Data underlying the PhD thesis "Precision nutrition for cardiometabolic health: insights from human dietary intervention studies"

This dataset contains the supplementary tables with fasting and postprandial plasma metabolomics data of chapters 5 and 7&nbsp;from Anouk Gijbels' PhD thesis, "Precision nutrition for cardiometabolic health: insights from human dietary intervention&nbsp;studies". The research in this thesis aimed to contribute to a better understanding of the role of metabolic heterogeneity in&nbsp;response to diet, with a specific focus on tissue-specific insulin resistance. In addition, we aimed to contribute to a better&nbsp;understanding of the mechanisms by which diet affects metabolic health by investigating postprandial metabolism.</p

Data underlying the PhD thesis "Precision nutrition for cardiometabolic health: insights from human dietary intervention studies"

This dataset contains the supplementary tables with fasting and postprandial plasma metabolomics data of chapters 5 and 7&nbsp;from Anouk Gijbels' PhD thesis, "Precision nutrition for cardiometabolic health: insights from human dietary intervention&nbsp;studies". The research in this thesis aimed to contribute to a better understanding of the role of metabolic heterogeneity in&nbsp;response to diet, with a specific focus on tissue-specific insulin resistance. In addition, we aimed to contribute to a better&nbsp;understanding of the mechanisms by which diet affects metabolic health by investigating postprandial metabolism.</p

Data and Code underlying the article Forest Disturbance and Recovery in Peruvian Amazonia

The data and code in this repository can be used to reproduce the analysis Requena Suarez et al. (2023), "Forest Disturbance and Recovery in Peruvian Amazonia". Spatial datasets used in this study are accessible from the sources cited in Table 1 of the main study. Estimation of disturbance and time since disturbance was done using the AVOCADO algorithm (Decuyper et al, 2022, https://doi.org/10.1016/j.rse.2021.112829), and Landsat imagery downloaded from Google Earth Engine. The underlying code for AVOCADO can be found in the following GitHub repository: https://github.com/MDecuy/AVOCADO, as well as a tutorial: https://www.pucv.cl/uuaa/labgrs/proyectos/avocado.&nbsp;</p

Data and Code underlying the article Forest Disturbance and Recovery in Peruvian Amazonia

The data and code in this repository can be used to reproduce the analysis Requena Suarez et al. (2023), "Forest Disturbance and Recovery in Peruvian Amazonia". Spatial datasets used in this study are accessible from the sources cited in Table 1 of the main study. Estimation of disturbance and time since disturbance was done using the AVOCADO algorithm (Decuyper et al, 2022, https://doi.org/10.1016/j.rse.2021.112829), and Landsat imagery downloaded from Google Earth Engine. The underlying code for AVOCADO can be found in the following GitHub repository: https://github.com/MDecuy/AVOCADO, as well as a tutorial: https://www.pucv.cl/uuaa/labgrs/proyectos/avocado.&nbsp;</p

The BRI1-Associated Kinase 1, BAK1, Has a Brassinolide-Independent Role in Plant Cell-Death Control

Programmed cell death (PCD) is a common host response to microbial infection [1], [2] and [3]. In plants, PCD is associated with immunity to biotrophic pathogens, but it can also promote disease upon infection by necrotrophic pathogens [4]. Therefore, plant cell-suicide programs must be strictly controlled. Here we demonstrate that the Arabidopsis thaliana Brassinosteroid Insensitive 1 (BRI1)-associated receptor Kinase 1 (BAK1), which operates as a coreceptor of BRI1 in brassinolide (BL)-dependent plant development, also regulates the containment of microbial infection-induced cell death. BAK1-deficient plants develop spreading necrosis upon infection. This is accompanied by production of reactive oxygen intermediates and results in enhanced susceptibility to necrotrophic fungal pathogens. The exogenous application of BL rescues growth defects of bak1 mutants but fails to restore immunity to fungal infection. Moreover, BL-insensitive and -deficient mutants do not exhibit spreading necrosis or enhanced susceptibility to fungal infections. Together, these findings suggest that plant steroid-hormone signaling is dispensable for the containment of infection-induced PCD. We propose a novel, BL-independent function of BAK1 in plant cell-death control that is distinct from its BL-dependent role in plant development