Evaluation of a low-phosphate cow's milk diet on growth and bone mineralization of full-term infants

Growth, bone mineralization and intestinal absorption were studied in 16 infants fed low-phosphate cow's milk and compared with a group of 15 infants fed conventional cow's milk. In the low-phosphate-fed infants the intestinal calcium absorption was as high as in infants fed human milk, the main characteristics of growth and bone mineralization being similar to those of infants fed conventional cow's milk. Urinary calcium was elevated with a low cAMP excretion. The relative hypercalciuria might be the result of the amount absorbed in excess of that used for the bone accretion.SCOPUS: cp.jinfo:eu-repo/semantics/publishe

Renal acidification process in vitamin D deficiency

SCOPUS: NotDefined.jinfo:eu-repo/semantics/publishe

Aspects physiopathologiques de la carence en vitamine D

Doctorat en sciences médicalesinfo:eu-repo/semantics/nonPublishe

Effect of thyrocalcitonin on renal reabsorption of bicarbonate

Porcine thyrocalcitonin (TCT) given intramuscularly at a dose of 2-3 MRC units/kg body weight induces a renal bicarbonate wasting probably at the level of the proximal tubule. This effect is found in normal children as well as in patients with renal tubular acidosis and with vitamin D deficiency. The mechanism by which TCT acts on the acidification mechanism is probably independent of parathyroid hormone (PTH) because the effect could also be obtained in hypoparathyroid patients.SCOPUS: ar.jinfo:eu-repo/semantics/publishe

LES RACHITISMES RESISTANT AUX HORMONES CALCIOTROPES

Resistance to vitamin D has been known of for a long time but was erroneously ascribed to phosphate deficiency (phosphate diabetes or deficiency in tubular phosphate reabsorption). True vitamin D resistance can either be due to lack of the receptor itself or to structural abnormality in the hormone- or DAN-binding site of the receptor whereas in a few cases post-receptor resistance has been documented. In view of the therapeutic consequences, diagnosis of vitamin D resistance is needed in every case of rickets not due to simple vitamin D deficiency. Such cases can be easily recognized by normal 25-hydroxyvitamin D concentration in the presence of biochemical, radiological and/or histological signs of rickets.SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Pseudohypoparathyroidism (PHP) secondary to vitamin D deficiency rickets

SCOPUS: NotDefined.jinfo:eu-repo/semantics/publishe

LES ANTICOAGULANTS DANS L'INFARCTUS DU MYOCARDE

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Proximal renal tubular acidosis in vitamin D deficiency rickets

A proximal renal tubular acidosis (RTA) is the mechanism underlying the systemic acidosis found in vitamin D deficiency rickets. Acidotic subjects have high levels of parathyroid hormone (PTH). In non acidotic subjects proximal bicarbonate wasting can be induced by exogenous PTH injection. Carbonic anhydrase activity is not involved in this process. Calcium infusion is able to suppress both the spontaneous and the PTH induced bicarbonate leak. The development of RTA in vitamin D deficiency is related to a particular equilibrium between two antagonizing factors at tubular level, parathyroid hormone and calcium. (35 references).SCOPUS: ar.jinfo:eu-repo/semantics/publishe

A new form of pseudohypoparathyroidism type II with abnormal response to TRH

SCOPUS: NotDefined.jinfo:eu-repo/semantics/publishe

LA CALCINOSE IDIOPATHIQUE

SCOPUS: ar.jinfo:eu-repo/semantics/publishe