A 12 GHz satellite video receiver: Low noise, low cost prototype model for TV reception from broadcast satellites

A 12-channel synchronous phase lock video receiver consisting of an outdoor downconverter unit and an indoor demodulator unit was developed to provide both low noise performance and low cost in production quantities of 1000 units. The prototype receiver can be mass produced at a cost under $1540 without sacrificing system performance. The receiver also has the capability of selecting any of the twelve assigned satellite broadcast channels in the frequency range 11.7 to 12.2 GHz

Predator-Induced Vertical Behavior of a Ctenophore

Although many studies have focused on Mnemiopsis leidyi predation, little is known about the role of this ctenophore as prey when abundant in native and invaded pelagic systems. We examined the response of the ctenophore M. leidyi to the predatory ctenophore Beroe ovata in an experiment in which the two species could potentially sense each other while being physically separated. On average, M. leidyi responded to the predator’s presence by increasing variability in swimming speeds and by lowering their vertical distribution. Such behavior may help explain field records of vertical migration, as well as stratified and near-bottom distributions of M. leidyi

Surrogate Measurement of the \u3csup\u3e238\u3c/sup\u3ePu(\u3cem\u3en,f\u3c/em\u3e\u3c/em\u3e) Cross Section

The neutron-induced fission cross section of 238Pu was determined using the surrogate ratio method. The (n,f) cross section over an equivalent neutron energy range 5–20 MeV was deduced from inelastic α-induced fission reactions on 239Pu, with 235U(α,α′f) and 236U(α,α′f) used as references. These reference reactions reflect 234U(n,f) and 235U(n,f) yields, respectively. The deduced 238Pu(n,f) cross section agrees well with standard data libraries up to ~10 MeV, although larger values are seen at higher energies. The difference at higher energies is less than 20%

On the genetic involvement of apoptosis-related genes in Crohn's disease as revealed by an extended association screen using 245 markers: no evidence for new predisposing factors

Crohn's disease (CD) presents as an inflammatory barrier disease with characteristic destructive processes in the intestinal wall. Although the pathomechanisms of CD are still not exactly understood, there is evidence that, in addition to e.g. bacterial colonisation, genetic predisposition contributes to the development of CD. In order to search for predisposing genetic factors we scrutinised 245 microsatellite markers in a population-based linkage mapping study. These microsatellites cover gene loci the encoded protein of which take part in the regulation of apoptosis and (innate) immune processes. Respective loci contribute to the activation/suppression of apoptosis, are involved in signal transduction and cell cycle regulators or they belong to the tumor necrosis factor superfamily, caspase related genes or the BCL2 family. Furthermore, several cytokines as well as chemokines were included. The approach is based on three steps: analyzing pooled DNAs of patients and controls, verification of significantly differing microsatellite markers by genotyping individual DNA samples and, finally, additional reinvestigation of the respective gene in the region covered by the associated microsatellite by analysing single-nucleotide polymorphisms (SNPs). Using this step-wise process we were unable to demonstrate evidence for genetic predisposition of the chosen apoptosis- and immunity-related genes with respect to susceptibility for CD

Photoemission analysis of chemically modified TlBr surfaces for improved radiation detectors

Abstract not provide

Methylation of class II transactivator gene promoter IV is not associated with susceptibility to Multiple Sclerosis

<p>Abstract</p> <p>Background</p> <p>Multiple sclerosis (MS) is a complex trait in which alleles at or near the class II loci <it>HLA-DRB1 </it>and <it>HLA-DQB1 </it>contribute significantly to genetic risk. The MHC class II transactivator (<it>MHC2TA</it>) is the master controller of expression of class II genes, and methylation of the promoter of this gene has been previously been shown to alter its function. In this study we sought to assess whether or not methylation of the <it>MHC2TA </it>promoter pIV could contribute to MS disease aetiology.</p> <p>Methods</p> <p>In DNA from peripheral blood mononuclear cells from a sample of 50 monozygotic disease discordant MS twins the <it>MHC2TA </it>promoter IV was sequenced and analysed by methylation specific PCR.</p> <p>Results</p> <p>No methylation or sequence variation of the <it>MHC2TA </it>promoter pIV was found.</p> <p>Conclusion</p> <p>The results of this study cannot support the notion that methylation of the pIV promoter of <it>MHC2TA </it>contributes to MS disease risk, although tissue and timing specific epigenetic modifications cannot be ruled out.</p

Interaction Analysis between HLA-DRB1 Shared Epitope Alleles and MHC Class II Transactivator CIITA Gene with Regard to Risk of Rheumatoid Arthritis

HLA-DRB1 shared epitope (SE) alleles are the strongest genetic determinants for autoantibody positive rheumatoid arthritis (RA). One of the key regulators in expression of HLA class II receptors is MHC class II transactivator (CIITA). A variant of the CIITA gene has been found to associate with inflammatory diseases

Using global team science to identify genetic parkinson's disease worldwide.

No abstract available

Company-level family policies: Who has access to it and what are some of its outcomes

Despite the increase in number of studies that examine the cross-national variation in the policy configuration that allow a better work-family integration, very few look beyond the national levels. It is also crucial to examine occupational level welfare since companies may restrict or expand the existing national level regulations, defining the “final availability” workers actual have towards various arrangements. In addition, companies may provide various additional arrangements through occupational policies which are not set out in the national level agreements that are crucial in addressing reconciliation needs of workers. This chapter examines what types of arrangements are provided at the company level to address work-family demands of workers. It further provides a synthesis of studies that examine both national level contexts and individual level characteristics that explain who gets access to company level family-friendly policies, which is linked to the possible outcomes of these policies