SYNTAX scale estimates of coronary stenosis severity and outcomes of various revascularisation strategies in patients with ST segment elevation myocardial infarction and multi-vessel coronary pathology

Aim. To assess 30-day and long-term outcomes of various revascularisation strategies in patients with myocardial infarction and ST segment elevation (STEMI) and multi-vessel coronary pathology (MVCP), in regard to the severity of coronary stenosis, as assessed by the SYNTAX scale. Material and methods. The 30-day and long-term outcomes of various strategies of primary percutaneous coronary intervention (PCI) were assessed in 227 STEMI patients, in regard to the severity of their coronary stenosis (as assessed by the SYNTAX scale). Group 1 included 40 patients who underwent multi-vessel stenting (MVS) as a part of their primary PCI, while Group 2 included 187 patients with indications for staged revascularisation (SR). Results. At baseline, the MVS and SR subgroups with severe coronary stenosis (SYNTAX scor

COMPARATIVE CHARACTERISTICS OF FUNCTIONAL CORONARY RESERVE AND MYOCARDIAL INFARCTION RISK IN PATIENTS WITH THE CARDIAC X SYNDROME AND STENOTIC CORONARY ATHEROSCLEROSIS

The aim of this research was to study the functional coronary reserve and frequency of miocardium infarction in patients with cardiologic syndrome X (CX) in comparison with patients with stenosing atherosclerosis of coronary artery (ACA). 54 patients with CX and 83 patients with ACA were examined. Coronaroventriculography,veloergometry, echo, daily ECG monitoring were conducted. It was proved that functional coronary reserve in CX patients is lower than in ACA patients. However risk of miocardial infarction development is higher in ACA patients. In the first place, functional coronary reserve depends on state of terminal sectors of coronary arterial channel both in CX and ACA patients. At the same time risk of miocardial infarction development is above all conditioned by damage of subepicardial arteries, i.e. presence of atheromas in them and relatively little depends on volume of coronary channel’s damage