10 research outputs found
A fucoidan plant drink reduces Helicobacter pylori load in the stomach: a real-world study
- Publication venue
- AME Publishing Company
- Publication date
- 30/10/2023
- Field of study
Get PDFBACKGROUND: Helicobacter pylori (Hp) infection is highly prevalent globally and is predominantly managed by antibiotics. Recently, the anti-adhesive, antioxidant, antitoxin, immunomodulatory, anti-coagulant, and anti-infective activities of fucoidan, a polysaccharide extracted from brown seaweeds, have been widely studied, and the results showed promise. Fucoidan has the potential to be utilized in Hp eradication therapy. Our present clinical study was designed to evaluate the efficiency of Lewuyou®, a fucoidan plant drink (FPD) in eradicating Hp in humans. METHODS: This multi-center, clinical study was conducted between October 2020 and July 2021. Hp infection was confirmed by urea breath test (UBT). A total of 122 patients with confirmed Hp infection were enrolled; after exclusion of incomplete data, 85 eligible patients (37 males and 48 females aged 20–81 years) were included in the final analysis. FPD (50 mL per vial) was orally administered twice daily for a 4-week cycle, and 41 patients completed an 8-week cycle. RESULTS: No adverse event (AE) was reported in all 122 participants who had consumed FPD. The Hp eradication rate and clearance rate were 77.6% (66/85) and 20.0% (17/85), respectively, after 4 weeks of FPD consumption and 80.5% (33/41) and 26.8 (11/41) , respectively, after 8 weeks of consumption. CONCLUSIONS: The 4- and 8-week protocols of FPD consumption were safe and effective at reducing Hp load on the gastric mucosa, with Hp eradicated in the majority of participants
New insights into the genetic etiology of Alzheimer's disease and related dementias
- Author
- Aaltonen L.
- Aaltonen V.
- Aarsland Dag
- Aavikko M.
- Abalos M.S.
- Abdelnour Carla
- Abner E.
- Abraham R.
- Adams H.
- Adams P.M.
- Adarmes-Gómez A.
- Adarmes-Gómez A.D.
- Aguilera N.
- Aguilera N.
- Aguirre A.
- Ahmad S.
- Akinyemi R.O.
- Al-Chalabi A.
- Alarcón-Martín Emilio
- Albert M.S.
- Albin R.L.
- Alcolea Daniel
- Alegret Montserrat
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Get PDFCharacterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE ε4 allele
Application of Real Option Theory to Mergers and Acquisitions (M&A) in the Pharmaceutical Industry
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- 'Elsevier BV'
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- Albert
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- D.E. Arking
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- Elkins
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- Joy
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- Klimkowicz-Mrowiec
- Knopman
- Kronfol
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- Rothwell
- Rozzini
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- Whalley
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- Zandi
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- Campbell I.H.
- Cao Q.
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Full text linkEncouraging Regeneration of Host Neurons:
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- A. Bignami
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- 'Springer Science and Business Media LLC'
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- Field of study
Full text linkSearch for a massless particle beyond the Standard Model in the Σ+ → p + invisible decay
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- A. Amoroso
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- A. Calcaterra
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- S.J. Wang
- S.J. Zhao
- S.K. Choi
- S.L. Chen
- S.L. Hu
- S.L. Olsen
- S.M. Chen
- S.N. Ruan
- S.P. Wen
- S.Q. Zhu
- S.S. Fang
- S.S. Jiang
- S.S. Sun
- S.X. Du
- S.X. Li
- S.Y. Shi
- S.Y. Xiao
- Shuihan Zhang
- Shulei Zhang
- T. Chen
- T. Holtmann
- T. Hu
- T. Hussain
- T. Johansson
- T. Lenz
- T. Li
- T. Lin
- T. Liu
- T. Luo
- T. Ma
- T. Sun
- T. Wang
- T. Xiang
- T. Yu
- T.J. Jiang
- T.J. Min
- T.J. Wang
- T.J. Zhu
- T.T. Han
- T.T. Lei
- T.Y. Qi
- T.Y. Xing
- T.Z. Song
- Tao Yang
- U. Wiedner
- V. Batozskaya
- V. Khachatryan
- V. Prasad
- V. Thoren
- W. Gradl
- W. Ji
- W. Kühn
- W. Shan
- W. Wang
- W. Wang
- W. Xu
- W.B. Qian
- W.B. Yan
- W.C. Yan
- W.D. Li
- W.D. Niu
- W.D. Zhu
- W.G. Li
- W.H. Shen
- W.H. Tian
- W.J. Zheng
- W.K. Liu
- W.L. Xu
- W.M. Liu
- W.M. Song
- W.P. Wang
- W.X. Fang
- W.X. Gong
- W.Y. Sun
- X. Cai
- X. Dong
- X. Kui
- X. Li
- X. Liu
- X. Liu
- X. Pan
- X. Shi
- X. Wang
- X. Wu
- X. Zeng
- X. Zhong
- X. Zhou
- X.B. Gao
- X.B. Ji
- X.C. Ai
- X.C. Lou
- X.D. Yu
- X.D. Zhang
- X.F. Wang
- X.H. Li
- X.H. Mo
- X.H. Wu
- X.H. Xie
- X.J. Wang
- X.K. Qiao
- X.K. Zhou
- X.L. Ji
- X.L. Kang
- X.L. Li
- X.L. Lu
- X.L. Luo
- X.L. Wang
- X.M. Jing
- X.M. Xian
- X.M. Zhang
- X.N. Wang
- X.P. Xu
- X.Q. Hao
- X.Q. Jia
- X.Q. Yan
- X.R. Chen
- X.R. Lyu
- X.R. Zhou
- X.S. Jiang
- X.S. Kang
- X.S. Qin
- X.T. Chen
- X.T. Hou
- X.T. Huang
- X.T. Ma
- X.X. Ding
- X.Y. Ma
- X.Y. Shan
- X.Y. Shen
- X.Y. Zhai
- X.Y. Zhang
- X.Y. Zhou
- X.Z. Li
- Xiaoyu Li
- Y. Bai
- Y. Ban
- Y. Ding
- Y. Ding
- Y. Fang
- Y. Hu
- Y. Jiang
- Y. Jin
- Y. Liu
- Y. Liu
- Y. Lu
- Y. Ma
- Y. Nefedov
- Y. Niu
- Y. Pan
- Y. Schelhaas
- Y. Sun
- Y. Tian
- Y. Wan
- Y. Wang
- Y. Wu
- Y. Xie
- Y. Yang
- Y. Yuan
- Y. Zeng
- Y. Zhang
- Y.A. Tang
- Y.B. Chen
- Y.B. Liu
- Y.B. Zhao
- Y.C. Xu
- Y.C. Yu
- Y.C. Zhai
- Y.C. Zhu
- Y.D. Wang
- Y.F. Liang
- Y.F. Lyu
- Y.F. Wang
- Y.F. Yang
- Y.G. Li
- Y.G. Xie
- Y.H. Fan
- Y.H. Wu
- Y.H. Xie
- Y.H. Zhan
- Y.H. Zhang
- Y.H. Zheng
- Y.J. Mao
- Y.J. Song
- Y.J. Su
- Y.J. Sun
- Y.J. Wu
- Y.J. Yuan
- Y.J. Zeng
- Y.K. Heng
- Y.L. Wang
- Y.L. Xiao
- Y.M. Ma
- Y.M. Zhang
- Y.N. Gao
- Y.N. Wang
- Y.P. Guo
- Y.P. Huang
- Y.P. Lu
- Y.P. Pei
- Y.Q. Chen
- Y.Q. Fang
- Y.Q. Wang
- Y.R. Hou
- Y.R. Wen
- Y.T. Feng
- Y.T. Gu
- Y.T. Liang
- Y.T. Zhang
- Y.W. Fu
- Y.X. Song
- Y.X. Yang
- Y.X. Zhao
- Y.Y. Ji
- Y.Y. Peng
- Y.Z. Sun
- Y.Z. Zhou
- Yan Zhang
- Yang Gao
- Yao Zhang
- Yaqian Wang
- Yi Wang
- Yifan Yang
- Z. Jiao
- Z. Ning
- Z. Wang
- Z. Wu
- Z.A. Liu
- Z.A. Zhu
- Z.D. Liu
- Z.D. Zhang
- Z.F. Tian
- Z.G. Zhao
- Z.H. Duan
- Z.H. Lei
- Z.H. Lu
- Z.H. Qin
- Z.H. Qu
- Z.H. Zhang
- Z.J. Chen
- Z.J. Li
- Z.J. Shang
- Z.J. Xiao
- Z.K. Jia
- Z.L. Guan
- Z.L. Hou
- Z.L. Wang
- Z.L. Zhang
- Z.P. Mao
- Z.P. Xie
- Z.P. Xu
- Z.P. Yao
- Z.Q. Liu
- Z.Q. Sun
- Z.S. Xu
- Z.T. Sun
- Z.W. Ge
- Z.W. Yang
- Z.X. Li
- Z.X. Meng
- Z.Y. Chen
- Z.Y. Deng
- Z.Y. Wang
- Z.Y. You
- Z.Y. Yuan
- Z.Y. Zhang
- Z.Y. Zhang
- Z.Z. Zhang
- Ziyi Wang
- Publication venue
- Elsevier
- Publication date
- 01/05/2024
- Field of study
Full text linkA massless particle beyond the Standard Model is searched for in the two-body decay Σ+→p+invisible using (1.0087±0.0044)×1010 J/ψ events collected at a center-of-mass energy of s=3.097GeV with the BESIII detector at the BEPCII collider. No significant signal is observed, and the upper limit on the branching fraction B(Σ+→p+invisible) is determined to be 3.2×10−5 at the 90% confidence level. This is the first search for a flavor-changing neutral current process with missing energy in hyperon decays which plays an important role in constraining new physics models
New insights into the genetic etiology of Alzheimer’s disease and related dementias
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
Full text linkCharacterization of the genetic landscape of Alzheimer’s disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/‘proxy’ AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE ε4 allele. © 2022, The Author(s)
