Experimental study of photon beam polarimeter based on nuclear e+e- pair production in an amorphous target

The experimental method of the linearly polarized photons polarimetry,using incoherent e+e- pair production process has been investigated on the beam of coherent bremsstrahlung (CB) photons in the energy range of 0.9-1.1 GeV at the Yerevan synchrotron.Comment: 6 pages (text),10 figure

Pauli blocking and final-state interaction in electron-nucleus quasielastic scattering

The nucleon final-state interaction in inclusive electron-nucleus quasielastic scattering is studied. Based on the unitarity equation satisfied by the scattering-wave operators, a doorway model is developed to take into account the final-state interaction including the Pauli blocking of nucleon knockout. The model uses only experimental form factors as the input and can be readily applied to light- and medium-mass nuclei. Pauli blocking effects in these latter nuclei are illustrated with the case of the Coulomb interaction. Significant effects are noted for beam energies below ~ 350 MeV and for low momentum transfers.Comment: 16 pages, 6 figure

Neutrino-nucleus reactions on ^{12}C and ^{16}O

Exclusive and inclusive $(\nu_\mu, \mu^-), (\nu_e, e^-)$ cross-sections and $\mu^-$-capture rates are calculated for ^{12}C and ^{16}O using the consistent random phase approximation (RPA) and pairing model. After a pairing correction is introduced to the RPA results the flux-averaged theoretical $(\nu_\mu, \mu^-), (\nu_e, e^-)$ cross-sections and $\mu^-$-capture rates in $^{12}$C are in good agreement with experiment. In particular when one takes into account the experimental error bars, the recently measured range of values for the $(\nu_\mu, \mu^-)$ cross-section is in agreement with the present theoretical results. Predictions of $(\nu_\mu, \mu^-)$ and $(\nu_e, e^-)$ cross-sections in ^{16}O are also presented.Comment: 13 pages, Revte

Muon capture by 3He nuclei followed by proton and deuteron production

The paper describes an experiment aimed at studying muon capture by ${}^{3}\mathrm{He}$ nuclei in pure ${}^{3}\mathrm{He}$ and $\mathrm{D}_2 + {}^{3}\mathrm{He}$ mixtures at various densities. Energy distributions of protons and deuterons produced via $\mu^-+{}^{3}\mathrm{He}\to p+n+n + \nu_{\mu }$ and $\mu^-+{}^{3} \mathrm{He} \to d+n + \nu_{\mu}$ are measured for the energy intervals $10 - 49$ MeV and $13 - 31$ MeV, respectively. Muon capture rates, $\lambda_\mathrm{cap}^p (\Delta E_p)$ and $\lambda_\mathrm{cap}^d (\Delta E_d)$ are obtained using two different analysis methods. The least--squares methods gives $\lambda_\mathrm{cap}^p = (36.7\pm 1.2) {s}^{- 1}$, $\lambda_\mathrm{cap}^d = (21.3 \pm 1.6) {s}^{- 1}$. The Bayes theorem gives $\lambda_\mathrm{cap}^p = (36.8 \pm 0.8) {s}^{- 1}$, $\lambda_\mathrm{cap}^d = (21.9 \pm 0.6) {s}^{- 1}$. The experimental differential capture rates, $d\lambda_\mathrm{cap}^p (E_p) / dE_p$ and $d\lambda_\mathrm{cap}^d (E_d) / dE_d$, are compared with theoretical calculations performed using the plane--wave impulse approximation (PWIA) with the realistic NN interaction Bonn B potential. Extrapolation to the full energy range yields total proton and deuteron capture rates in good agreement with former results.Comment: 17 pages, 13 figures, accepted for publication in PR

Transcriptional activation of c-fos by oncogenic Ha-Ras in mouse mammary epithelial cells requires the combined activities of PKC-lambda, epsilon and zeta.

The implication of protein kinase C (PKC) isoforms cPKC-alpha, nPKC-epsilon, aPKC-lambda and aPKC-zeta in the transcriptional activation of a c-fos promoter-driven CAT-reporter construct by transforming Ha-Ras has been investigated. This was achieved by employing antisense constructs encoding RNA directed against isoform-specific 5' sequences of the corresponding mRNA, and expression of PKC mutants representing either kinase-defective, dominant negative, or constitutively active forms of the PKC isoforms. The data indicate that in HC11 mouse mammary epithelial cells, transforming Ha-Ras requires the activities of the three PKC isozymes: aPKC-lambda, nPKC-epsilon and aPKC-zeta, not, however, of cPKC-alpha, for the transcriptional activation of c-fos. Co-expression of oncogenic Ha-Ras with combinations of kinase-defective, dominant negative and constitutively active mutants of the various PKC isozymes are in agreement with a tentative model suggesting that, in the signaling pathway from Ha-Ras to the c-fos promoter, aPKC-lambda acts upstream whereas aPKC-zeta functions downstream of nPKC-epsilon