of interactions between the service and users that has remarkable optimality properties: it is privacy-preserving, in that no inference algorithm can succeed in inferring a user's private attribute with a probability better than random guessing; it has maximal accuracy, in that no other privacy-preserving protocol improves rating prediction; and, finally, it involves a minimal disclosure, as the prediction accuracy strictly decreases when the service reveals less information. We extensively evaluate our protocol using several rating datasets, demonstrating that it successfully blocks the inference of gender, age and political affiliation, while incurring less than 5% decrease in the accuracy of rating prediction.Comment: Extended version of the paper appearing in SIGMETRICS 201

    GraphSC: Parallel secure computation made easy

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    Abstract-We propose introducing modern parallel programming paradigms to secure computation, enabling their secure execution on large datasets. To address this challenge, we present GraphSC, a framework that (i) provides a programming paradigm that allows non-cryptography experts to write secure code; (ii) brings parallelism to such secure implementations; and (iii) meets the needs for obliviousness, thereby not leaking any private information. Using GraphSC, developers can efficiently implement an oblivious version of graph-based algorithms (including sophisticated data mining and machine learning algorithms) that execute in parallel with minimal communication overhead. Importantly, our secure version of graph-based algorithms incurs a small logarithmic overhead in comparison with the non-secure parallel version. We build GraphSC and demonstrate, using several algorithms as examples, that secure computation can be brought into the realm of practicality for big data analysis. Our secure matrix factorization implementation can process 1 million ratings in 13 hours, which is a multiple order-of-magnitude improvement over the only other existing attempt, which requires 3 hours to process 16K ratings

    Understanding Malvertising Through Ad-Injecting Browser Extensions

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    Malvertising is a malicious activity that leverages advertising to distribute various forms of malware. Because advertising is the key revenue generator for numerous Internet companies, large ad networks, such as Google, Yahoo and Microsoft, invest a lot of effort to mitigate malicious ads from their ad networks. This drives adversaries to look for alternative methods to deploy malvertising. In this paper, we show that browser extensions that use ads as their monetization strategy often facilitate the deployment of malver-tising. Moreover, while some extensions simply serve ads from ad networks that support malvertising, other extensions maliciously alter the content of visited webpages to force users into installing malware. To measure the extent of these behaviors we developed Expector, a system that automatically inspects and identifies browser extensions that inject ads, and then classifies these ads as malicious or benign based on their landing pages. Using Expector, we auto-matically inspected over 18,000 Chrome browser extensions. We found 292 extensions that inject ads, and detected 56 extensions that participate in malvertising using 16 different ad networks and with a total user base of 602,417

    Inhibition of HERG1 K+ channel protein expression decreases cell proliferation of human small cell lung cancer cells

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    HERG (human ether-à-go-go-related gene) K+ currents fulfill important ionic functions in cardiac and other excitable cells. In addition, HERG channels influence cell growth and migration in various types of tumor cells. The mechanisms underlying these functions are still not resolved. Here, we investigated the role of HERG channels for cell growth in a cell line (SW2) derived from small cell lung cancer (SCLC), a malignant variant of lung cancer. The two HERG1 isoforms (HERG1a, HERG1b) as well as HERG2 and HERG3 are expressed in SW2 cells. Inhibition of HERG currents by acute or sustained application of E-4031, a specific ERG channel blocker, depolarized SW2 cells by 10–15 mV. This result indicated that HERG K+ conductance contributes considerably to the maintenance of the resting potential of about −45 mV. Blockage of HERG channels by E-4031 for up to 72 h did not affect cell proliferation. In contrast, siRNA-induced inhibition of HERG1 protein expression decreased cell proliferation by about 50%. Reduction of HERG1 protein expression was confirmed by Western blots. HERG current was almost absent in SW2 cells transfected with siRNA against HERG1. Qualitatively similar results were obtained in three other SCLC cell lines (OH1, OH3, H82), suggesting that the HERG1 channel protein is involved in SCLC cell growth, whereas the ion-conducting function of HERG1 seems not to be important for cell growth
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