Addendum to: Search for anomalous top-gluon couplings at LHC revisited

In our latest paper "Search for anomalous top-gluon couplings at LHC revisited" in Eur. Phys. J. C65 (2010), 127-135 (arXiv:0910.3049 [hep-ph]), we studied possible effects of nonstandard top-gluon couplings through the chromoelectric and chromomagnetic moments of the top quark using the total cross section of ppbar/pp --> ttbar X at Tevatron/LHC. There we pointed out that LHC data could give a stronger constraint on those two parameters, which would be hard to obtain from Tevatron data alone. We show here the first CMS measurement of this cross section actually makes it possible.Comment: 5 pages, 1 figure, LaTeX2e, Final version (to appear in Eur. Phys. C

Proposing "b-Parity" - a New Approximate Quantum Number in Inclusive b-jet Production - as an Efficient Probe of New Flavor Physics

We consider the inclusive reaction \ell^+ \ell^- -> nb +X (n = number of b-jets) in lepton colliders for which we propose a useful approximately conserved quantum number b_P=(-1)^n that we call b-Parity (b_P). We make the observation that the Standard Model (SM) is essentially b_P-even since SM b_P-violating signals are necessarily CKM suppressed. In contrast new flavor physics can produce b_P=-1 signals whose only significant SM background is due to b-jet misidentification. Thus, we show that b-jet counting, which relies primarily on b-tagging, becomes a very simple and sensitive probe of new flavor physics (i.e., of b_P-violation).Comment: 5 pages using revtex, 2 figures embadded in the text using epsfig. As will appear in Phys.Rev.Lett.. Considerable improvement was made in the background calculation as compared to version 1, by including purity parameters, QCD effects and 4-jets processe

A Model-independent Description of New Physics effects in e+e- to t tbar

We study the potential of a future $e^+e^-$ collider for the search of anomalous gamma t t bar and Z t t bar couplings, assuming that CP-invariance holds. This is done in a model-independent way, considering that all six possible couplings do appear. Two experimental situations are envisaged, with and without electron beam polarization. Observability limits in the form of domains in the 6-dimensional parameter space are established. Illustrations for specific constrained models are also presented and implications for new physics searches are discussed.Comment: 26 pages and 5 figures. e-mail: [email protected]

Exploring the Eastern Frontier: A First Look at Mobile App Tracking in China

Many mobile apps are integrated with mobile advertising and tracking services running in the background to collect information for tracking users. Considering China currently tops mobile traffic growth globally, this paper aims to take a first look at China’s mobile tracking patterns from a large 4G network. We observe the dominance of the top popular domestic trackers and the pervasive tracking on mobile apps. We also discover a very well-connected tracking community, where the non-popular trackers form many local communities with each community tracking a particular category of mobile apps. We further conclude that some trackers have a monopoly on specific groups of mobile users and 10% of users upload Personally Identifiable Information (PII) to trackers (with 90% of PII tracking flows local to China). Our results consistently show a distinctive mobile tracking market in China. We hope the results can inform users and stakeholders on the interplay between mobile tracking and potential security and privacy issues

Inhibition of DNA damage response at telomeres improves the detrimental phenotypes of Hutchinson–Gilford Progeria Syndrome

Hutchinson–Gilford progeria syndrome (HGPS) is a genetic disorder characterized by premature aging features. Cells from HGPS patients express progerin, a truncated form of Lamin A, which perturbs cellular homeostasis leading to nuclear shape alterations, genome instability, heterochromatin loss, telomere dysfunction and premature entry into cellular senescence. Recently, we reported that telomere dysfunction induces the transcription of telomeric non-coding RNAs (tncRNAs) which control the DNA damage response (DDR) at dysfunctional telomeres. Here we show that progerin-induced telomere dysfunction induces the transcription of tncRNAs. Their functional inhibition by sequence-specific telomeric antisense oligonucleotides (tASOs) prevents full DDR activation and premature cellular senescence in various HGPS cell systems, including HGPS patient fibroblasts. We also show in vivo that tASO treatment significantly enhances skin homeostasis and lifespan in a transgenic HGPS mouse model. In summary, our results demonstrate an important role for telomeric DDR activation in HGPS progeroid detrimental phenotypes in vitro and in vivo

CP violation in gauge theories

We define the CP transformation properties of scalars, fermions and vectors in a gauge theory and show that only three types of interactions can lead to CP violation: scalar interactions, fermion-scalar interactions and $F \tilde F$ associated with the strong CP problem and which involve only the gauge fields. For technicolor theories this implies the absence of CP violation within perturbation theory.Comment: 5 pages, 1 figure, revtex and epsf require

Dimension-Six Terms in the Standard Model Lagrangian

When the Standard Model is considered as an effective low-energy theory, higher dimensional interaction terms appear in the Lagrangian. Dimension-six terms have been enumerated in the classical article by Buchmueller and Wyler [3]. Although redundance of some of those operators has been already noted in the literature, no updated complete list has been published to date. Here we perform their classification once again from the outset. Assuming baryon number conservation, we find 15 + 19 + 25 = 59 independent operators (barring flavour structure and Hermitian conjugations), as compared to 16 + 35 + 29 = 80 in Ref.[3]. The three summed numbers refer to operators containing 0, 2 and 4 fermion fields. If the assumption of baryon number conservation is relaxed, 4 new operators arise in the four-fermion sector.Comment: 16 pages, no figures, v3: Redundant B-violating operator remove

Four-Fermi Effective Operators in Top-Quark Production and Decay

Effects of four-Fermi-type new interactions are studied in top-quark pair production and their subsequent decays at future e^+e^- colliders. Secondary-lepton-energy distributions are calculated for arbitrary longitudinal beam polarizations. An optimal-observables procedure is applied for the determination of new parameters.Comment: Polarized e^- plus unpolarized e^+ collisions were include

Anomalous Neutrino Reactions at HERA

We study the sensitivity of HERA to new physics using the helicity suppressed reaction $e_R p \rightarrow \nu X$, where the final neutrino can be a standard model one or a heavy neutrino. The approach is model independent and is based on an effective lagrangian parametrization. It is shown that HERA will put significant bounds on the scale of new physics, though, in general, these are more modest than previously thought. If deviations from the standard model are observed in the above processes, future colliders such as the SSC and LHC will be able to directly probe the physics responsible for these discrepancies}Comment: 11 Pages + 2 figures is TOPDRAWER (included at the end or available by mail). Report UCRHEP-T113 (requires the macropackage PHYZZX). A line in the TeX file requesting an input file has been removed, it caused problem

Co-Expression of Wild-Type P2X7R with Gln460Arg Variant Alters Receptor Function

The P2X7 receptor is a member of the P2X family of ligand-gated ion channels. A single-nucleotide polymorphism leading to a glutamine (Gln) by arginine (Arg) substitution at codon 460 of the purinergic P2X7 receptor (P2X7R) has been associated with mood disorders. No change in function (loss or gain) has been described for this SNP so far. Here we show that although the P2X7R-Gln460Arg variant per se is not compromised in its function, co-expression of wild-type P2X7R with P2X7R-Gln460Arg impairs receptor function with respect to calcium influx, channel currents and intracellular signaling in vitro. Moreover, co-immunoprecipitation and FRET studies show that the P2X7R-Gln460Arg variant physically interacts with P2X7R-WT. Specific silencing of either the normal or polymorphic variant rescues the heterozygous loss of function phenotype and restores normal function. The described loss of function due to co-expression, unique for mutations in the P2RX7 gene so far, explains the mechanism by which the P2X7R-Gln460Arg variant affects the normal function of the channel and may represent a mechanism of action for other mutations.Fil: Aprile García, Fernando. Universidad de Buenos Aires. Facultad de Ciencias Exactas y Naturales. Departamento de Fisiología, Biología Molecular y Celular; Argentina. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; ArgentinaFil: Metzger, Michael W.. Max Planck Institute of Psychiatry; AlemaniaFil: Paez Pereda, Marcelo. Max Planck Institute of Psychiatry; AlemaniaFil: Stadler, Herbert. Affectis Pharmaceuticals; AlemaniaFil: Acuña, Matias. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; ArgentinaFil: Liberman, Ana Clara. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; ArgentinaFil: Senin, Sergio Ariel. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; ArgentinaFil: Gerez, Juan Atilio. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; ArgentinaFil: Hoijman, Esteban. Universidad de Buenos Aires. Facultad de Ciencias Exactas y Naturales. Centro de Microscopías Avanzadas; Argentina. Consejo Nacional de Investigaciones Científicas y Técnicas; ArgentinaFil: Refojo, Damian. Max Planck Institute of Psychiatry; Alemania. Consejo Nacional de Investigaciones Científicas y Técnicas; ArgentinaFil: Mitkovski, Mišo. Max Planck Institute of Experimental Medicine; AlemaniaFil: Panhuysen, Markus. Affectis Pharmaceuticals; AlemaniaFil: Stühmer, Walter. Max Planck Institute of Experimental Medicine; AlemaniaFil: Holsboer, Florian. Max Planck Institute of Psychiatry; Alemania. HMNC Brain Health; AlemaniaFil: Deussing, Jan M.. Max Planck Institute of Psychiatry; AlemaniaFil: Arzt, Eduardo Simon. Max Planck Institute of Psychiatry; Alemania. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Parque Centenario. Instituto de Investigación en Biomedicina de Buenos Aires - Instituto Partner de la Sociedad Max Planck; Argentina. Universidad de Buenos Aires. Facultad de Ciencias Exactas y Naturales. Departamento de Fisiología, Biología Molecular y Celular; Argentin