Electron and neutron electric dipole moment in the 3-3-1 model with heavy leptons

We calculate the electric dipole moment for the electron and neutron in the framework of the 3-3-1 model with heavy charged leptons. We assume that the only source of $CP$ violation arises from a complex trilinear coupling constant and the three complex vacuum expectation values. However, two of the vacua phases are absorbed and the other two are equal up to a minus sign. Hence only one physical phase survives. In order to be compatible with the experimental data this phase has to be smaller than $10^{-6}$.Comment: 21 pages, 4 figures. Version published in PR

Electron and muon anomalous magnetic dipole moment in a 3-3-1 model

We calculate, in the context of a 3-3-1 model with heavy charged leptons, constraints on some parameters of the extra particles in the model by imposing that their contributions to both the electron and muon $(g-2)$ factors are in agreement with experimental data up to 1$\sigma$-3$\sigma$. In order to obtain realistic results we use some of the possible solutions of the left- and right- unitary matrices that diagonalize the lepton mass matrices, giving the observed lepton masses and at the same time allowing to accommodate the Pontecorvo-Maki-Nakagawa-Sakata (PMNS) mixing matrix. We show that, at least up to 1-loop order, in the particular range of the space parameter that we have explored, it is not possible to fit the observed electron and muon $(g-2)$ factors at the same time unless one of the extra leptons has a mass of the order of 20-40 GeVs and the energy scale of the 331 symmetry to be of around 60-80 TeVs.Comment: 34 pages, 19 figure

Aplicativo com análise de rentabilidade para sistemas de produção de florestas cultivadas e de grãos.

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Host-cell dependent role of phosphorylated keratin 8 during influenza A/NWS/33 virus (H1N1) infection in mammalian cells

In this study, we investigated the involvement of keratin 8 during human influenza A/NWS/33 virus (H1N1) infection in semi-permissive rhesus monkey-kidney (LLC-MK2) and permissive human type II alveolar epithelial (A549) cells. In A549 cells, keratin 8 showed major expression and phosphorylation levels. Influenza A/NWS/33 virus was able to subvert keratin 8 structural organization at late stages of infection in both cell models, promoting keratin 8 phosphorylation in A549 cells at early phases of infection. Accordingly, partial colocalizations of the viral nucleoprotein with keratin 8 and its phosphorylated form were assessed by confocal microscopy at early stages of infection in A549 cells. The employment of chemical activators of phosphorylation resulted in structural changes as well as increased phosphorylation of keratin 8 in both cell models, favoring the influenza A/NWS/33 virus's replicative efficiency in A549 but not in LLC-MK2 cells. In A549 and human larynx epidermoid carcinoma (HEp-2) cells inoculated with respiratory secretions from pediatric patients positive for, respectively, influenza A virus or respiratory syncytial virus, the keratin 8 phosphorylation level had increased only in the case of influenza A virus infection. The results obtained suggest that in A549 cells the influenza virus is able to induce keratin 8 phosphorylation thereby enhancing its replicative efficiency