18 research outputs found
Response of three class‐IV major histocompatability complex haplotypes to Eimeria acervulina
No full textA GENE MAPPING BETWEEN THE S AND D REGIONS OF THE H-2 COMPLEX INFLUENCES RESISTANCE TO TRICHINELLA SPIRALIS INFECTIONS OF MICE
No full textImmunofluorescence identification of eosinophil granule major basic protein in the flame figures of Wells' syndrome
No full textGenetic control of immunity to Trichinella spiralis in mice. Response of rapid- and slow-responder strains to immunization with parasite antigens
No full textSummary: Slow-responder C57BL/10 (BIO) mice responded poorly to immunization with muscle larval antigen of Trichinella spiralis showing no accelerated loss of worms from a subsequent challenge infection. In contrast, rapid-responder NIH mice and (BlOxNIH) F, mice developed high levels of immunity after immunization. Lymphocyte proliferation studies showed that immunized BIO mice did respond to in vitro restimulation with antigen, though less well than NIH mice. Failure of B10 mice to respond to immunization did not therefore reflect a failure to recognize larval antigen, a view confirmed by the fact that immunization was achieved using abbreviated enteral infections and, to a smaller extent, by parenterally administered muscle larvae
Genetic control of immunity to Trichinella spiralis in mice. Response of rapid- and slow-responder strains to immunization with parasite antigens
No full textGenetic control of the immune response to Trichinella spiralis: recognition of muscle larval antigens
No full textGenetic control of immunity to Heligmosomoides polygyrus: fixed H-2 E positive but not H-2 negative cells can present antigen to a parasite-specific T cell hybridoma
No full textEffects of trichinellosis on levels of eosinophils, eosinophil major basic protein, creatine kinase and basophils in the guinea pig
No full textGenetic influence on the antibody response to antigens of Schistosoma mansoni in chronically infected mice
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