Equivalence between non-bilinear spin-SS Ising model and Wajnflasz model

We propose the mapping of polynomial of degree 2S constructed as a linear combination of powers of spin-$S$ (for simplicity, we called as spin-$S$ polynomial) onto spin-crossover state. The spin-$S$ polynomial in general can be projected onto non-symmetric degenerated spin up (high-spin) and spin down (low-spin) momenta. The total number of mapping for each general spin-$S$ is given by $2(2^{2S}-1)$. As an application of this mapping, we consider a general non-bilinear spin-$S$ Ising model which can be transformed onto spin-crossover described by Wajnflasz model. Using a further transformation we obtain the partition function of the effective spin-1/2 Ising model, making a suitable mapping the non-symmetric contribution leads us to a spin-1/2 Ising model with a fixed external magnetic field, which in general cannot be solved exactly. However, for a particular case of non-bilinear spin-$S$ Ising model could become equivalent to an exactly solvable Ising model. The transformed Ising model exhibits a residual entropy, then it should be understood also as a frustrated spin model, due to competing parameters coupling of the non-bilinear spin-$S$ Ising model

Quality of work life as a mediator between emotional labor and work family interference

10.1007/s10869-009-9103-7Journal of Business and Psychology243245-25

Efficient long-distance quantum communication using microtoroidal resonators

Based on the interaction between a three-level system and a microtoroidal resonator, we present a scheme for long-distance quantum communication in which entanglement generation with near 0.5 success probability and swaps can be implemented by accurate state detection via measuring about 100 photons. With this scheme the average time of successful entanglement distribution over 2500 km with high fidelity can be decreased to only about 30 ms

The Influence of Emotional Intelligence and Affectivity on Emotional Labor Strategies at Work

10.1027/1614-0001.30.2.75Journal of Individual Differences30275-8

The effect of emotional dissonance and emotional intelligence on work-family interference

10.1037/a0025798Canadian Journal of Behavioural Science44150-5

Comparison of two bioassay methods for determining deltamethrin resistance in German cockroaches (blattodea: blattellidae)

Journal of Economic Entomology933905-910JEEN

Bioactive responses of Hep-G2 cells to soyasaponin extracts differs with respect to extraction conditions

10.1016/j.fct.2009.06.006Food and Chemical Toxicology4792202-2208FCTO

Green tea catechin inhibits ephrin-A1-mediated cell migration and angiogenesis of human umbilical vein endothelial cells

Angiogenesis, the formation of new blood vessels from preexisting capillaries, is essential for tumor progression and metastasis. During tumor neovascularization, vascular endothelial growth factor and ephrin (Eph) families emerge as critical mediators of angiogenesis. The green tea catechin epigallocatechin gallate (EGCG), a tyrosine kinase inhibitor, has been demonstrated in previous studies to be an effective anti angiogenesis agent. However, the inhibitory effect of green tea catechins on ephrin-A1-mediated tumor angiogenesis has not been demonstrated yet. Thus, in this study, we investigated the molecular mechanism of ephrin-A1-mediated cell migration and angiogenesis, as well as the inhibitory effects of EGCG. Here we show that ephrin-A1 mediates endothelial cell migration and regulates vascular remodeling in tumor neovascularization in vitro. We also demonstrated that ephrin-A1-mediated cell migration required the activation of extracellular-regulated kinase (ERK-1/2) but not of phosphatidylinositol-3-kinase. The green tea catechin EGCG inhibited ephrin-A1-mediated endothelial cell migration, as well as tumor angiogenesis, in a dose-dependent manner. Furthermore, EGCG inhibited the ephrin-A1-mediated phosphorylation of EphA2 and EPK-1/2. Taken together, these data indicated that activation of ERK-1/2 plays an essential role in ephrin-A1-mediated cell migration. EGCG inhibited ephrin-A1-mediated endothelial migration and angiogenesis. It suggests a novel antiangiogenesis application of EGCG in cancer chemoprevention. (C) 2007 Elsevier Inc. All rights reserved

Consumption of S-Allylcysteine Inhibits the Growth of Human Non-Small-Cell Lung Carcinoma in a Mouse Xenograft Model

Lung cancer is one of the leading causes of cancer death in the world. Human non-small-cell lung carcinoma (NSCLC) accounts for almost 80% of lung cancer cases. Aberrant phosphoinositide 3-kinase (PI3K)/Akt/mTOR signaling pathways play important roles and have been widely observed in the development of NSCLC. Previous studies indicated that garlic extracts such as diallyl disulfide (DADS) and diallyl trisulfide (DATS) could inhibit the proliferation of several types of cancer in vitro. However, the inhibitory effects of S-allylcysteine (SAC) on the growth of NSCLC have not been demonstrated yet. Therefore, this study investigated whether consumption of SAC could prevent the growth of NSCLC in both in vitro and in vivo models. It was found that SAC significantly inhibited the proliferation of human NSCLC A-549 cells in vitro. Treatment of the NF-kappa B inhibitor, Bay-11-7082, could significantly inhibit the proliferation of NSCLC A-549 cells. The results demonstrated that SAC significantly suppressed the activation of mTOR, NF-kappa B, and cyclin D1 molecules in vitro. Furthermore, the results demonstrated that consumption of SAC significantly inhibited the growth of highly metastatic human NSCLC cells in tumor-bearing mice. Bioluminescence imaging and pathological and immunohistochemical (IHC) staining results also indicated that SAC could effectively suppress the growth and malignant progression of human NSCLC in vivo. The chemopreventive effects of SAC were associated with suppression of mTOR and NF-kappa B molecules in vivo. These results suggested that SAC could act as an effective agent against the malignant progression of human NSCLC in both in vitro and in vivo models