2,259 research outputs found
Bosons after Symmetry Breaking in Quantum Field Theory
We present a unified description of the spontaneous symmetry breaking and its
associated bosons in fermion field theory. There is no Goldstone boson in the
fermion field theory models of Nambu-Jona-Lasinio, Thirring and QCD after
the chiral symmetry is spontaneously broken in the new vacuum. The defect of
the Goldstone theorem is clarified, and the "massless boson" predicted by the
theorem is virtual and corresponds to just a massless fermion and
antifermion pair. Further, we discuss the exact spectrum of the Thirring model
by the Bethe ansatz solutions, and the analytical expressions of all the
physical observables enable us to understand the essence of the spontaneous
symmetry breaking in depth. Also, we examine the boson spectrum in QCD, and
show that bosons always have a finite mass for colors. The problem of
the light cone prescription in QCD is discussed, and it is shown that the
trivial light cone vacuum is responsible for the wrong prediction of the boson
mass.Comment: 56 pages,8 figure
Lower restrictions for start-ups to list on stock exchanges have mixed results
Firms that use the system grow faster in size, but not in profits, find Hidenori Takahashi and Kazuo Yamad
Spontaneous spin current near the interface between unconventional superconductors and ferromagnets
We study theoretically the proximity effect between ferromagnets (F) and
superconductors (S) with broken time-reversal symmetry (). A chiral
-wave, and a -wave superconductor, the latter of
which can form -breaking surface state, i.e., -state, are considered for the S side. The spatial variations of the
superconducting order parameters and the magnetization are determined by
solving the Bogoliubov de Gennes equation. In the case of a chiral -wave superconductor, a spontaneous spin current flows along the
interface, but not in the case of a -wave superconductor. For
F/S/F trilayer system, total spin current can be finite while total
charge current vanishes, if the magnetization of two F layers are antiparallel.Comment: 6 pages, 11 figures. Accepted for publication in Physical Review
Widely Extended [OIII] 88 um Line Emission around the 30 Doradus Region Revealed with AKARI FIS-FTS
We present the distribution map of the far-infrared [OIII] 88um line emission
around the 30 Doradus (30 Dor) region in the Large Magellanic Cloud obtained
with the Fourier Transform Spectrometer of the Far-Infrared Surveyor onboard
AKARI. The map reveals that the [OIII] emission is widely distributed by more
than 10' around the super star cluster R136, implying that the 30 Dor region is
affluent with interstellar radiation field hard enough to ionize O^{2+}. The
observed [OIII] line intensities are as high as (1-2) x 10^{-6} W m^{-2}
sr^{-1} on the peripheral regions 4'-5' away from the center of 30 Dor, which
requires gas densities of 60-100 cm^{-3}. However the observed size of the
distribution of the [OIII] emission is too large to be explained by massive
stars in the 30 Dor region enshrouded by clouds with the constant gas density
of 10^2 cm^{-3}. Therefore the surrounding structure is likely to be highly
clumpy. We also find a global correlation between the [OIII] and the
far-infrared continuum emission, suggesting that the gas and dust are well
mixed in the highly-ionized region where the dust survives in clumpy dense
clouds shielded from the energetic photons.Comment: 17 pages, 9 figures, accepted for publication in Publications of the
Astronomical Society of Japan (PASJ
High mobility group box 1 complexed with heparin induced angiogenesis in a matrigel plug assay
Angiogenesis involves complex processes mediated by several factors and is associated with inflammation and wound healing. High mobility group box 1 (HMGB1) is released from necrotic cells as well as macrophages and plays proinflammatory roles. In the present study, we examined whether HMGB1 would exhibit angiogenic activity in a matrigel plug assay in mice. HMGB1 in combination with heparin strongly induced angiogenesis, whereas neither HMGB1 nor heparin alone showed such angiogenic activity. The heparin-dependent induction of angiogenesis by HMGB1 was accompanied by increases in the expression of tumor necrosis factor-alpha (TNF-alpha) and vascular endothelial growth factor-A120 (VEGF-A120). It is likely that the dependence of the angiogenic activity of HMGB1 on heparin was due to the efficiency of the diffusion of the HMGB1-heparin complex from matrigel to the surrounding areas. VEGF-A165 possessing a heparin-binding domain showed a pattern of heparin-dependent angiogenic activity similar to that of HMGB1. The presence of heparin also inhibited the degradation of HMGB1 by plasmin in vitro. Taken together, these results suggested that HMGB1 in complex with heparin possesses remarkable angiogenic activity, probably through the induction of TNF-alpha and VEGF-A120.</p
- …
